Hailiang Wei

430 total citations
22 papers, 301 citations indexed

About

Hailiang Wei is a scholar working on Molecular Biology, Cancer Research and Epidemiology. According to data from OpenAlex, Hailiang Wei has authored 22 papers receiving a total of 301 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 9 papers in Cancer Research and 5 papers in Epidemiology. Recurrent topics in Hailiang Wei's work include Circular RNAs in diseases (6 papers), Cancer-related molecular mechanisms research (6 papers) and MicroRNA in disease regulation (4 papers). Hailiang Wei is often cited by papers focused on Circular RNAs in diseases (6 papers), Cancer-related molecular mechanisms research (6 papers) and MicroRNA in disease regulation (4 papers). Hailiang Wei collaborates with scholars based in China, Canada and Japan. Hailiang Wei's co-authors include Shuguang Yan, Jiaxiang Zhang, Hui Guo, Yonggang Liu, Jingtao Li, Yan Liang, Qian Li, Xiaofan Xu, Yingjun Guo and Ting Chen and has published in prestigious journals such as SHILAP Revista de lepidopterología, Journal of Ethnopharmacology and Frontiers in Pharmacology.

In The Last Decade

Hailiang Wei

22 papers receiving 297 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Hailiang Wei China 10 168 102 44 39 39 22 301
Xiaocui Zheng China 9 181 1.1× 60 0.6× 20 0.5× 50 1.3× 83 2.1× 15 327
Xinpei Yu China 9 203 1.2× 66 0.6× 14 0.3× 36 0.9× 37 0.9× 15 346
Yajing Weng China 10 169 1.0× 56 0.5× 19 0.4× 31 0.8× 62 1.6× 15 358
Xiaoxiao Yao China 8 191 1.1× 102 1.0× 25 0.6× 18 0.5× 40 1.0× 9 350
Myoung-Ok Kim South Korea 9 227 1.4× 60 0.6× 70 1.6× 24 0.6× 33 0.8× 25 351
Yinyin Xu China 5 137 0.8× 49 0.5× 36 0.8× 40 1.0× 41 1.1× 11 277
Huimin Lu China 6 175 1.0× 88 0.9× 31 0.7× 54 1.4× 48 1.2× 7 291
Gaojue Wu China 10 223 1.3× 55 0.5× 18 0.4× 16 0.4× 39 1.0× 20 385
Dezhi Li China 13 259 1.5× 102 1.0× 15 0.3× 16 0.4× 56 1.4× 25 425

Countries citing papers authored by Hailiang Wei

Since Specialization
Citations

This map shows the geographic impact of Hailiang Wei's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hailiang Wei with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hailiang Wei more than expected).

Fields of papers citing papers by Hailiang Wei

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hailiang Wei. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hailiang Wei. The network helps show where Hailiang Wei may publish in the future.

Co-authorship network of co-authors of Hailiang Wei

This figure shows the co-authorship network connecting the top 25 collaborators of Hailiang Wei. A scholar is included among the top collaborators of Hailiang Wei based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hailiang Wei. Hailiang Wei is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Yang, Kai, et al.. (2025). Macrophages as Multifaceted Orchestrators of Tissue Repair: Bridging Inflammation, Regeneration, and Therapeutic Innovation. Journal of Inflammation Research. Volume 18. 8945–8959. 7 indexed citations
2.
Wang, Rui, Ruijuan Yan, Feilong Li, et al.. (2024). Fruit and vegetable intake and the risk of non-alcoholic fatty liver disease: a meta-analysis of observational studies. Frontiers in Nutrition. 11. 1398184–1398184. 8 indexed citations
3.
Zhang, Jiaxiang, Juan Chen, Ting Chen, et al.. (2023). Xiaojianzhong decoction prevents gastric precancerous lesions in rats by inhibiting autophagy and glycolysis in gastric mucosal cells. World Journal of Gastrointestinal Oncology. 15(3). 464–489. 6 indexed citations
4.
Chen, Juan, Jiaxiang Zhang, Hailiang Wei, et al.. (2023). Mechanism of Xiaojianzhong decoction in alleviating aspirin-induced gastric mucosal injury revealed by transcriptomics and metabolomics. Journal of Ethnopharmacology. 318(Pt A). 116910–116910. 4 indexed citations
5.
Chen, Juan, Jiaxiang Zhang, Hailiang Wei, et al.. (2023). Xiaojianzhong decoction attenuates aspirin-induced gastric mucosal injury via the PI3K/AKT/mTOR/ULK1 and AMPK/ULK1 pathways. Pharmaceutical Biology. 61(1). 1234–1248. 9 indexed citations
6.
Yan, Shuguang, et al.. (2022). Treatment of ulcerative colitis with Wu-Mei-Wan by inhibiting intestinal inflammatory response and repairing damaged intestinal mucosa. Phytomedicine. 105. 154362–154362. 33 indexed citations
7.
Yan, Shuguang, et al.. (2022). Wu-Mei-Wan Ameliorates Murine Ulcerative Colitis by Regulating Macrophage Polarization. Frontiers in Pharmacology. 13. 859167–859167. 31 indexed citations
8.
Yu, Weichao, Zhenyu Liu, Bolin Huang, et al.. (2022). An Optimal Flow Allocation Model of the Natural Gas Pipeline Network Considering User Characteristics. Journal of Pipeline Systems Engineering and Practice. 13(3). 3 indexed citations
9.
10.
Chen, Juan, Jiaxiang Zhang, Ting Chen, et al.. (2022). Xiaojianzhong decoction attenuates gastric mucosal injury by activating the p62/Keap1/Nrf2 signaling pathway to inhibit ferroptosis. Biomedicine & Pharmacotherapy. 155. 113631–113631. 34 indexed citations
11.
Wang, Zhixin, et al.. (2022). Forecast of Ground Deformation Caused by Tunnel Excavation Based on Intelligent Neural Network Model. Mobile Information Systems. 2022. 1–12. 2 indexed citations
12.
Ma, Tao, Hailiang Wei, Shuguang Yan, et al.. (2021). Long Non-coding RNA SNHG17 Upregulates RFX1 by Sponging miR-3180-3p and Promotes Cellular Function in Hepatocellular Carcinoma. Frontiers in Genetics. 11. 607636–607636. 21 indexed citations
13.
14.
Gao, Shan, Wei Hu, Xin Huang, et al.. (2020). Circ_0001178 regulates miR-382/VEGFA axis to facilitate hepatocellular carcinoma progression. Cellular Signalling. 72. 109621–109621. 21 indexed citations
15.
Yan, Shuguang, et al.. (2020). CircTP53 promotes colorectal cancer by acting as a miR-876-3p sponge to increase cyclin-dependent kinase-like 3 expression. Cellular Signalling. 78. 109845–109845. 14 indexed citations
16.
Wei, Hailiang, et al.. (2020). Curcumin Inhibits Hepatocellular Carcinoma via Regulating miR‐21/TIMP3 Axis. Evidence-based Complementary and Alternative Medicine. 2020(1). 2892917–2892917. 37 indexed citations
17.
Yan, Shuguang, et al.. (2020). Glutamine relieves oxidative stress through PI3K/Akt signaling pathway in DSS-induced ulcerative colitis mice.. SHILAP Revista de lepidopterología. 23(9). 1124–1129. 22 indexed citations
18.
Yan, Shuguang, et al.. (2020). [Roumudan formulation attenuated liver fibrosis by inhibiting TGF-β1/Smad4 pathway in mice].. PubMed. 36(6). 576–581. 1 indexed citations
19.
Yan, Shuguang, et al.. (2020). [Effects of 6-Shogaol on Notch signaling pathway in colonic epithelial cells of ulcerative colitis mice].. PubMed. 36(1). 90–93. 4 indexed citations
20.
Yan, Shuguang, et al.. (2019). [Progress in the study of macrophage activation and ulcerative colitis from the Notch/NF-κB/NLRP3 signaling pathway].. PubMed. 44(10). 1174–1178. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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