Hadhimulya Asmara

459 total citations
10 papers, 336 citations indexed

About

Hadhimulya Asmara is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Hadhimulya Asmara has authored 10 papers receiving a total of 336 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 7 papers in Cellular and Molecular Neuroscience and 5 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Hadhimulya Asmara's work include Ion channel regulation and function (8 papers), Neuroscience and Neuropharmacology Research (6 papers) and Cardiac electrophysiology and arrhythmias (5 papers). Hadhimulya Asmara is often cited by papers focused on Ion channel regulation and function (8 papers), Neuroscience and Neuropharmacology Research (6 papers) and Cardiac electrophysiology and arrhythmias (5 papers). Hadhimulya Asmara collaborates with scholars based in Canada, Japan and United States. Hadhimulya Asmara's co-authors include Ray W. Turner, Gerald W. Zamponi, Jordan D. T. Engbers, Etsuko Minobe, Masaki Kameyama, Zahangir Alam Saud, Giriraj Sahu, Shahid Hameed, Fang‐Xiong Zhang and W. Hamish Mehaffey and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Hadhimulya Asmara

10 papers receiving 336 citations

Peers

Countries citing papers authored by Hadhimulya Asmara

Since Specialization

Citations

This map shows the geographic impact of Hadhimulya Asmara's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hadhimulya Asmara with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hadhimulya Asmara more than expected).

Fields of papers citing papers by Hadhimulya Asmara

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hadhimulya Asmara. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hadhimulya Asmara. The network helps show where Hadhimulya Asmara may publish in the future.

Co-authorship network of co-authors of Hadhimulya Asmara

This figure shows the co-authorship network connecting the top 25 collaborators of Hadhimulya Asmara. A scholar is included among the top collaborators of Hadhimulya Asmara based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hadhimulya Asmara. Hadhimulya Asmara is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

#	Work	Indexed citations
1	Calcium-Dependent Regulation of Neuronal Excitability Is Rescued in Fragile X Syndrome by a Tat-Conjugated N-Terminal Fragment of FMRP 2024 ·Journal of Neuroscience ·(unknown), Hadhimulya Asmara, (unknown), Ray W. Turner	3
2	FMRP(1–297)-tat restores ion channel and synaptic function in a model of Fragile X syndrome 2020 ·Nature Communications ·(unknown), Hadhimulya Asmara, Ning Cheng, Giriraj Sahu, Eduardo Sánchez, Fang‐Xiong Zhang, Gerald W. Zamponi, Jong M. Rho, Ray W. Turner	24
3	A T-type channel-calmodulin complex triggers αCaMKII activation 2017 ·Molecular Brain ·Hadhimulya Asmara, Ileana Micu, (unknown), Giriraj Sahu, Brett Simms, Fang‐Xiong Zhang, Jordan D. T. Engbers, Peter K. Stys, Gerald W. Zamponi, Ray W. Turner	32
4	Activity-Dependent Facilitation of Ca_V1.3 Calcium Channels Promotes KCa3.1 Activation in Hippocampal Neurons 2017 ·Journal of Neuroscience ·Giriraj Sahu, Hadhimulya Asmara, Fang‐Xiong Zhang, Gerald W. Zamponi, Ray W. Turner	29
5	Assessing the role of IKCa channels in generating the sAHP of CA1 hippocampal pyramidal cells 2016 ·Channels ·Ray W. Turner, Hadhimulya Asmara, Jordan D. T. Engbers, (unknown), (unknown), Giriraj Sahu, Gerald W. Zamponi	21
6	IKCa Channels Are a Critical Determinant of the Slow AHP in CA1 Pyramidal Neurons 2015 ·Cell Reports ·Brian King, (unknown), Hadhimulya Asmara, (unknown), Jordan D. T. Engbers, Steven Dykstra, Theodore M. Bartoletti, Shahid Hameed, Gerald W. Zamponi, Ray W. Turner	72
7	Intermediate conductance calcium-activated potassium channels modulate summation of parallel fiber input in cerebellar Purkinje cells 2012 ·Proceedings of the National Academy of Sciences ·Jordan D. T. Engbers, Dustin Anderson, Hadhimulya Asmara, Renata Rehak, W. Hamish Mehaffey, Shahid Hameed, Bruce E. McKay, (unknown), Gerald W. Zamponi, Ray W. Turner	81
8	Calpastatin Domain L Is a Partial Agonist of the Calmodulin-binding Site for Channel Activation in Cav1.2 Ca2+ Channels 2011 ·Journal of Biological Chemistry ·Etsuko Minobe, Hadhimulya Asmara, Zahangir Alam Saud, Masaki Kameyama	26
9	Interactions of Calmodulin With the Multiple Binding Sites of Cav1.2 Ca2+ Channels 2010 ·Journal of Pharmacological Sciences ·Hadhimulya Asmara, Etsuko Minobe, Zahangir Alam Saud, Masaki Kameyama	37
10	Both N- and C-lobes of calmodulin are required for Ca2+-dependent regulations of CaV1.2 Ca2+ channels 2009 ·Biochemical and Biophysical Research Communications ·Feng Guo, Etsuko Minobe, Kazuto Yazawa, Hadhimulya Asmara, (unknown), (unknown), Liying Hao, Masaki Kameyama	11

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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