Gréta Kis

560 total citations
23 papers, 455 citations indexed

About

Gréta Kis is a scholar working on Physiology, Cellular and Molecular Neuroscience and Molecular Biology. According to data from OpenAlex, Gréta Kis has authored 23 papers receiving a total of 455 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Physiology, 9 papers in Cellular and Molecular Neuroscience and 8 papers in Molecular Biology. Recurrent topics in Gréta Kis's work include Pain Mechanisms and Treatments (6 papers), Neuroscience and Neuropharmacology Research (5 papers) and PARP inhibition in cancer therapy (5 papers). Gréta Kis is often cited by papers focused on Pain Mechanisms and Treatments (6 papers), Neuroscience and Neuropharmacology Research (5 papers) and PARP inhibition in cancer therapy (5 papers). Gréta Kis collaborates with scholars based in Hungary, United States and Germany. Gréta Kis's co-authors include Miklós Antal, Krisztina Holló, Zoltán Hegyi, Péter Bai, Lilla Nagy, Catherine Ledent, Judit Márton, András Vida, Krisztina Hegedűs and L Jankó and has published in prestigious journals such as PLoS ONE, The Journal of Comparative Neurology and International Journal of Molecular Sciences.

In The Last Decade

Gréta Kis

23 papers receiving 452 citations

Peers

Gréta Kis
Jennifer Brelsfoard United States
Meeyul Hwang South Korea
Elizabeth Snella United States
Gréta Kis
Citations per year, relative to Gréta Kis Gréta Kis (= 1×) peers Honglin Yan

Countries citing papers authored by Gréta Kis

Since Specialization
Citations

This map shows the geographic impact of Gréta Kis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gréta Kis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gréta Kis more than expected).

Fields of papers citing papers by Gréta Kis

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gréta Kis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gréta Kis. The network helps show where Gréta Kis may publish in the future.

Co-authorship network of co-authors of Gréta Kis

This figure shows the co-authorship network connecting the top 25 collaborators of Gréta Kis. A scholar is included among the top collaborators of Gréta Kis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Gréta Kis. Gréta Kis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Hegedűs, Krisztina, et al.. (2023). Synaptic Targets of Glycinergic Neurons in Laminae I–III of the Spinal Dorsal Horn. International Journal of Molecular Sciences. 24(8). 6943–6943. 5 indexed citations
2.
Hegedűs, Krisztina, Gréta Kis, Roland Takács, et al.. (2023). Neuronal P2X4 receptor may contribute to peripheral inflammatory pain in rat spinal dorsal horn. Frontiers in Molecular Neuroscience. 16. 1115685–1115685. 5 indexed citations
3.
Gönczi, Mónika, Zsolt Ráduly, László Szabó, et al.. (2022). Septin7 is indispensable for proper skeletal muscle architecture and function. eLife. 11. 13 indexed citations
4.
Debreceni, Ildikó Beke, Gréta Kis, János Kappelmayer, et al.. (2022). Activation mechanism dependent surface exposure of cellular factor XIII on activated platelets and platelet microparticles. Journal of Thrombosis and Haemostasis. 20(5). 1223–1235. 20 indexed citations
5.
Ujlaki, Gyula, Szilárd Póliska, Gréta Kis, et al.. (2022). Tissue Transglutaminase Knock-Out Preadipocytes and Beige Cells of Epididymal Fat Origin Possess Decreased Mitochondrial Functions Required for Thermogenesis. International Journal of Molecular Sciences. 23(9). 5175–5175. 2 indexed citations
6.
Jankó, L, Tündé Kovàcs, Zsanett Sári, et al.. (2021). Silencing of Poly(ADP-Ribose) Polymerase-2 Induces Mitochondrial Reactive Species Production and Mitochondrial Fragmentation. Cells. 10(6). 1387–1387. 7 indexed citations
7.
Szabó, Gábor, Miklós Fagyas, Tünde Tarr, et al.. (2020). Distinct and overlapping effects of β2-glycoprotein I conformational variants in ligand interactions and functional assays. Journal of Immunological Methods. 487. 112877–112877. 5 indexed citations
8.
Sztretye, Mónika, et al.. (2020). The Role of Orai1 in Regulating Sarcoplasmic Calcium Release, Mitochondrial Morphology and Function in Myostatin Deficient Skeletal Muscle. Frontiers in Physiology. 11. 601090–601090. 4 indexed citations
9.
Jakab, Ágnes, Edina Baranyai, Lajos Daróczi, et al.. (2020). Rare earth element sequestration by Aspergillus oryzae biomass. Environmental Technology. 42(24). 3725–3735. 9 indexed citations
10.
Nagy, Lilla, Gyula Ujlaki, Gréta Kis, et al.. (2019). Olaparib induces browning of in vitro cultures of human primary white adipocytes. Biochemical Pharmacology. 167. 76–85. 21 indexed citations
11.
Hegedűs, Csaba, Gábor Boros, Gréta Kis, et al.. (2019). PARP1 Inhibition Augments UVB-Mediated Mitochondrial Changes—Implications for UV-Induced DNA Repair and Photocarcinogenesis. Cancers. 12(1). 5–5. 37 indexed citations
12.
Márton, Judit, Tamás Fodor, Lilla Nagy, et al.. (2018). PARP10 (ARTD10) modulates mitochondrial function. PLoS ONE. 13(1). e0187789–e0187789. 45 indexed citations
13.
Kis, Gréta, et al.. (2018). Macrophages engulf apoptotic and primary necrotic thymocytes through similar phosphatidylserine‐dependent mechanisms. FEBS Open Bio. 9(3). 446–456. 25 indexed citations
14.
Sztretye, Mónika, János Vincze, Tamás Oláh, et al.. (2017). SOCE Is Important for Maintaining Sarcoplasmic Calcium Content and Release in Skeletal Muscle Fibers. Biophysical Journal. 113(11). 2496–2507. 27 indexed citations
15.
Holló, Krisztina, Zoltán Hegyi, Krisztina Hegedűs, et al.. (2017). Interleukin-1 receptor type 1 is overexpressed in neurons but not in glial cells within the rat superficial spinal dorsal horn in complete Freund adjuvant-induced inflammatory pain. Journal of Neuroinflammation. 14(1). 125–125. 26 indexed citations
16.
Nagy, Lilla, Judit Márton, András Vida, et al.. (2017). Glycogen phosphorylase inhibition improves beta cell function. British Journal of Pharmacology. 175(2). 301–319. 38 indexed citations
17.
Hegyi, Zoltán, et al.. (2014). Selective axonal and glial distribution of monoacylglycerol lipase immunoreactivity in the superficial spinal dorsal horn of rodents. Brain Structure and Function. 220(5). 2625–2637. 10 indexed citations
18.
Kish, Gary, et al.. (2012). Computer‐assisted learning in anatomy at the International Medical School in Debrecen, Hungary: A preliminary report. Anatomical Sciences Education. 6(1). 42–47. 16 indexed citations
19.
Hegyi, Zoltán, Krisztina Holló, Gréta Kis, Ken Mackie, & Miklós Antal. (2012). Differential distribution of diacylglycerol lipase‐alpha and N‐acylphosphatidylethanolamine‐specific phospholipase d immunoreactivity in the superficial spinal dorsal horn of rats. Glia. 60(9). 1316–1329. 27 indexed citations
20.
Hegyi, Zoltán, Gréta Kis, Krisztina Holló, Catherine Ledent, & Miklós Antal. (2009). Neuronal and glial localization of the cannabinoid‐1 receptor in the superficial spinal dorsal horn of the rodent spinal cord. European Journal of Neuroscience. 30(2). 251–262. 46 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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