H Pelletier

968 total citations · 1 hit paper
16 papers, 879 citations indexed

About

H Pelletier is a scholar working on Oncology, Molecular Biology and Immunology. According to data from OpenAlex, H Pelletier has authored 16 papers receiving a total of 879 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Oncology, 5 papers in Molecular Biology and 5 papers in Immunology. Recurrent topics in H Pelletier's work include Cancer Research and Treatments (3 papers), Immune Cell Function and Interaction (3 papers) and Cancer Immunotherapy and Biomarkers (3 papers). H Pelletier is often cited by papers focused on Cancer Research and Treatments (3 papers), Immune Cell Function and Interaction (3 papers) and Cancer Immunotherapy and Biomarkers (3 papers). H Pelletier collaborates with scholars based in France, United Kingdom and United States. H Pelletier's co-authors include Amalendra Kumar, Samuel H. Wilson, M.R. Sawaya, Joseph Kraut, Florian Martin, Anne Caignard, Bruno Chauffert, Arlette Hammann, Miguel Martín and Philippe Genne and has published in prestigious journals such as British Journal of Cancer, International Journal of Cancer and Cancer Immunology Immunotherapy.

In The Last Decade

H Pelletier

16 papers receiving 863 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

STRUCTURES OF TERNARY COMPLEXES OF RAT DNA POLYMERASE BETA, A DNA TEMPLATE-PRIMER, AND DDCTP

1994 635 citations H Pelletier, M.R. Sawaya et al. PubMed profile →

Peers

H Pelletier

ONCOL

GENET

IMMUN

Charles Garrett United States

R. Sh. Beabealashvilli Russia

Priscilla P. Saunders United States

Z.Y. Zhang United States

Dag E. Helland Norway

Bernard Bayard France

S. I. Foundling United States

David H. Ives United States

Jon R. Katze United States

G. Steinheider Germany

Charles Garrett United States

H Pelletier

1.2k ×1.7

181 ×1.0

209 ×1.4

ONCOL

146 ×1.3

GENET

34 ×0.4

IMMUN

Citations per year, relative to H Pelletier H Pelletier (= 1×) peers Charles Garrett

Countries citing papers authored by H Pelletier

Since Specialization

Citations

This map shows the geographic impact of H Pelletier's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by H Pelletier with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites H Pelletier more than expected).

Fields of papers citing papers by H Pelletier

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by H Pelletier. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by H Pelletier. The network helps show where H Pelletier may publish in the future.

Co-authorship network of co-authors of H Pelletier

This figure shows the co-authorship network connecting the top 25 collaborators of H Pelletier. A scholar is included among the top collaborators of H Pelletier based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with H Pelletier. H Pelletier is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

16 of 16 papers shown

Pelletier, H, M.R. Sawaya, Amalendra Kumar, Samuel H. Wilson, & Joseph Kraut. (1994). STRUCTURES OF TERNARY COMPLEXES OF RAT DNA POLYMERASE BETA, A DNA TEMPLATE-PRIMER, AND DDCTP. PubMed. 264(5167). 1891–903. 635 indexed citations breakdown →

Chauffert, Bruno, et al.. (1992). Use of a specific monoclonal antibody for studying the liver metastatic invasion of a rat colon cancer.. PubMed. 2(5). 301–5. 8 indexed citations

Chauffert, Bruno, H Pelletier, Éric Solary, et al.. (1990). Potential usefulness of quinine to circumvent the anthracycline resistance in clinical practice. British Journal of Cancer. 62(3). 395–397. 25 indexed citations

Pelletier, H, J Millot, Bruno Chauffert, et al.. (1990). Mechanisms of resistance of confluent human and rat colon cancer cells to anthracyclines: alteration of drug passive diffusion.. PubMed. 50(20). 6626–31. 44 indexed citations

Genne, Philippe, et al.. (1990). Serum concentrations of amiodarone required for an in vivo modulation of anthracycline resistance.. PubMed. 9(6). 1655–9. 9 indexed citations

Pelletier, H, et al.. (1989). Comparative effect of rat and fetal calf serum on measurement of the natural tumoricidal activity of rat lymphocytes, macrophages and polymorphonuclear cells. Cancer Immunology Immunotherapy. 28(1). 34–6. 3 indexed citations

Lagadec, Patricia, et al.. (1989). Involvement of effector cells in the treatment with endotoxins of peritoneal carcinomatosis induced by colon tumour cells.. PubMed. 9(2). 421–5. 3 indexed citations

Martín, Miguel, Bruno Chauffert, Anne Caignard, et al.. (1989). Histoimmunological characterization of the cellular reaction to liver metastases induced by colon cancer cells in syngeneic rats.. PubMed. 9(4). 216–30. 21 indexed citations

Pelletier, H, et al.. (1988). Differential sensitivity to natural cell-mediated cytotoxicity of two rat colon adenocarcinoma variants differing in their tumorigenicity: identification of the effector cells as natural killer cells. Cancer Immunology Immunotherapy. 26(3). 263–8. 8 indexed citations

10.

Lagadec, Patricia, et al.. (1988). In vivo and in vitro effects of fish-containing diets on colon tumour cells in rats.. PubMed. 8(2). 225–8. 3 indexed citations

11.

Jeannin, J.F., et al.. (1988). Regression induced by lentinan, of peritoneal carcinomatoses in a model of colon cancer in rat. International Journal of Immunopharmacology. 10(7). 855–861. 16 indexed citations

12.

Caignard, Anne, H Pelletier, & Florian Martin. (1988). Specificity of the immune response leading to protection or enhancement by regressive and progressive variants of a rat colon carcinoma. International Journal of Cancer. 42(6). 883–886. 36 indexed citations

13.

Shimizu, Toshio, et al.. (1987). Effects of a single injection of anti‐asialo GM1 serum on natural cytotoxicity and the growth of a regressive colonic tumor in syngeneic rats. International Journal of Cancer. 40(5). 676–680. 18 indexed citations

14.

Lagadec, Patricia, et al.. (1987). Treatment with endotoxins of peritoneal carcinomatosis induced by colon tumor cells in the rat.. PubMed. 7(2). 83–95. 15 indexed citations

15.

Martín, Miguel, Anne Caignard, Arlette Hammann, H Pelletier, & Florian Martin. (1987). An immunohistological study of cells infiltrating progressive and regressive tumors induced by two variant subpopulations of a rat colon cancer cell line. International Journal of Cancer. 40(1). 87–93. 34 indexed citations

16.

Lagadec, Patricia, et al.. (1986). [In vitro tumoricidal activity of peritoneal macrophages and splenic lymphocytes of rats under diet with or without fish].. PubMed. 180(4). 426–33. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact