Girish C. Melkani

2.1k total citations · 1 hit paper
57 papers, 1.5k citations indexed

About

Girish C. Melkani is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Physiology. According to data from OpenAlex, Girish C. Melkani has authored 57 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 34 papers in Molecular Biology, 18 papers in Cardiology and Cardiovascular Medicine and 17 papers in Physiology. Recurrent topics in Girish C. Melkani's work include Cardiomyopathy and Myosin Studies (17 papers), Genetics, Aging, and Longevity in Model Organisms (16 papers) and Muscle Physiology and Disorders (15 papers). Girish C. Melkani is often cited by papers focused on Cardiomyopathy and Myosin Studies (17 papers), Genetics, Aging, and Longevity in Model Organisms (16 papers) and Muscle Physiology and Disorders (15 papers). Girish C. Melkani collaborates with scholars based in United States, United Kingdom and India. Girish C. Melkani's co-authors include Satchidananda Panda, Sanford I. Bernstein, Amandine Chaix, Emily N. C. Manoogian, Hiep D. Le, Shubhroz Gill, Adriana S. Trujillo, Anthony Cammarato, Karen Ocorr and Rolf Bodmer and has published in prestigious journals such as Science, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Girish C. Melkani

57 papers receiving 1.5k citations

Hit Papers

Time-Restricted Eating to Prevent and Manage Chronic Meta... 2019 2026 2021 2023 2019 50 100 150 200 250

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Girish C. Melkani United States 17 704 552 527 283 252 57 1.5k
April D. Strader United States 19 857 1.2× 248 0.4× 721 1.4× 53 0.2× 35 0.1× 35 1.7k
Iveta Herichová Slovakia 20 243 0.3× 244 0.4× 587 1.1× 58 0.2× 125 0.5× 60 1.1k
Silke Kießling Germany 17 539 0.8× 244 0.4× 950 1.8× 92 0.3× 17 0.1× 23 1.4k
Kayoko Ogimoto United States 24 729 1.0× 432 0.8× 822 1.6× 11 0.0× 87 0.3× 38 1.8k
Frank C. Tinsley United States 12 861 1.2× 325 0.6× 1.3k 2.5× 28 0.1× 53 0.2× 22 2.1k
Shinya Nishide Japan 13 188 0.3× 146 0.3× 281 0.5× 24 0.1× 53 0.2× 22 648
Mirosław Mackiewicz United States 13 345 0.5× 101 0.2× 555 1.1× 35 0.1× 25 0.1× 15 1.1k
I. М. Kvetnoy Russia 17 271 0.4× 226 0.4× 557 1.1× 30 0.1× 16 0.1× 105 1.1k
Ilya Trakht Russia 9 136 0.2× 199 0.4× 508 1.0× 18 0.1× 75 0.3× 17 973

Countries citing papers authored by Girish C. Melkani

Since Specialization
Citations

This map shows the geographic impact of Girish C. Melkani's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Girish C. Melkani with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Girish C. Melkani more than expected).

Fields of papers citing papers by Girish C. Melkani

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Girish C. Melkani. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Girish C. Melkani. The network helps show where Girish C. Melkani may publish in the future.

Co-authorship network of co-authors of Girish C. Melkani

This figure shows the co-authorship network connecting the top 25 collaborators of Girish C. Melkani. A scholar is included among the top collaborators of Girish C. Melkani based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Girish C. Melkani. Girish C. Melkani is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Halder, Sajal Kumar & Girish C. Melkani. (2025). The Interplay of Genetic Predisposition, Circadian Misalignment, and Metabolic Regulation in Obesity. Current Obesity Reports. 14(1). 21–21. 6 indexed citations
2.
Zhang, Jianhua, et al.. (2025). Linkage of circadian rhythm disruptions with Alzheimer's disease and therapeutic interventions. Acta Pharmaceutica Sinica B. 15(6). 2945–2965. 3 indexed citations
4.
Mao, Haibin, et al.. (2024). Apolipoprotein E Induces Lipid Accumulation Through Dgat2 That Is Prevented with Time-Restricted Feeding in Drosophila. Genes. 15(11). 1376–1376. 1 indexed citations
5.
Bhide, Shruti, Sahaana Chandran, Namakkal S. Rajasekaran, & Girish C. Melkani. (2024). Genetic and Pathophysiological Basis of Cardiac and Skeletal Muscle Laminopathies. Genes. 15(8). 1095–1095. 4 indexed citations
6.
Guo, Yiming, et al.. (2024). Time‐restricted feeding mediated modulation of microbiota leads to changes in muscle physiology in Drosophila obesity models. Aging Cell. 24(2). e14382–e14382. 1 indexed citations
7.
Guo, Yiming, et al.. (2023). Time-restricted feeding promotes muscle function through purine cycle and AMPK signaling in Drosophila obesity models. Nature Communications. 14(1). 949–949. 23 indexed citations
8.
Melkani, Girish C., et al.. (2023). Circadian‐mediated regulation of cardiometabolic disorders and aging with time‐restricted feeding. Obesity. 31(S1). 40–49. 14 indexed citations
9.
Trujillo, Adriana S., Sahaana Chandran, Hiep D. Le, et al.. (2019). Time-restricted feeding restores muscle function in Drosophila models of obesity and circadian-rhythm disruption. Nature Communications. 10(1). 2700–2700. 107 indexed citations
10.
Kronert, William A., Meera Viswanathan, Girish C. Melkani, et al.. (2018). Prolonged cross-bridge binding triggers muscle dysfunction in a Drosophila model of myosin-based hypertrophic cardiomyopathy. eLife. 7. 23 indexed citations
11.
Bhide, Shruti, Adriana S. Trujillo, Diane E. Cryderman, et al.. (2018). Increasing autophagy and blocking Nrf2 suppress laminopathy‐induced age‐dependent cardiac dysfunction and shortened lifespan. Aging Cell. 17(3). e12747–e12747. 40 indexed citations
12.
Gill, Shubhroz, Hiep D. Le, Girish C. Melkani, & Satchidananda Panda. (2015). Time-restricted feeding attenuates age-related cardiac decline in Drosophila. Science. 347(6227). 1265–1269. 212 indexed citations
13.
Kronert, William A., et al.. (2015). A Failure to Communicate. Journal of Biological Chemistry. 290(49). 29270–29280. 15 indexed citations
14.
Kronert, William A., et al.. (2014). Mapping Interactions between Myosin Relay and Converter Domains That Power Muscle Function. Journal of Biological Chemistry. 289(18). 12779–12790. 14 indexed citations
15.
Trujillo, Adriana S., et al.. (2013). Exploration and Suppression of Tau-Induced Cardiac and Skeletal Muscle Defects in a Drosophila Model. Biophysical Journal. 104(2). 486a–486a. 1 indexed citations
16.
Melkani, Girish C., Adriana S. Trujillo, Raúl Ramos, et al.. (2013). Huntington's Disease Induced Cardiac Amyloidosis Is Reversed by Modulating Protein Folding and Oxidative Stress Pathways in the Drosophila Heart. PLoS Genetics. 9(12). e1004024–e1004024. 66 indexed citations
17.
Melkani, Girish C., et al.. (2012). Expression of the inclusion body myopathy 3 mutation in Drosophila depresses myosin function and stability and recapitulates muscle inclusions and weakness. Molecular Biology of the Cell. 23(11). 2057–2065. 12 indexed citations
18.
Melkani, Girish C., Rolf Bodmer, Karen Ocorr, & Sanford I. Bernstein. (2011). The UNC-45 Chaperone Is Critical for Establishing Myosin-Based Myofibrillar Organization and Cardiac Contractility in the Drosophila Heart Model. PLoS ONE. 6(7). e22579–e22579. 43 indexed citations
19.
Melkani, Girish C., Gustavo Zardeneta, & Jose A. Mendoza. (2005). On the chaperonin activity of GroEL at heat-shock temperature. The International Journal of Biochemistry & Cell Biology. 37(7). 1375–1385. 11 indexed citations
20.
Melkani, Girish C., Gustavo Zardeneta, & Jose A. Mendoza. (2002). GroEL interacts transiently with oxidatively inactivated rhodanese facilitating its reactivation. Biochemical and Biophysical Research Communications. 294(4). 893–899. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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