H. Choritz
- Hematology top 1%
- Genetics top 1%
- Molecular Biology
- Rheumatology top 5%
- Endocrinology, Diabetes and Metabolism top 10%
- Topics
- Myeloproliferative Neoplasms: Diagnosis and Treatment (17 papers)Acute Myeloid Leukemia Research (16 papers)Eosinophilic Disorders and Syndromes (7 papers)
- Cited by
- HematologyGeneticsRheumatology
- Journals
- The American Journal of Surgical PathologyAmerican Journal of Clinical PathologyArchiv für Pathologische Anatomie und Physiologie und für Klinische Medicin
- Partner nations
- Germany
In The Last Decade
H. Choritz
32 papers receiving 1.0k citations
Peers
Comparison fields: 5 of 72
- Hematology 653
- Genetics 606
- Molecular Biology 248
- Rheumatology 192
- Endocrinology, Diabetes and Metabolism 147
Countries citing papers authored by H. Choritz
This map shows the geographic impact of H. Choritz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by H. Choritz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites H. Choritz more than expected).
Fields of papers citing papers by H. Choritz
This network shows the impact of papers produced by H. Choritz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by H. Choritz. The network helps show where H. Choritz may publish in the future.
Co-authorship network of co-authors of H. Choritz
This figure shows the co-authorship network connecting the top 25 collaborators of H. Choritz. A scholar is included among the top collaborators of H. Choritz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with H. Choritz. H. Choritz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 57 | |
| 2 | 3 | |
| 3 | 57 | |
| 4 | 14 | |
| 5 | 45 | |
| 6 | 5 | |
| 7 | 5 | |
| 8 | 84 | |
| 9 | 8 | |
| 10 | 1 | |
| 11 | 38 | |
| 12 | 70 | |
| 13 | 66 | |
| 14 | 33 | |
| 15 | 12 | |
| 16 | 17 | |
| 17 | 10 | |
| 18 | Elliptic Fourier analysis of megakaryocyte nuclei in chronic myeloproliferative disorders. | 11 |
| 19 | 21 | |
| 20 | Predictive implications of bioptic diagnosis in cardiac allografts. | 5 |
About H. Choritz
H. Choritz is a scholar working on Hematology, Genetics and Transplantation, having authored 32 papers that have together received 1.1k indexed citations. Recurring topics across this work include Myeloproliferative Neoplasms: Diagnosis and Treatment (17 papers), Acute Myeloid Leukemia Research (16 papers) and Eosinophilic Disorders and Syndromes (7 papers). The work is most often cited by research in Hematology (653 citations), Genetics (606 citations) and Rheumatology (192 citations). H. Choritz has collaborated with scholars based in Germany. Frequent co-authors include A. Georgii, T. Buhr, H. Hundeshagen, W. Lang, V. Kaloutsi, Martin Werner, H. Maschek, Hans Kreipe, Guntram Büsche and Reinhold Nafe. Their work appears in journals such as The American Journal of Surgical Pathology, American Journal of Clinical Pathology and Archiv für Pathologische Anatomie und Physiologie und für Klinische Medicin.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.