Guido Palamara
- Epidemiology top 10%
- Infectious Diseases top 5%
- Virology top 5%
- Surgery
- Cell Biology
- Co-authors
- Massimo GiulianiA. Di CarloDomenico MorettoR. MercantiniAlessandra LatiniGiovanni RezzaCarlo Federico PernoGrazia Prignano
- Topics
- HIV Research and Treatment (11 papers)HIV/AIDS Research and Interventions (7 papers)HIV/AIDS drug development and treatment (7 papers)
- Journals
- SHILAP Revista de lepidopterologíaPLoS ONEJournal of Virology
- Partner nations
- ItalyUnited StatesCzechia
In The Last Decade
Guido Palamara
27 papers receiving 539 citations
Peers
Comparison fields: 5 of 73
- Epidemiology 296
- Infectious Diseases 263
- Virology 198
- Surgery 76
- Cell Biology 73
Countries citing papers authored by Guido Palamara
This map shows the geographic impact of Guido Palamara's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Guido Palamara with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Guido Palamara more than expected).
Fields of papers citing papers by Guido Palamara
This network shows the impact of papers produced by Guido Palamara. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Guido Palamara. The network helps show where Guido Palamara may publish in the future.
Co-authorship network of co-authors of Guido Palamara
This figure shows the co-authorship network connecting the top 25 collaborators of Guido Palamara. A scholar is included among the top collaborators of Guido Palamara based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Guido Palamara. Guido Palamara is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 6 | |
| 2 | 10 | |
| 3 | 53 | |
| 4 | 25 | |
| 5 | 14 | |
| 6 | 3 | |
| 7 | 13 | |
| 8 | 38 | |
| 9 | CD81 expression on CD19+ peripheral blood lymphocytes is associated with chronic HCV disease and increased risk for HCV infection: a putative role for inflammatory cytokines. | 13 |
| 10 | 15 | |
| 11 | 20 | |
| 12 | 22 | |
| 13 | 25 | |
| 14 | 18 | |
| 15 | 34 | |
| 16 | 55 | |
| 17 | 3 | |
| 18 | 6 | |
| 19 | 30 | |
| 20 | 3 |
About Guido Palamara
Guido Palamara is a scholar working on Virology, Infectious Diseases and Epidemiology, having authored 27 papers that have together received 549 indexed citations. Recurring topics across this work include HIV Research and Treatment (11 papers), HIV/AIDS Research and Interventions (7 papers) and HIV/AIDS drug development and treatment (7 papers). The work is most often cited by research in Virology (198 citations), Infectious Diseases (263 citations) and Epidemiology (296 citations). Guido Palamara has collaborated with scholars based in Italy, United States and Czechia. Frequent co-authors include Massimo Giuliani, A. Di Carlo, Domenico Moretto, R. Mercantini, Alessandra Latini, Giovanni Rezza, Carlo Federico Perno, Grazia Prignano, F. Caprilli and Maria Mercedes Santoro. Their work appears in journals such as SHILAP Revista de lepidopterología, PLoS ONE and Journal of Virology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.