Gloria E. Mao

411 total citations
9 papers, 329 citations indexed

About

Gloria E. Mao is a scholar working on Molecular Biology, Genetics and Biochemistry. According to data from OpenAlex, Gloria E. Mao has authored 9 papers receiving a total of 329 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 4 papers in Genetics and 4 papers in Biochemistry. Recurrent topics in Gloria E. Mao's work include Retinoids in leukemia and cellular processes (7 papers), Antioxidant Activity and Oxidative Stress (4 papers) and Estrogen and related hormone effects (4 papers). Gloria E. Mao is often cited by papers focused on Retinoids in leukemia and cellular processes (7 papers), Antioxidant Activity and Oxidative Stress (4 papers) and Estrogen and related hormone effects (4 papers). Gloria E. Mao collaborates with scholars based in United States, India and United Kingdom. Gloria E. Mao's co-authors include Michael D. Collins, Howard I. Scher, Jean B. deKernion, Guido Dalbagni, Carlos Cordon‐Cardo, Zuo‐Feng Zhang, Victor E. Reuter, Jianyu Rao, Fadila Derguini and Victor E. Reuter and has published in prestigious journals such as Journal of Nutrition, The Annual Review of Pharmacology and Toxicology and Toxicology and Applied Pharmacology.

In The Last Decade

Gloria E. Mao

9 papers receiving 320 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Gloria E. Mao United States 7 239 74 58 36 26 9 329
Patrick W. Turk United States 7 397 1.7× 57 0.8× 19 0.3× 50 1.4× 31 1.2× 9 545
Valeria Simonelli Italy 10 217 0.9× 33 0.4× 8 0.1× 13 0.4× 36 1.4× 15 357
Brenda L. Court United States 4 251 1.1× 37 0.5× 9 0.2× 38 1.1× 13 0.5× 5 437
Jessica Liao United States 4 255 1.1× 118 1.6× 8 0.1× 27 0.8× 25 1.0× 5 354
Kanur Srinivasan United States 6 324 1.4× 71 1.0× 6 0.1× 24 0.7× 5 0.2× 7 392
Emery Di Cicco Italy 16 141 0.6× 37 0.5× 7 0.1× 11 0.3× 9 0.3× 21 428
Lorena Peiró-Chova Spain 12 367 1.5× 52 0.7× 4 0.1× 18 0.5× 15 0.6× 17 521
Fabiana Vaccina Italy 8 134 0.6× 52 0.7× 5 0.1× 17 0.5× 94 3.6× 13 437
Debra Q Y Quek Singapore 8 230 1.0× 26 0.4× 6 0.1× 25 0.7× 11 0.4× 11 412
Xiaoke Huang China 9 229 1.0× 36 0.5× 5 0.1× 10 0.3× 10 0.4× 11 367

Countries citing papers authored by Gloria E. Mao

Since Specialization
Citations

This map shows the geographic impact of Gloria E. Mao's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gloria E. Mao with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gloria E. Mao more than expected).

Fields of papers citing papers by Gloria E. Mao

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gloria E. Mao. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gloria E. Mao. The network helps show where Gloria E. Mao may publish in the future.

Co-authorship network of co-authors of Gloria E. Mao

This figure shows the co-authorship network connecting the top 25 collaborators of Gloria E. Mao. A scholar is included among the top collaborators of Gloria E. Mao based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Gloria E. Mao. Gloria E. Mao is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

9 of 9 papers shown
1.
2.
Mao, Gloria E., Qing‐Yi Lu, Wei Cao, et al.. (2004). Glutathione S-transferase P1 Ile105Val polymorphism, cigarette smoking and prostate cancer. Cancer Detection and Prevention. 28(5). 368–374. 34 indexed citations
3.
Mao, Gloria E., Victor E. Reuter, Carlos Cordon‐Cardo, et al.. (2004). Decreased Retinoid X Receptor-α Protein Expression in Basal Cells Occurs in the Early Stage of Human Prostate Cancer Development. Cancer Epidemiology Biomarkers & Prevention. 13(3). 383–390. 17 indexed citations
4.
Mao, Gloria E., Victor E. Reuter, Carlos Cordon‐Cardo, et al.. (2004). Decreased retinoid X receptor-alpha protein expression in basal cells occurs in the early stage of human prostate cancer development.. PubMed. 13(3). 383–90. 19 indexed citations
5.
Martin, Lisa J., et al.. (2003). Effect of arsenite, maternal age, and embryonic sex on spina bifida, exencephaly, and resorption rates in the splotch mouse. Birth Defects Research Part A Clinical and Molecular Teratology. 67(4). 231–239. 13 indexed citations
6.
Santos‐Guzmán, Jesús, Gloria E. Mao, Marian E. Swendseid, et al.. (2003). Antagonism of Hypervitaminosis A-Induced Anterior Neural Tube Closure Defects with a Methyl-Donor Deficiency in Murine Whole-Embryo Culture. Journal of Nutrition. 133(11). 3561–3570. 9 indexed citations
7.
8.
Mao, Gloria E., Michael D. Collins, & Fadila Derguini. (2000). Teratogenicity, Tissue Distribution, and Metabolism of the retro-Retinoids, 14-Hydroxy-4,14-retro-retinol and Anhydroretinol, in the C57BL/6J Mouse. Toxicology and Applied Pharmacology. 163(1). 38–49. 8 indexed citations
9.
Collins, Michael D. & Gloria E. Mao. (1999). Teratology of Retinoids. The Annual Review of Pharmacology and Toxicology. 39(1). 399–430. 224 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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