GL Logue

413 total citations
14 papers, 318 citations indexed

About

GL Logue is a scholar working on Immunology, Hematology and Genetics. According to data from OpenAlex, GL Logue has authored 14 papers receiving a total of 318 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Immunology, 6 papers in Hematology and 5 papers in Genetics. Recurrent topics in GL Logue's work include Complement system in diseases (5 papers), Immunodeficiency and Autoimmune Disorders (5 papers) and Blood groups and transfusion (5 papers). GL Logue is often cited by papers focused on Complement system in diseases (5 papers), Immunodeficiency and Autoimmune Disorders (5 papers) and Blood groups and transfusion (5 papers). GL Logue collaborates with scholars based in United States. GL Logue's co-authors include Pradip K. Rustagi, JW Fay, M S Currie, Amir Tanay, Gerald Schiffman and G D Ross and has published in prestigious journals such as Journal of Clinical Investigation, Blood and The Journal of Immunology.

In The Last Decade

GL Logue

14 papers receiving 288 citations

Peers

GL Logue

IMMUN

GENET

HEMAT

ONCOL

GENET

Andrews Df United States

WC Moloney United States

Bishan S. Charak United States

D G Nathan United States

Shulman Hm United States

G. J. M. Lafeber Netherlands

AP Sheridan United States

Colin Story Australia

Gianni Perla Italy

G Sogno Italy

Andrews Df United States

GL Logue

118 ×0.6

IMMUN

82 ×0.5

GENET

175 ×1.9

HEMAT

78 ×1.2

ONCOL

75 ×1.3

GENET

Citations per year, relative to GL Logue GL Logue (= 1×) peers Andrews Df

Countries citing papers authored by GL Logue

Since Specialization

Citations

This map shows the geographic impact of GL Logue's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by GL Logue with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites GL Logue more than expected).

Fields of papers citing papers by GL Logue

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by GL Logue. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by GL Logue. The network helps show where GL Logue may publish in the future.

Co-authorship network of co-authors of GL Logue

This figure shows the co-authorship network connecting the top 25 collaborators of GL Logue. A scholar is included among the top collaborators of GL Logue based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with GL Logue. GL Logue is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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14 of 14 papers shown

1.

Logue, GL, et al.. (1993). Autoimmune neutropenia [see comments]. Blood. 81(8). 1984–1995. 74 indexed citations

2.

Logue, GL, et al.. (1993). Autoimmune neutropenia [see comments]. Blood. 81(8). 1984–1995. 71 indexed citations

3.

Logue, GL, et al.. (1988). Effect of RBCs on the activation of human complement by heparin- protamine complexes. Blood. 71(1). 36–40. 7 indexed citations

4.

Ross, G D, et al.. (1988). Effect of antigen site and complement receptor status on the rate of cleavage of C3c antigen from red cell bound C3b. Blood. 71(3). 786–790. 1 indexed citations

5.

Ross, G D, et al.. (1988). Effect of antigen site and complement receptor status on the rate of cleavage of C3c antigen from red cell bound C3b. Blood. 71(3). 786–790. 4 indexed citations

6.

Logue, GL, et al.. (1987). Antibodies to granulocyte precursors in selective myeloid hypoplasia and other suspected autoimmune neutropenias: use of HL-60 cells as targets. Blood. 69(2). 529–536. 14 indexed citations

7.

Logue, GL, et al.. (1987). Antibodies to granulocyte precursors in selective myeloid hypoplasia and other suspected autoimmune neutropenias: use of HL-60 cells as targets. Blood. 69(2). 529–536. 14 indexed citations

8.

Tanay, Amir, et al.. (1984). Use of total lymphoid irradiation (TLI) in studies of the T cell-dependence of autoantibody production in rheumatoid arthritis.. The Journal of Immunology. 132(2). 1036–1040. 14 indexed citations

9.

Rustagi, Pradip K., M S Currie, & GL Logue. (1982). Activation of human complement by immunoglobulin G antigranulocyte antibody.. Journal of Clinical Investigation. 70(6). 1137–1147. 20 indexed citations

10.

Fay, JW, et al.. (1979). Leukopheresis therapy of leukemic reticuloendotheliosis (hairy cell leukemia). Blood. 54(3). 747–749. 27 indexed citations

11.

Fay, JW, et al.. (1979). Leukopheresis therapy of leukemic reticuloendotheliosis (hairy cell leukemia). Blood. 54(3). 747–749. 1 indexed citations

12.

Logue, GL, et al.. (1978). Antibody-dependent lymphocyte-mediated granulocyte cytotoxicity in man. Blood. 51(1). 97–108. 27 indexed citations

13.

Logue, GL. (1977). Effect of heparin on complement activation and lysis of paroxysmal nocturnal hemoglobinuria (PNH) red cells. Blood. 50(2). 239–247. 41 indexed citations

14.

Logue, GL. (1977). Effect of heparin on complement activation and lysis of paroxysmal nocturnal hemoglobinuria (PNH) red cells. Blood. 50(2). 239–247. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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