Hae‐Ok Byun

876 total citations
13 papers, 626 citations indexed

About

Hae‐Ok Byun is a scholar working on Molecular Biology, Physiology and Immunology. According to data from OpenAlex, Hae‐Ok Byun has authored 13 papers receiving a total of 626 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 5 papers in Physiology and 3 papers in Immunology. Recurrent topics in Hae‐Ok Byun's work include Telomeres, Telomerase, and Senescence (5 papers), Mitochondrial Function and Pathology (3 papers) and Herpesvirus Infections and Treatments (2 papers). Hae‐Ok Byun is often cited by papers focused on Telomeres, Telomerase, and Senescence (5 papers), Mitochondrial Function and Pathology (3 papers) and Herpesvirus Infections and Treatments (2 papers). Hae‐Ok Byun collaborates with scholars based in South Korea and United States. Hae‐Ok Byun's co-authors include Gyesoon Yoon, Young‐Kyoung Lee, Yonghak Seo, Jeong‐Min Kim, Hwan Myung Kim, Cheol Ho Heo, Debabrata Sen, Hyo Won Lee, Hyeseong Cho and Young‐Sil Yoon and has published in prestigious journals such as Journal of Biological Chemistry, Analytical Chemistry and Frontiers in Immunology.

In The Last Decade

Hae‐Ok Byun

13 papers receiving 619 citations

Peers

Hae‐Ok Byun
Víctor García‐González Mexico
Hiroyuki Kawagishi Japan
Nadiya Druzhyna United States
Chang‐Nim Im South Korea
Elizabeth A. Rowland United States
Alexander Polinsky United States
Suresh N. Kumar United States
Keon‐Hyoung Song South Korea
Orna Nadiv Israel
Heike Adler Germany
Víctor García‐González Mexico
Hae‐Ok Byun
Citations per year, relative to Hae‐Ok Byun Hae‐Ok Byun (= 1×) peers Víctor García‐González

Countries citing papers authored by Hae‐Ok Byun

Since Specialization
Citations

This map shows the geographic impact of Hae‐Ok Byun's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hae‐Ok Byun with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hae‐Ok Byun more than expected).

Fields of papers citing papers by Hae‐Ok Byun

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hae‐Ok Byun. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hae‐Ok Byun. The network helps show where Hae‐Ok Byun may publish in the future.

Co-authorship network of co-authors of Hae‐Ok Byun

This figure shows the co-authorship network connecting the top 25 collaborators of Hae‐Ok Byun. A scholar is included among the top collaborators of Hae‐Ok Byun based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hae‐Ok Byun. Hae‐Ok Byun is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
Islam, Sidra, Hae‐Ok Byun, Ju‐Yang Jung, et al.. (2021). Eubacterium rectale Attenuates HSV-1 Induced Systemic Inflammation in Mice by Inhibiting CD83. Frontiers in Immunology. 12. 712312–712312. 35 indexed citations
2.
3.
Islam, Sidra, et al.. (2019). Inhibition of CD83 Alleviates Systemic Inflammation in Herpes Simplex Virus Type 1-Induced Behçet’s Disease Model Mouse. Mediators of Inflammation. 2019. 1–15. 7 indexed citations
4.
Jung, Hyun-Jung, Hae‐Ok Byun, Byul A Jee, et al.. (2017). The Ubiquitin-like with PHD and Ring Finger Domains 1 (UHRF1)/DNA Methyltransferase 1 (DNMT1) Axis Is a Primary Regulator of Cell Senescence. Journal of Biological Chemistry. 292(9). 3729–3739. 35 indexed citations
5.
Byun, Hae‐Ok, Young‐Kyoung Lee, Jeong‐Min Kim, & Gyesoon Yoon. (2015). From cell senescence to age-related diseases: differential mechanisms of action of senescence-associated secretory phenotypes. BMB Reports. 48(10). 549–558. 89 indexed citations
6.
Lee, Jong‐Hyuk, Young‐Kyoung Lee, Hae‐Ok Byun, et al.. (2015). Mitochondrial Respiratory Dysfunction Induces Claudin-1 Expression via Reactive Oxygen Species-mediated Heat Shock Factor 1 Activation, Leading to Hepatoma Cell Invasiveness. Journal of Biological Chemistry. 290(35). 21421–21431. 17 indexed citations
7.
Song, Insun, Kabsun Kim, Jung Ha Kim, et al.. (2014). GATA4 negatively regulates osteoblast differentiation by downregulation of Runx2. BMB Reports. 47(8). 463–468. 16 indexed citations
8.
Byun, Hae‐Ok, et al.. (2014). PKCδ phosphorylation is an upstream event of GSK3 inactivation-mediated ROS generation in TGF-β1-induced senescence. Free Radical Research. 48(9). 1100–1108. 20 indexed citations
9.
Lee, Hyo Won, Cheol Ho Heo, Debabrata Sen, et al.. (2014). Ratiometric Two-Photon Fluorescent Probe for Quantitative Detection of β-Galactosidase Activity in Senescent Cells. Analytical Chemistry. 86(20). 10001–10005. 136 indexed citations
10.
Byun, Hae‐Ok, Byul A Jee, Hyunwoo Cho, et al.. (2013). Implications of time‐series gene expression profiles of replicative senescence. Aging Cell. 12(4). 622–634. 75 indexed citations
11.
Byun, Hae‐Ok, Hyun-Jung Jung, Yonghak Seo, et al.. (2012). GSK3 inactivation is involved in mitochondrial complex IV defect in transforming growth factor (TGF) β1-induced senescence. Experimental Cell Research. 318(15). 1808–1819. 53 indexed citations
12.
Byun, Hae‐Ok, et al.. (2008). Mitochondrial dysfunction by complex II inhibition delays overall cell cycle progression via reactive oxygen species production. Journal of Cellular Biochemistry. 104(5). 1747–1759. 57 indexed citations
13.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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