Guohua Ding

650 total citations
23 papers, 536 citations indexed

About

Guohua Ding is a scholar working on Nephrology, Molecular Biology and Genetics. According to data from OpenAlex, Guohua Ding has authored 23 papers receiving a total of 536 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Nephrology, 13 papers in Molecular Biology and 3 papers in Genetics. Recurrent topics in Guohua Ding's work include Renal Diseases and Glomerulopathies (14 papers), Chronic Kidney Disease and Diabetes (7 papers) and Renal and related cancers (4 papers). Guohua Ding is often cited by papers focused on Renal Diseases and Glomerulopathies (14 papers), Chronic Kidney Disease and Diabetes (7 papers) and Renal and related cancers (4 papers). Guohua Ding collaborates with scholars based in China, United States and United Kingdom. Guohua Ding's co-authors include Pravin C. Singhal, Nicholas Franki, Qian Yang, Madhu Bhaskaran, Krishna Reddy, Wei Liang, Yiqiong Ma, Richard M. Hays, Yingjie Yang and Xinghua Chen and has published in prestigious journals such as Scientific Reports, Molecular Biology of the Cell and American Journal of Physiology-Cell Physiology.

In The Last Decade

Guohua Ding

22 papers receiving 532 citations

Peers

Guohua Ding

NEPHR

CCM

GENET

EDM

Ana Velić Germany

Yihung Huang United States

Jili Zhu China

Nobuyuki Kajiwara Japan

Jun Feng China

Ihsan Gadi Germany

Ferhan Siddiqi Canada

Bozena Krolewski United States

Sun Ha Lee South Korea

Nikoo Alavi United States

Ana Velić Germany

Guohua Ding

406 ×1.6

271 ×1.1

NEPHR

85 ×1.0

CCM

63 ×1.1

GENET

51 ×1.0

EDM

Citations per year, relative to Guohua Ding Guohua Ding (= 1×) peers Ana Velić

Countries citing papers authored by Guohua Ding

Since Specialization

Citations

This map shows the geographic impact of Guohua Ding's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Guohua Ding with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Guohua Ding more than expected).

Fields of papers citing papers by Guohua Ding

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Guohua Ding. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Guohua Ding. The network helps show where Guohua Ding may publish in the future.

Co-authorship network of co-authors of Guohua Ding

This figure shows the co-authorship network connecting the top 25 collaborators of Guohua Ding. A scholar is included among the top collaborators of Guohua Ding based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Guohua Ding. Guohua Ding is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Huang, Xiao‐Xiao, Weiwei Li, Xinghua Chen, et al.. (2025). USP22 promotes angiotensin II-induced podocyte injury by deubiquitinating and stabilizing HMGB1. Cellular Signalling. 131. 111771–111771. 5 indexed citations

Wang, Juan, Jijia Hu, Qian Guan, et al.. (2025). APT1-derived depalmitoylation of CD36 alleviates diabetes-induced lipotoxicity in podocytes. International Journal of Biological Sciences. 21(9). 3852–3866.

Hu, Jijia, Zijing Zhu, Yiqun Hao, et al.. (2024). RBBP6‐Mediated ERRα Degradation Contributes to Mitochondrial Injury in Renal Tubular Cells in Diabetic Kidney Disease. Advanced Science. 11(46). e2405153–e2405153. 3 indexed citations

Ma, Yiqiong, Qian Yang, Wei Liang, et al.. (2018). Role of c-Abl and nephrin in podocyte cytoskeletal remodeling induced by angiotensin II. Cell Death and Disease. 9(2). 185–185. 24 indexed citations

Yang, Yingjie, Qian Yang, Jian Yang, Yiqiong Ma, & Guohua Ding. (2017). Angiotensin II induces cholesterol accumulation and injury in podocytes. Scientific Reports. 7(1). 10672–10672. 48 indexed citations

Gao, Zhao, Xinghua Chen, Kai Zhu, Ping Zeng, & Guohua Ding. (2017). Dab1 Contributes to Angiotensin II-Induced Apoptosis via p38 Signaling Pathway in Podocytes. BioMed Research International. 2017. 1–11. 13 indexed citations

Ding, Guohua, et al.. (2017). Blood purification treatment initiated at the time of sepsis diagnosis effectively attenuates serum HMGB1 upregulation and improves patient prognosis. Experimental and Therapeutic Medicine. 14(4). 3029–3035. 9 indexed citations

Ma, Yiqiong, et al.. (2016). c-Abl contributes to glucose-promoted apoptosis via p53 signaling pathway in podocytes. Diabetes Research and Clinical Practice. 113. 171–178. 9 indexed citations

Zhang, Lu, et al.. (2016). Csk regulates angiotensin II-induced podocyte apoptosis. APOPTOSIS. 21(7). 846–855. 22 indexed citations

10.

Liu, Yipeng, Wei Liang, Yingjie Yang, et al.. (2015). IQGAP1 regulates actin cytoskeleton organization in podocytes through interaction with nephrin. Cellular Signalling. 27(4). 867–877. 26 indexed citations

11.

Wang, Siyuan, Cheng Chen, Ke Su, et al.. (2015). Angiotensin II induces reorganization of the actin cytoskeleton and myosin light-chain phosphorylation in podocytes through rho/ROCK-signaling pathway*. Renal Failure. 38(2). 268–275. 23 indexed citations

12.

Yang, Qian, Yiqiong Ma, Yipeng Liu, et al.. (2015). Angiotensin II down-regulates nephrin–Akt signaling and induces podocyte injury: role of c-Abl. Molecular Biology of the Cell. 27(1). 197–208. 29 indexed citations

13.

Wan, Jianxin, Yipeng Liu, Qian Yang, et al.. (2014). sPLA2 IB induces human podocyte apoptosis via the M-type phospholipase A2 receptor. Scientific Reports. 4(1). 6660–6660. 28 indexed citations

14.

Liu, Yipeng, Wei Liang, Qian Yang, et al.. (2013). IQGAP1 Mediates Angiotensin II-Induced Apoptosis of Podocytes via the ERK1/2 MAPK Signaling Pathway. American Journal of Nephrology. 38(5). 430–444. 20 indexed citations

15.

Cheng, Hui, Cheng Chen, Siyuan Wang, Guohua Ding, & Ming Shi. (2013). The effects of urokinase-type plasminogen activator (uPA) on cell proliferation and phenotypic transformation of rat mesangial cells induced by high glucose. Diabetes Research and Clinical Practice. 103(3). 489–495. 5 indexed citations

16.

Yadav, Anju, et al.. (2012). Sirolimus modulates HIVAN phenotype through inhibition of epithelial mesenchymal transition. Experimental and Molecular Pathology. 93(1). 173–181. 10 indexed citations

17.

Liang, Wei, et al.. (2011). Angiotensin II induces nephrin dephosphorylation and podocyte injury: Role of caveolin-1. Cellular Signalling. 24(2). 443–450. 56 indexed citations

18.

Hayakawa, Tõru, et al.. (2004). Pharmacokinetic interaction of ibuprofen enantiomers in rabbits. Journal of Pharmacy and Pharmacology. 56(3). 317–321. 7 indexed citations

19.

Franki, Nicholas, Guohua Ding, Yang Gao, & Richard M. Hays. (1992). Effect of cytochalasin D on the actin cytoskeleton of the toad bladder epithelial cell. American Journal of Physiology-Cell Physiology. 263(5). C995–C1000. 44 indexed citations

20.

Ding, Guohua, Nicholas Franki, J. Bourguet, & Richard M. Hays. (1988). Role of vesicular transport in ADH-stimulated aggregate delivery. American Journal of Physiology-Cell Physiology. 255(5). C641–C652. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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