Guillermo Dı́az-Araya

3.0k total citations
88 papers, 2.4k citations indexed

About

Guillermo Dı́az-Araya is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Cancer Research. According to data from OpenAlex, Guillermo Dı́az-Araya has authored 88 papers receiving a total of 2.4k indexed citations (citations by other indexed papers that have themselves been cited), including 55 papers in Cardiology and Cardiovascular Medicine, 37 papers in Molecular Biology and 17 papers in Cancer Research. Recurrent topics in Guillermo Dı́az-Araya's work include Cardiac Fibrosis and Remodeling (36 papers), Peptidase Inhibition and Analysis (12 papers) and Protease and Inhibitor Mechanisms (10 papers). Guillermo Dı́az-Araya is often cited by papers focused on Cardiac Fibrosis and Remodeling (36 papers), Peptidase Inhibition and Analysis (12 papers) and Protease and Inhibitor Mechanisms (10 papers). Guillermo Dı́az-Araya collaborates with scholars based in Chile, United States and Spain. Guillermo Dı́az-Araya's co-authors include Sergio Lavandero, Raúl Vivar, Mario Chiong, Claudio Humeres, Miguel Copaja, Lorena Garcı́a, Pía Boza, Claudia Muñoz, Samir Bolívar and Pablo Castro and has published in prestigious journals such as Circulation, Biochemical and Biophysical Research Communications and FEBS Letters.

In The Last Decade

Guillermo Dı́az-Araya

87 papers receiving 2.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Guillermo Dı́az-Araya Chile 29 948 935 306 298 254 88 2.4k
Vijayakumar Sukumaran Japan 26 736 0.8× 468 0.5× 372 1.2× 190 0.6× 221 0.9× 43 2.0k
Yu‐Hsun Kao Taiwan 29 1.1k 1.1× 1.2k 1.3× 338 1.1× 412 1.4× 183 0.7× 131 2.8k
Tiziana Bachetti Italy 23 857 0.9× 1.1k 1.2× 245 0.8× 202 0.7× 282 1.1× 56 2.5k
Keiko Maeda Japan 29 746 0.8× 1.7k 1.8× 312 1.0× 653 2.2× 306 1.2× 114 3.5k
Subha Sen United States 29 1.0k 1.1× 1.5k 1.6× 463 1.5× 284 1.0× 180 0.7× 60 3.1k
Pierre Sicard France 28 853 0.9× 747 0.8× 135 0.4× 289 1.0× 225 0.9× 93 2.2k
Ken‐ichi Aihara Japan 29 813 0.9× 561 0.6× 682 2.2× 453 1.5× 215 0.8× 117 2.8k
Alessandro Cannavò Italy 32 1.2k 1.2× 873 0.9× 377 1.2× 246 0.8× 216 0.9× 74 2.6k
Yasukatsu Izumi Japan 33 1.7k 1.8× 1.1k 1.2× 298 1.0× 569 1.9× 178 0.7× 87 3.5k
Dongqi Xing United States 25 903 1.0× 518 0.6× 397 1.3× 259 0.9× 167 0.7× 53 2.3k

Countries citing papers authored by Guillermo Dı́az-Araya

Since Specialization
Citations

This map shows the geographic impact of Guillermo Dı́az-Araya's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Guillermo Dı́az-Araya with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Guillermo Dı́az-Araya more than expected).

Fields of papers citing papers by Guillermo Dı́az-Araya

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Guillermo Dı́az-Araya. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Guillermo Dı́az-Araya. The network helps show where Guillermo Dı́az-Araya may publish in the future.

Co-authorship network of co-authors of Guillermo Dı́az-Araya

This figure shows the co-authorship network connecting the top 25 collaborators of Guillermo Dı́az-Araya. A scholar is included among the top collaborators of Guillermo Dı́az-Araya based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Guillermo Dı́az-Araya. Guillermo Dı́az-Araya is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Catalán, Mabel, Juan Diego Maya, Óscar Lorenzo, et al.. (2024). Boldine prevents the inflammatory response of cardiac fibroblasts induced by SGK1-NFκB signaling pathway activation. Cellular Signalling. 120. 111241–111241. 3 indexed citations
2.
Valencia, Inés, Fernando de la Cuesta, Guillermo Dı́az-Araya, et al.. (2024). SARS-CoV-2 S Protein Reduces Cytoprotective Defenses and Promotes Human Endothelial Cell Senescence. Aging and Disease. 16(3). 0–0. 4 indexed citations
3.
Catalán, Mabel, et al.. (2023). Lipoxin A4 prevents high glucose-induced inflammatory response in cardiac fibroblast through FOXO1 inhibition. Cellular Signalling. 106. 110657–110657. 2 indexed citations
4.
Dı́az-Araya, Guillermo, et al.. (2023). Exposure of the Gestating Mother to Sympathetic Stress Modifies the Cardiovascular Function of the Progeny in Male Rats. International Journal of Environmental Research and Public Health. 20(5). 4285–4285. 1 indexed citations
5.
Catalán, Mabel, et al.. (2023). SGK1 is necessary to FoxO3a negative regulation, oxidative stress and cardiac fibroblast activation induced by TGF-β1. Cellular Signalling. 109. 110778–110778. 9 indexed citations
6.
Romero, Alejandra, et al.. (2022). Resolvin E1 attenuates doxorubicin-induced endothelial senescence by modulating NLRP3 inflammasome activation. Biochemical Pharmacology. 201. 115078–115078. 16 indexed citations
7.
Kemmerling, Ulrike, et al.. (2021). Aspirin-triggered resolvin D1 reduces parasitic cardiac load by decreasing inflammation in a murine model of early chronic Chagas disease. PLoS neglected tropical diseases. 15(11). e0009978–e0009978. 12 indexed citations
8.
Letelier, A, et al.. (2020). TGF-β1 decreases CHOP expression and prevents cardiac fibroblast apoptosis induced by endoplasmic reticulum stress. Toxicology in Vitro. 70. 105041–105041. 10 indexed citations
9.
Vivar, Raúl, Claudio Humeres, Samir Bolívar, et al.. (2020). Role of FoxO3a as a negative regulator of the cardiac myofibroblast conversion induced by TGF-β1. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1867(7). 118695–118695. 21 indexed citations
10.
11.
Zapata‐Torres, Gerald, Jorge Hidalgo, Pedro Ayala, et al.. (2014). A novel dihydropyridine with 3-aryl meta-hydroxyl substitution blocks L-type calcium channels in rat cardiomyocytes. Toxicology and Applied Pharmacology. 279(1). 53–62. 6 indexed citations
12.
Vivar, Raúl, Claudio Humeres, Pedro Ayala, et al.. (2013). TGF-β1 prevents simulated ischemia/reperfusion-induced cardiac fibroblast apoptosis by activation of both canonical and non-canonical signaling pathways. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1832(6). 754–762. 45 indexed citations
13.
Ibacache, Mauricio, Gina Sánchez, Zully Pedrozo, et al.. (2011). Dexmedetomidine preconditioning activates pro-survival kinases and attenuates regional ischemia/reperfusion injury in rat heart. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1822(4). 537–545. 85 indexed citations
14.
15.
Greig, Douglas, Mario Chiong, Hugo Verdejo, et al.. (2008). Serum Uric Acid Correlates with Extracellular Superoxide Dismutase Activity in Patients with Chronic Heart Failure. European Journal of Heart Failure. 10(7). 646–651. 35 indexed citations
16.
Ocaranza, María Paz, Jorge Jalil, Carlos P. Vío, et al.. (2008). Early expression of monocyte chemoattractant protein-1 correlates with the onset of isoproterenol-induced cardiac fibrosis in rats with distinct angiotensin-converting enzyme polymorphism. Journal of the Renin-Angiotensin-Aldosterone System. 9(3). 154–162. 8 indexed citations
17.
Eisner, Verónica, Alfredo Criollo, Clara Quiroga, et al.. (2006). Hyperosmotic stress‐dependent NFκB activation is regulated by reactive oxygen species and IGF‐1 in cultured cardiomyocytes. FEBS Letters. 580(18). 4495–4500. 35 indexed citations
18.
Eisner, Verónica, Clara Quiroga, Alfredo Criollo, et al.. (2006). Hyperosmotic stress activates p65/RelB NFκB in cultured cardiomyocytes with dichotomic actions on caspase activation and cell death. FEBS Letters. 580(14). 3469–3476. 15 indexed citations
19.
Castro, Pablo, Mario Chiong, Guillermo Dı́az-Araya, et al.. (2005). Effects of Carvedilol On Oxidative Stress and Chronotropic Response to Exercise in Patients with Chronic Heart Failure. European Journal of Heart Failure. 7(6). 1033–1039. 33 indexed citations
20.
Moraga, Francisco, et al.. (2004). Oxidative stress in pericardial fluid and plasma and its association with ventricular function. International Journal of Cardiology. 101(2). 197–201. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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