Haiyun Ling

1.2k total citations
18 papers, 987 citations indexed

About

Haiyun Ling is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Pathology and Forensic Medicine. According to data from OpenAlex, Haiyun Ling has authored 18 papers receiving a total of 987 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 12 papers in Cardiology and Cardiovascular Medicine and 6 papers in Pathology and Forensic Medicine. Recurrent topics in Haiyun Ling's work include Cardiac electrophysiology and arrhythmias (11 papers), Ion channel regulation and function (9 papers) and Cardiac Ischemia and Reperfusion (6 papers). Haiyun Ling is often cited by papers focused on Cardiac electrophysiology and arrhythmias (11 papers), Ion channel regulation and function (9 papers) and Cardiac Ischemia and Reperfusion (6 papers). Haiyun Ling collaborates with scholars based in United States, Netherlands and Germany. Haiyun Ling's co-authors include Joan Heller Brown, Donald M. Bers, Kirk L. Peterson, Yusu Gu, Nancy D. Dalton, Michael Grimm, Laëtitia Pereira, Ju Chen, Hongqiang Cheng and Tong Zhang and has published in prestigious journals such as Journal of Biological Chemistry, Circulation and Journal of Clinical Investigation.

In The Last Decade

Haiyun Ling

18 papers receiving 974 citations

Peers

Haiyun Ling

CCM

PFM

CMN

PHYSI

Matilde Said Argentina

Guangyu Wu United States

Z. Maggie Huang United States

Gemin Ni United States

Doris Gehring Germany

Gerhild Taimor Germany

Christopher K. Means United States

Dominique Thuringer France

Artavazd Tadevosyan Canada

Kin M. Choi United States

Matilde Said Argentina

Haiyun Ling

578 ×0.8

548 ×0.9

CCM

232 ×1.7

PFM

115 ×0.9

CMN

86 ×1.1

PHYSI

Citations per year, relative to Haiyun Ling Haiyun Ling (= 1×) peers Matilde Said

Countries citing papers authored by Haiyun Ling

Since Specialization

Citations

This map shows the geographic impact of Haiyun Ling's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Haiyun Ling with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Haiyun Ling more than expected).

Fields of papers citing papers by Haiyun Ling

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Haiyun Ling. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Haiyun Ling. The network helps show where Haiyun Ling may publish in the future.

Co-authorship network of co-authors of Haiyun Ling

This figure shows the co-authorship network connecting the top 25 collaborators of Haiyun Ling. A scholar is included among the top collaborators of Haiyun Ling based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Haiyun Ling. Haiyun Ling is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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18 of 18 papers shown

Dewenter, Matthias, Julia H. Steinbrecher, B. Daan Westenbrink, et al.. (2024). Ca2+/Calmodulin-Dependent Kinase IIδC-Induced Chronic Heart Failure Does Not Depend on Sarcoplasmic Reticulum Ca2+ Leak. ESC Heart Failure. 11(4). 2191–2199. 4 indexed citations

Chi, Liguo, Luiz Belardinelli, Ryoko Hirakawa, et al.. (2016). Inhibition of late Na⁺ current, a novel target to improve diastolic function and electrical abnormalities in Dahl salt-sensitive rats. American Journal of Physiology-Heart and Circulatory Physiology. 310(10). H1313–H1320. 5 indexed citations

Grimm, Michael, Haiyun Ling, Andrew Willeford, et al.. (2015). CaMKIIδ mediates β-adrenergic effects on RyR2 phosphorylation and SR Ca2+ leak and the pathophysiological response to chronic β-adrenergic stimulation. Journal of Molecular and Cellular Cardiology. 85. 282–291. 63 indexed citations

Westenbrink, B. Daan, Haiyun Ling, Ajit S. Divakaruni, et al.. (2015). Mitochondrial Reprogramming Induced by CaMKIIδ Mediates Hypertrophy Decompensation. Circulation Research. 116(5). e28–39. 49 indexed citations

Said, Matilde, Haiyun Ling, Carlos A. Valverde, et al.. (2014). CaMKII-dependent phosphorylation of cardiac ryanodine receptors regulates cell death in cardiac ischemia/reperfusion injury. Journal of Molecular and Cellular Cardiology. 74. 274–283. 65 indexed citations

Suárez, Jorge, Hong Wang, Brian T. Scott, et al.. (2014). In vivo selective expression of thyroid hormone receptor α₁in endothelial cells attenuates myocardial injury in experimental myocardial infarction in mice. American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 307(3). R340–R346. 19 indexed citations

Chi, Liguo, Haiyun Ling, Dillon Phan, et al.. (2013). Abstract 15308: Reduction of LV Diastolic Wall Tension by Ranolazine Led to Improved Diastolic Coronary Blood Flow During Demand-Induced Acute Myocardial Ischemia in Anesthetized Canine. Circulation. 128. 1 indexed citations

Ling, Haiyun, Charles B.B. Gray, Alexander C. Zambon, et al.. (2013). Ca ²⁺ /Calmodulin-Dependent Protein Kinase II δ Mediates Myocardial Ischemia/Reperfusion Injury Through Nuclear Factor-κB. Circulation Research. 112(6). 935–944. 142 indexed citations

Westenbrink, B. Daan, Haiyun Ling, Shigeki Miyamoto, et al.. (2012). Abstract 18574: Mitochondrial Reprogramming By Camkiiδ Underlies Gq-induced Cardiomyopathy. Circulation. 126(suppl_21). 1 indexed citations

10.

Ling, Haiyun, Tong Zhang, Laëtitia Pereira, et al.. (2012). Requirement for Ca 2+/calmodulin-dependent kinase II in the transition from pressure overload-induced cardiac hypertrophy to heart failure in mice (Journal of Clinical Investigation (2009) 119, 5, (1230-1240) doi: 10.1172/JCI38022). Journal of Clinical Investigation. 122(4). 1 indexed citations

11.

Ling, Haiyun, Tong Zhang, Laëtitia Pereira, et al.. (2012). Requirement for Ca2+/calmodulin–dependent kinase II in the transition from pressure overload–induced cardiac hypertrophy to heart failure in mice. Journal of Clinical Investigation. 122(4). 1584–1584. 3 indexed citations

12.

Grimm, Michael, Haiyun Ling, & Joan Heller Brown. (2011). Crossing signals: relationships between β-adrenergic stimulation and CaMKII activation. Heart Rhythm. 8(8). 1296–1298. 14 indexed citations

13.

Xiang, Sunny, Davy Vanhoutte, Dominic P. Del Re, et al.. (2011). RhoA protects the mouse heart against ischemia/reperfusion injury. Journal of Clinical Investigation. 121(8). 3269–3276. 80 indexed citations

14.

Mishra, Shikha, Haiyun Ling, Michael Grimm, et al.. (2010). Cardiac Hypertrophy and Heart Failure Development Through Gq and CaM Kinase II Signaling. Journal of Cardiovascular Pharmacology. 56(6). 598–603. 48 indexed citations

15.

Ling, Haiyun, Tong Zhang, Laëtitia Pereira, et al.. (2009). Requirement for Ca2+/calmodulin–dependent kinase II in the transition from pressure overload–induced cardiac hypertrophy to heart failure in mice. Journal of Clinical Investigation. 119(5). 1230–1240. 293 indexed citations

16.

Ling, Haiyun, Tong Zhang, & Joan Heller Brown. (2008). CaMKII δ Deletion Protects Against Apoptosis In Myocardial Ischemia/Reperfusion Injury In Vivo. The FASEB Journal. 22(S1). 1 indexed citations

17.

Zhang, Tong, Michael Kohlhaas, Johannes Backs, et al.. (2007). CaMKIIδ Isoforms Differentially Affect Calcium Handling but Similarly Regulate HDAC/MEF2 Transcriptional Responses. Journal of Biological Chemistry. 282(48). 35078–35087. 166 indexed citations

18.

Ling, Haiyun. (1959). ACTIONS OF DIMETHYLPHENYLPIPERAZINIUM. British Journal of Pharmacology and Chemotherapy. 14(4). 505–511. 32 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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