Haihai Liang

4.6k total citations
108 papers, 3.4k citations indexed

About

Haihai Liang is a scholar working on Molecular Biology, Cancer Research and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Haihai Liang has authored 108 papers receiving a total of 3.4k indexed citations (citations by other indexed papers that have themselves been cited), including 65 papers in Molecular Biology, 50 papers in Cancer Research and 26 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Haihai Liang's work include Cancer-related molecular mechanisms research (28 papers), MicroRNA in disease regulation (23 papers) and Cardiac Fibrosis and Remodeling (15 papers). Haihai Liang is often cited by papers focused on Cancer-related molecular mechanisms research (28 papers), MicroRNA in disease regulation (23 papers) and Cardiac Fibrosis and Remodeling (15 papers). Haihai Liang collaborates with scholars based in China, Macao and Switzerland. Haihai Liang's co-authors include Hongli Shan, Yanjie Lu, Xiaoguang Zhao, Xuelian Li, Baofeng Yang, Yunyan Gu, Huitong Shan, Chaoqian Xu, Jian Sun and Rui Yang and has published in prestigious journals such as SHILAP Revista de lepidopterología, Scientific Reports and The FASEB Journal.

In The Last Decade

Haihai Liang

100 papers receiving 3.4k citations

Peers

Haihai Liang
Kang Yang China
Wei Ding China
Ursula Mayr United Kingdom
Sharon Lim Sweden
Qian Zhao China
Haihai Liang
Citations per year, relative to Haihai Liang Haihai Liang (= 1×) peers Hongli Shan

Countries citing papers authored by Haihai Liang

Since Specialization
Citations

This map shows the geographic impact of Haihai Liang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Haihai Liang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Haihai Liang more than expected).

Fields of papers citing papers by Haihai Liang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Haihai Liang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Haihai Liang. The network helps show where Haihai Liang may publish in the future.

Co-authorship network of co-authors of Haihai Liang

This figure shows the co-authorship network connecting the top 25 collaborators of Haihai Liang. A scholar is included among the top collaborators of Haihai Liang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Haihai Liang. Haihai Liang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ma, Huimin, et al.. (2025). Proprotein convertase subtilisin/kexin type 9 contributes to cisplatin-induced acute kidney injury by interacting with cyclase-associated protein 1 to promote megalin lysosomal degradation. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1872(6). 119984–119984. 1 indexed citations
2.
Dong, Qi, Yingying Guo, Chen Lv, et al.. (2025). Unveiling a novel cancer hallmark by evaluation of neural infiltration in cancer. Briefings in Bioinformatics. 26(2). 1 indexed citations
3.
Bi, Fangfang, Lifang Lv, Hua Tian, et al.. (2024). ITFG2, an immune-modulatory protein, targets ATP 5b to maintain mitochondrial function in myocardial infarction. Biochemical Pharmacology. 226. 116338–116338. 2 indexed citations
4.
Chen, Tingting, Yuquan Wang, Yan Wang, et al.. (2023). Up-regulated SPP1 increases the risk from IPF to lung cancer via activating the pro-tumor macrophages. Computational and Structural Biotechnology Journal. 21. 5751–5764. 8 indexed citations
5.
Xie, Yilin, Huifang Wang, Wenxian Chen, et al.. (2023). Berberine inhibits NLRP3 inflammasome activation by regulating mTOR/mtROS axis to alleviate diabetic cardiomyopathy. European Journal of Pharmacology. 964. 176253–176253. 19 indexed citations
6.
Yang, Rui, Liangliang Li, Yingnan Li, et al.. (2023). Long non-coding RNA KCND1 protects hearts from hypertrophy by targeting YBX1. Cell Death and Disease. 14(5). 344–344. 16 indexed citations
7.
Li, Tianyu, Jingrui Li, Xin Lü, et al.. (2023). Nucleosome assembly protein 1 like 1 (NAP1L1) promotes cardiac fibrosis by inhibiting YAP1 ubiquitination and degradation. SHILAP Revista de lepidopterología. 4(5). e348–e348. 6 indexed citations
8.
Wang, Yujing, Yue Li, Yao Liu, et al.. (2023). Neuroepithelial cell-transforming 1 promotes cardiac fibrosis via the Wnt/β-catenin signaling pathway. iScience. 26(10). 107888–107888. 7 indexed citations
9.
Yu, Tong, Yanyan Liu, Xiang Sun, et al.. (2022). Gankyrin modulated non-small cell lung cancer progression via glycolysis metabolism in a YAP1-dependent manner. Cell Death Discovery. 8(1). 312–312. 9 indexed citations
10.
Zhang, Yue, Zhiying Wang, Jingjing Zhao, et al.. (2022). MicroRNA-24-3p alleviates cardiac fibrosis by suppressing cardiac fibroblasts mitophagy via downregulating PHB2. Pharmacological Research. 177. 106124–106124. 42 indexed citations
11.
Su, Wei, Liangliang Li, Xiaoguang Zhao, et al.. (2022). Critical role of PAFR/YAP1 positive feedback loop in cardiac fibrosis. Acta Pharmacologica Sinica. 43(11). 2862–2872. 10 indexed citations
12.
Zhao, Xiaoguang, et al.. (2020). The influences of driving forces on behaviors of Na+ and H2O in cyclic octa-peptide nanotube: investigated by steered molecular dynamics. Materials Research Express. 7(6). 65010–65010. 1 indexed citations
13.
Liang, Haihai, et al.. (2019). Prospect of immunotherapy combined with anti-angiogenic agents in patients with advanced non-small cell lung cancer. SHILAP Revista de lepidopterología. 1 indexed citations
14.
Zhang, Mingyu, Yuan Jiang, Xiaofei Guo, et al.. (2019). Long non‐coding RNA cardiac hypertrophy‐associated regulator governs cardiac hypertrophy via regulating miR‐20b and the downstream PTEN/AKT pathway. Journal of Cellular and Molecular Medicine. 23(11). 7685–7698. 45 indexed citations
15.
Han, Yue, Chengyu Wang, Qi Dong, et al.. (2019). Genetic Interaction-Based Biomarkers Identification for Drug Resistance and Sensitivity in Cancer Cells. Molecular Therapy — Nucleic Acids. 17. 688–700. 14 indexed citations
16.
Zhao, Dandan, Li Cui, Yan He, et al.. (2018). Cardiomyocyte Derived miR-328 Promotes Cardiac Fibrosis by Paracrinely Regulating Adjacent Fibroblasts. Cellular Physiology and Biochemistry. 46(4). 1555–1565. 21 indexed citations
17.
Lv, Lifang, Tianyu Li, Xuelian Li, et al.. (2017). The lncRNA Plscr4 Controls Cardiac Hypertrophy by Regulating miR-214. Molecular Therapy — Nucleic Acids. 10. 387–397. 99 indexed citations
18.
Su, Xiaomin, Haihai Liang, He Wang, et al.. (2017). Over-expression of microRNA-1 causes arrhythmia by disturbing intracellular trafficking system. Scientific Reports. 7(1). 46259–46259. 22 indexed citations
19.
Chi, Jinyu, et al.. (2015). MicroRNA-377 Mediates Cardiomyocyte Apoptosis Induced by Cyclosporin A. Canadian Journal of Cardiology. 32(10). 1249–1259. 20 indexed citations
20.
Du, Weijie, Zhenwei Pan, Xu Chen, et al.. (2014). By Targeting Stat3 microRNA-17-5p Promotes Cardiomyocyte Apoptosis in Response to Ischemia Followed by Reperfusion. Cellular Physiology and Biochemistry. 34(3). 955–965. 74 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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