Guangyan Miao

1.5k total citations · 1 hit paper
13 papers, 1.1k citations indexed

About

Guangyan Miao is a scholar working on Epidemiology, Cell Biology and Physiology. According to data from OpenAlex, Guangyan Miao has authored 13 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Epidemiology, 7 papers in Cell Biology and 4 papers in Physiology. Recurrent topics in Guangyan Miao's work include Autophagy in Disease and Therapy (9 papers), Endoplasmic Reticulum Stress and Disease (5 papers) and Lysosomal Storage Disorders Research (3 papers). Guangyan Miao is often cited by papers focused on Autophagy in Disease and Therapy (9 papers), Endoplasmic Reticulum Stress and Disease (5 papers) and Lysosomal Storage Disorders Research (3 papers). Guangyan Miao collaborates with scholars based in China, United States and Japan. Guangyan Miao's co-authors include Hong Zhang, Yong Chen, Yan Zhao, Hongyu Zhao, Du Feng, Hong Zhang, Nan Liu, Yi Shi, Ming-Ming Ji and Yuhai Bi and has published in prestigious journals such as Cell, Molecular Cell and PLoS ONE.

In The Last Decade

Guangyan Miao

13 papers receiving 1.1k citations

Hit Papers

ORF3a of the COVID-19 virus SARS-CoV-2 blocks HOPS comple... 2020 2026 2022 2024 2020 50 100 150 200 250

Peers

Guangyan Miao
Guangyan Miao
Citations per year, relative to Guangyan Miao Guangyan Miao (= 1×) peers Aliaksandr Khaminets

Countries citing papers authored by Guangyan Miao

Since Specialization
Citations

This map shows the geographic impact of Guangyan Miao's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Guangyan Miao with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Guangyan Miao more than expected).

Fields of papers citing papers by Guangyan Miao

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Guangyan Miao. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Guangyan Miao. The network helps show where Guangyan Miao may publish in the future.

Co-authorship network of co-authors of Guangyan Miao

This figure shows the co-authorship network connecting the top 25 collaborators of Guangyan Miao. A scholar is included among the top collaborators of Guangyan Miao based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Guangyan Miao. Guangyan Miao is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
Miao, Guangyan, Tina M. Fortier, Haibo Liu, et al.. (2025). Microglia promote inflammatory cell death upon neuronal mitochondrial impairment during neurodegeneration. Nature Structural & Molecular Biology. 32(10). 2046–2059. 2 indexed citations
2.
Wang, Ruoxi, Tina M. Fortier, Guangyan Miao, et al.. (2023). PINK1, Keap1, and Rtnl1 regulate selective clearance of endoplasmic reticulum during development. Cell. 186(19). 4172–4188.e18. 28 indexed citations
3.
Wang, Ruoxi, Guangyan Miao, James Shen, Tina M. Fortier, & Eric H. Baehrecke. (2022). ESCRT dysfunction compromises endoplasmic reticulum maturation and autophagosome biogenesis in Drosophila. Current Biology. 32(6). 1262–1274.e4. 12 indexed citations
4.
Miao, Guangyan, Hongyu Zhao, Yan Li, et al.. (2020). ORF3a of the COVID-19 virus SARS-CoV-2 blocks HOPS complex-mediated assembly of the SNARE complex required for autolysosome formation. Developmental Cell. 56(4). 427–442.e5. 261 indexed citations breakdown →
5.
Li, Dongfang, Yan Zhao, Di Li, et al.. (2019). The ER-Localized Protein DFCP1 Modulates ER-Lipid Droplet Contact Formation. Cell Reports. 27(2). 343–358.e5. 87 indexed citations
6.
Miao, Guangyan, Yujie Zhang, Di Chen, & Hong Zhang. (2019). The ER-Localized Transmembrane Protein TMEM39A/SUSR2 Regulates Autophagy by Controlling the Trafficking of the PtdIns(4)P Phosphatase SAC1. Molecular Cell. 77(3). 618–632.e5. 37 indexed citations
7.
Zhao, Yan, Nan Liu, Guangyan Miao, et al.. (2018). The ER Contact Proteins VAPA/B Interact with Multiple Autophagy Proteins to Modulate Autophagosome Biogenesis. Current Biology. 28(8). 1234–1245.e4. 141 indexed citations
8.
Zhao, Yan, Yong Chen, Guangyan Miao, et al.. (2017). The ER-Localized Transmembrane Protein EPG-3/VMP1 Regulates SERCA Activity to Control ER-Isolation Membrane Contacts for Autophagosome Formation. Molecular Cell. 67(6). 974–989.e6. 169 indexed citations
9.
Zheng, Wang, Guangyan Miao, Xue Xue, et al.. (2016). The Vici Syndrome Protein EPG5 Is a Rab7 Effector that Determines the Fusion Specificity of Autophagosomes with Late Endosomes/Lysosomes. Molecular Cell. 63(5). 781–795. 232 indexed citations
10.
Miao, Guangyan, Yan Zhao, Hongyu Zhao, et al.. (2016). Mice deficient in the Vici syndrome gene Epg5 exhibit features of retinitis pigmentosa. Autophagy. 12(12). 2263–2270. 22 indexed citations
11.
Zhang, Chengyi, et al.. (2016). Convergence on successive over-relaxed iterative methods for non-Hermitian positive definite linear systems. Journal of Inequalities and Applications. 2016(1). 1 indexed citations
12.
Zhao, Yan, Le Sun, Guangyan Miao, et al.. (2015). The autophagy geneWdr45/Wipi4regulates learning and memory function and axonal homeostasis. Autophagy. 11(6). 881–890. 111 indexed citations
13.
Zhao, Yuanbo, Jia Hu, Guangyan Miao, et al.. (2013). Transmembrane Protein 208: A Novel ER-Localized Protein That Regulates Autophagy and ER Stress. PLoS ONE. 8(5). e64228–e64228. 23 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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