Guang Wang

661 total citations
42 papers, 467 citations indexed

About

Guang Wang is a scholar working on Molecular Biology, Epidemiology and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, Guang Wang has authored 42 papers receiving a total of 467 indexed citations (citations by other indexed papers that have themselves been cited), including 26 papers in Molecular Biology, 8 papers in Epidemiology and 7 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in Guang Wang's work include Glutathione Transferases and Polymorphisms (7 papers), Thyroid Disorders and Treatments (5 papers) and Liver Disease Diagnosis and Treatment (4 papers). Guang Wang is often cited by papers focused on Glutathione Transferases and Polymorphisms (7 papers), Thyroid Disorders and Treatments (5 papers) and Liver Disease Diagnosis and Treatment (4 papers). Guang Wang collaborates with scholars based in China, United Kingdom and United States. Guang Wang's co-authors include Jia Liu, Xuesong Yang, Manli Chuai, Xin Cheng, Ying Wang, KK Lee, Xiaoyu Wang, Shuai Li, Liu Cao and Ziheng Chen and has published in prestigious journals such as Nucleic Acids Research, The Journal of Clinical Endocrinology & Metabolism and Journal of Agricultural and Food Chemistry.

In The Last Decade

Guang Wang

39 papers receiving 460 citations

Peers

Guang Wang
Comparison fields: 5 of 87
  • Molecular Biology 165
  • Pulmonary and Respiratory Medicine 84
  • Epidemiology 73
  • Endocrinology, Diabetes and Metabolism 69
  • Surgery 67
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Lara Valiño‐Rivas Spain
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Macarena Orejudo Spain View profile →
Citations per field, relative to Guang Wang
Guang Wang · 1×
Citations per year, relative to Guang Wang
Guang Wang · 1×

Countries citing papers authored by Guang Wang

Since Specialization
Citations

This map shows the geographic impact of Guang Wang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Guang Wang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Guang Wang more than expected).

Fields of papers citing papers by Guang Wang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Guang Wang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Guang Wang. The network helps show where Guang Wang may publish in the future.

Co-authorship network of co-authors of Guang Wang

This figure shows the co-authorship network connecting the top 25 collaborators of Guang Wang. A scholar is included among the top collaborators of Guang Wang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Guang Wang. Guang Wang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Work Indexed citations
1 0
2 4
3 0
4 9
5 1
6 12
7 4
8 2
9 40
10 1
11 6
12 4
13 53
14 19
15 11
16 18
17 17
18 43
19
GSTM1 gene polymorphism and lung cancer susceptibility in Man population
3
20
Susceptibility of hepatocellular carcinoma with the polymorphism of CYP2C19 genes
1

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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