Maria Luisa Cotrina
- Molecular Biology top 5%
- Cellular and Molecular Neuroscience top 1%
- Neurology top 1%
- Physiology top 0.2%
- Physiology top 5%
- Co-authors
- Maiken NedergaardJane H.-C. LinShujun LiuChristian C. NausJian KangSteven A. GoldmanTakahiro TakanoAlexandra Alves‐Rodrigues
- Topics
- Connexins and lens biology (13 papers)Neuroscience and Neuropharmacology Research (11 papers)Neuroinflammation and Neurodegeneration Mechanisms (6 papers)
- Partner nations
- United StatesDenmarkCanada
In The Last Decade
Maria Luisa Cotrina
29 papers receiving 3.7k citations
Hit Papers
Peers
Comparison fields: 5 of 129
- Molecular Biology 2.0k
- Cellular and Molecular Neuroscience 1.4k
- Neurology 852
- Physiology 624
- Physiology 475
Countries citing papers authored by Maria Luisa Cotrina
This map shows the geographic impact of Maria Luisa Cotrina's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Maria Luisa Cotrina with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Maria Luisa Cotrina more than expected).
Fields of papers citing papers by Maria Luisa Cotrina
This network shows the impact of papers produced by Maria Luisa Cotrina. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Maria Luisa Cotrina. The network helps show where Maria Luisa Cotrina may publish in the future.
Co-authorship network of co-authors of Maria Luisa Cotrina
This figure shows the co-authorship network connecting the top 25 collaborators of Maria Luisa Cotrina. A scholar is included among the top collaborators of Maria Luisa Cotrina based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Maria Luisa Cotrina. Maria Luisa Cotrina is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 6 | |
| 3 | 46 | |
| 4 | 54 | |
| 5 | 13 | |
| 6 | 204 | |
| 7 | 26 | |
| 8 | 66 | |
| 9 | 85 | |
| 10 | 231 | |
| 11 | 62 | |
| 12 | 131 | |
| 13 | 26 | |
| 14 | 1 | |
| 15 | 338 | |
| 16 | 41 | |
| 17 | 49 | |
| 18 | 1 | |
| 19 | 419 | |
| 20 | 174 |
About Maria Luisa Cotrina
Maria Luisa Cotrina is a scholar working on Cellular and Molecular Neuroscience, Neurology and Physiology, having authored 29 papers that have together received 3.7k indexed citations. Recurring topics across this work include Connexins and lens biology (13 papers), Neuroscience and Neuropharmacology Research (11 papers) and Neuroinflammation and Neurodegeneration Mechanisms (6 papers). The work is most often cited by research in Physiology (624 citations), Neurology (852 citations) and Developmental Neuroscience (310 citations). Maria Luisa Cotrina has collaborated with scholars based in United States, Denmark and Canada. Frequent co-authors include Maiken Nedergaard, Jane H.-C. Lin, Shujun Liu, Christian C. Naus, Jian Kang, Steven A. Goldman, Takahiro Takano, Alexandra Alves‐Rodrigues, Jiang Li and Thomas W. Hansen. Their work appears in journals such as Proceedings of the National Academy of Sciences, Nature Medicine and Journal of Neuroscience.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.