Hai Jiang

1.3k total citations
36 papers, 842 citations indexed

About

Hai Jiang is a scholar working on Molecular Biology, Oncology and Immunology. According to data from OpenAlex, Hai Jiang has authored 36 papers receiving a total of 842 indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Molecular Biology, 8 papers in Oncology and 8 papers in Immunology. Recurrent topics in Hai Jiang's work include DNA Repair Mechanisms (5 papers), Cancer therapeutics and mechanisms (4 papers) and Phagocytosis and Immune Regulation (4 papers). Hai Jiang is often cited by papers focused on DNA Repair Mechanisms (5 papers), Cancer therapeutics and mechanisms (4 papers) and Phagocytosis and Immune Regulation (4 papers). Hai Jiang collaborates with scholars based in China, United States and United Kingdom. Hai Jiang's co-authors include Honglin Li, Michael T. Hemann, Michael B. Yaffe, Hans Christian Reinhardt, Heli Nevanlinna, Jiřina Bártková, Jiří Bártek, Carl Blomqvist, Johanna Tommiska and Shouqing Luo and has published in prestigious journals such as Journal of Biological Chemistry, Nature Communications and Journal of Clinical Oncology.

In The Last Decade

Hai Jiang

33 papers receiving 824 citations

Peers

Hai Jiang
Jaine K. Blayney United Kingdom
Dae-Seok Kim South Korea
Xi Rao China
Federica Brugnoli Italy
Wolfgang Hulla Austria
Richard Marcotte Canada
Patrick Bhola United States
Gabriela Paroni Italy
Eric D. Slosberg United States
Anthony E. Rizzardi United States
Jaine K. Blayney United Kingdom
Hai Jiang
Citations per year, relative to Hai Jiang Hai Jiang (= 1×) peers Jaine K. Blayney

Countries citing papers authored by Hai Jiang

Since Specialization
Citations

This map shows the geographic impact of Hai Jiang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hai Jiang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hai Jiang more than expected).

Fields of papers citing papers by Hai Jiang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hai Jiang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hai Jiang. The network helps show where Hai Jiang may publish in the future.

Co-authorship network of co-authors of Hai Jiang

This figure shows the co-authorship network connecting the top 25 collaborators of Hai Jiang. A scholar is included among the top collaborators of Hai Jiang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hai Jiang. Hai Jiang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Jiang, Hai, et al.. (2025). Quercetin alleviates acute pancreatitis by modulating glycolysis and mitochondrial function via PFKFB3 inhibition. Cellular and Molecular Life Sciences. 82(1). 311–311.
2.
Zheng, Chuanming, Rui Tao, Zhenjie Wang, et al.. (2025). CLDN4 promotes ferroptosis and inflammation involving JAK2/STAT3 pathway in acute pancreatitis. Functional & Integrative Genomics. 25(1). 184–184.
3.
Ye, Bao‐Lin, et al.. (2024). A Space Telescope Scheduling Approach Combining Observation Priority Coding with Problem Decomposition Strategies. Biomimetics. 9(12). 718–718. 1 indexed citations
4.
Zhang, Wanlin, et al.. (2024). A hsa_circ_001726 axis regulated by E2F6 contributes to metastasis of hepatocellular carcinoma. BMC Cancer. 24(1). 14–14. 5 indexed citations
5.
Jiang, Hai, et al.. (2023). An emergency observation method for space debris dangerous conjunction events based on nanosatellite constellation. Advances in Space Research. 73(11). 5545–5559. 2 indexed citations
6.
Li, Lei, et al.. (2022). Effect of miR-21-3p on lung injury in rats with traumatic hemorrhagic shock resuscitated with sodium bicarbonate Ringer’s solution. Annals of Translational Medicine. 10(24). 1331–1331. 1 indexed citations
7.
Xu, Feng, Zhenjie Wang, Peng Yang, et al.. (2021). Blockade of JAK2 signaling produces immunomodulatory effect to preserve pancreatic homeostasis in severe acute pancreatitis. Biochemistry and Biophysics Reports. 28. 101133–101133. 7 indexed citations
8.
Ding, Hongyu, Jie Zhao, Yanli Zhang, et al.. (2019). Systematic Analysis of Drug Vulnerabilities Conferred by Tumor Suppressor Loss. Cell Reports. 27(11). 3331–3344.e6. 18 indexed citations
9.
Bihani, Teeru, Hitisha Patel, Heike Arlt, et al.. (2017). Elacestrant (RAD1901), a Selective Estrogen Receptor Degrader (SERD), Has Antitumor Activity in Multiple ER+ Breast Cancer Patient-derived Xenograft Models. Clinical Cancer Research. 23(16). 4793–4804. 119 indexed citations
10.
Yang, Wei, Shutan Jiang, Feng Xiao, et al.. (2017). Dynamic regulation of CD28 conformation and signaling by charged lipids and ions. Nature Structural & Molecular Biology. 24(12). 1081–1092. 49 indexed citations
11.
Wang, Daisong, Chunye Liu, Jingqiang Wang, et al.. (2017). Protein C receptor stimulates multiple signaling pathways in breast cancer cells. Journal of Biological Chemistry. 293(4). 1413–1424. 25 indexed citations
12.
He, Chuanchao, Zhenyu Zhou, Hai Jiang, et al.. (2016). Epithelial-Mesenchymal Transition is Superior to Vessels-Encapsulate Tumor Cluster in Promoting Metastasis of Hepatocellular Carcinoma: a Morphological Evidence. Journal of Cancer. 8(1). 39–47. 17 indexed citations
13.
Han, Hui, Chenyu Pan, Chunying Liu, et al.. (2015). Gut–neuron interaction via Hh signaling regulates intestinal progenitor cell differentiation in Drosophila. Cell Discovery. 1(1). 15006–15006. 18 indexed citations
14.
Ding, Hongyu, Xiaoxi Li, Christian P. Pallasch, et al.. (2015). A DNA / HDAC dual‐targeting drug CY 190602 with significantly enhanced anticancer potency. EMBO Molecular Medicine. 7(4). 438–449. 24 indexed citations
15.
Pritchard, Justin R., Luke A. Gilbert, Corbin E. Meacham, et al.. (2011). Bcl-2 Family Genetic Profiling Reveals Microenvironment-Specific Determinants of Chemotherapeutic Response. Cancer Research. 71(17). 5850–5858. 1 indexed citations
16.
Gilbert, Luke A., Corbin E. Meacham, Hai Jiang, et al.. (2011). BCL-2 family genetic profiling reveals microenvironment-specific determinants of chemotherapeutic response. DSpace@MIT (Massachusetts Institute of Technology). 13 indexed citations
17.
Jiang, Hai, Justin R. Pritchard, Richard T. Williams, Douglas A. Lauffenburger, & Michael T. Hemann. (2010). A mammalian functional-genetic approach to characterizing cancer therapeutics. Nature Chemical Biology. 7(2). 92–100. 2 indexed citations
18.
Jiang, Hai, Jianchun Wu, Chen He, Wending Yang, & Honglin Li. (2009). Tumor suppressor protein C53 antagonizes checkpoint kinases to promote cyclin-dependent kinase 1 activation. Cell Research. 19(4). 458–468. 46 indexed citations
19.
Reinhardt, Hans Christian, Hai Jiang, Michael T. Hemann, & Michael B. Yaffe. (2009). Exploiting synthetic lethal interactions for targeted cancer therapy. Cell Cycle. 8(19). 3112–3119. 4 indexed citations
20.
Jiang, Hai, Hans Christian Reinhardt, Jiřina Bártková, et al.. (2009). The combined status of ATM and p53 link tumor development with therapeutic response. Genes & Development. 23(16). 1895–1909. 231 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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