H. Weigel

813 total citations
8 papers, 707 citations indexed

About

H. Weigel is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, H. Weigel has authored 8 papers receiving a total of 707 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 5 papers in Cellular and Molecular Neuroscience and 3 papers in Neurology. Recurrent topics in H. Weigel's work include Neuroscience and Neuropharmacology Research (4 papers), Neuroinflammation and Neurodegeneration Mechanisms (3 papers) and Connexins and lens biology (2 papers). H. Weigel is often cited by papers focused on Neuroscience and Neuropharmacology Research (4 papers), Neuroinflammation and Neurodegeneration Mechanisms (3 papers) and Connexins and lens biology (2 papers). H. Weigel collaborates with scholars based in Germany, Netherlands and Denmark. H. Weigel's co-authors include Thomas W. Hansen, Steven A. Goldman, Anker Jón Hansen, Jane H.-C. Lin, Maria Luisa Cotrina, Maiken Nedergaard, Shujun Liu, Ulf T. Eysel, Rainald Schmidt‐Kastner and K.‐P. Hoffmann and has published in prestigious journals such as Nature Neuroscience, The Journal of Comparative Neurology and Neuroscience.

In The Last Decade

H. Weigel

8 papers receiving 690 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
H. Weigel Germany	6	423	266	207	83	73	8	707
John Dodge United States	10	190 0.4×	196 0.7×	125 0.6×	94 1.1×	72 1.0×	11	536
Johanna Knöferle Germany	12	299 0.7×	284 1.1×	135 0.7×	191 2.3×	105 1.4×	12	731
Yunju Jin United States	9	398 0.9×	204 0.8×	156 0.8×	120 1.4×	58 0.8×	12	721
JA Kessler United States	9	479 1.1×	451 1.7×	77 0.4×	167 2.0×	70 1.0×	11	792
Nabil A. Azzam United States	16	215 0.5×	166 0.6×	113 0.5×	125 1.5×	69 0.9×	26	636
Carl Moll Switzerland	12	630 1.5×	814 3.1×	144 0.7×	70 0.8×	79 1.1×	18	1.1k
Yosuke M. Morizawa Japan	9	167 0.4×	175 0.7×	357 1.7×	91 1.1×	89 1.2×	11	660
Terence H. Williams United States	18	274 0.6×	348 1.3×	58 0.3×	126 1.5×	56 0.8×	36	727
E. Farkas‐Bargeton France	12	247 0.6×	211 0.8×	78 0.4×	65 0.8×	67 0.9×	36	522
Michele Curcio Italy	10	284 0.7×	335 1.3×	111 0.5×	97 1.2×	152 2.1×	12	687

Countries citing papers authored by H. Weigel

Since Specialization

Citations

This map shows the geographic impact of H. Weigel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by H. Weigel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites H. Weigel more than expected).

Fields of papers citing papers by H. Weigel

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by H. Weigel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by H. Weigel. The network helps show where H. Weigel may publish in the future.

Co-authorship network of co-authors of H. Weigel

This figure shows the co-authorship network connecting the top 25 collaborators of H. Weigel. A scholar is included among the top collaborators of H. Weigel based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with H. Weigel. H. Weigel is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Lin, Jane H.-C., H. Weigel, Maria Luisa Cotrina, et al.. (1998). Correction: Gap-junction-mediated propagation and amplification of cell injury. Nature Neuroscience. 1(8). 743–743. 1 indexed citations

2.

Lin, Jane H.-C., H. Weigel, Maria Luisa Cotrina, et al.. (1998). Gap-junction-mediated propagation and amplification of cell injury. Nature Neuroscience. 1(6). 494–500. 419 indexed citations

3.

Schroeter, Michael, Klaus Schiene, Matthias Kraemer, et al.. (1995). Astroglial responses in photochemically induced focal ischemia of the rat cortex. Experimental Brain Research. 106(1). 1–6. 90 indexed citations

4.

Mittmann, Thomas, Heiko J. Luhmann, Rainald Schmidt‐Kastner, et al.. (1994). Lesion-induced transient suppression of inhibitory function in rat neocortex in vitro. Neuroscience. 60(4). 891–906. 77 indexed citations

5.

Schmidt‐Kastner, Rainald, et al.. (1994). Visualization of axonal degeneration after optic nerve lesion in rat by immunohistochemical labelling for myelin basic protein (MBP). Restorative Neurology and Neuroscience. 6(2). 145–149. 3 indexed citations

6.

Blaauwgeers, J. L. G., et al.. (1993). Langerhans cell histiocytosis of the vulva. European Journal of Obstetrics & Gynecology and Reproductive Biology. 48(2). 145–148. 5 indexed citations

7.

Distler, C., H. Weigel, & K.‐P. Hoffmann. (1993). Glia cells of the monkey retina. I. Astrocytes. The Journal of Comparative Neurology. 333(1). 134–147. 42 indexed citations

8.

Schmidt‐Kastner, Rainald, et al.. (1993). Immunohistochemical staining for glial fibrillary acidic protein (GFAP) after deafferentation or ischemic infarction in rat visual system: Features of reactive and damaged astrocytes. International Journal of Developmental Neuroscience. 11(2). 157–174. 70 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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