Giacomo Casella

948 total citations
21 papers, 746 citations indexed

About

Giacomo Casella is a scholar working on Molecular Biology, Neurology and Immunology. According to data from OpenAlex, Giacomo Casella has authored 21 papers receiving a total of 746 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 13 papers in Neurology and 11 papers in Immunology. Recurrent topics in Giacomo Casella's work include Neuroinflammation and Neurodegeneration Mechanisms (13 papers), Extracellular vesicles in disease (9 papers) and Immune cells in cancer (4 papers). Giacomo Casella is often cited by papers focused on Neuroinflammation and Neurodegeneration Mechanisms (13 papers), Extracellular vesicles in disease (9 papers) and Immune cells in cancer (4 papers). Giacomo Casella collaborates with scholars based in United States, Italy and China. Giacomo Casella's co-authors include Annamaria Finardi, Roberto Furlan, Abdolmohamad Rostami, Xing Li, Federico Colombo, Luca Muzio, Gianvito Martino, Yuan Zhang, Livia Garzetti and Jing Tian and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Clinical Investigation and Scientific Reports.

In The Last Decade

Giacomo Casella

20 papers receiving 741 citations

Peers

Giacomo Casella
Deanna M. Patmore United States
Hong Wei China
Yazhong Tao United States
Giacomo Casella
Citations per year, relative to Giacomo Casella Giacomo Casella (= 1×) peers Marc Charabati

Countries citing papers authored by Giacomo Casella

Since Specialization
Citations

This map shows the geographic impact of Giacomo Casella's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Giacomo Casella with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Giacomo Casella more than expected).

Fields of papers citing papers by Giacomo Casella

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Giacomo Casella. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Giacomo Casella. The network helps show where Giacomo Casella may publish in the future.

Co-authorship network of co-authors of Giacomo Casella

This figure shows the co-authorship network connecting the top 25 collaborators of Giacomo Casella. A scholar is included among the top collaborators of Giacomo Casella based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Giacomo Casella. Giacomo Casella is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Hwang, Daniel, Larissa Lumi Watanabe Ishikawa, Alexandra Boehm, et al.. (2022). CSF-1 maintains pathogenic but not homeostatic myeloid cells in the central nervous system during autoimmune neuroinflammation. Proceedings of the National Academy of Sciences. 119(14). e2111804119–e2111804119. 17 indexed citations
2.
Zhang, Weifeng, Dan Xiao, Xing Li, et al.. (2022). SIRT1 inactivation switches reactive astrocytes to an antiinflammatory phenotype in CNS autoimmunity. Journal of Clinical Investigation. 132(22). 28 indexed citations
3.
Rasouli, Javad, Giacomo Casella, Weifeng Zhang, et al.. (2022). Transcription Factor RUNX3 Mediates Plasticity of ThGM Cells Toward Th1 Phenotype. Frontiers in Immunology. 13. 912583–912583. 6 indexed citations
4.
Rasouli, Javad, Giacomo Casella, Larissa Lumi Watanabe Ishikawa, et al.. (2021). IFN-β Acts on Monocytes to Ameliorate CNS Autoimmunity by Inhibiting Proinflammatory Cross-Talk Between Monocytes and Th Cells. Frontiers in Immunology. 12. 679498–679498. 10 indexed citations
5.
Xiao, Dan, Weifeng Zhang, Qing Wang, et al.. (2021). CRISPR-mediated rapid generation of neural cell-specific knockout mice facilitates research in neurophysiology and pathology. Molecular Therapy — Methods & Clinical Development. 20. 755–764. 8 indexed citations
6.
Xiao, Yun, Yuan Zhang, Abdolmohamad Rostami, et al.. (2021). Role of extracellular vesicles in neurodegenerative diseases. Progress in Neurobiology. 201. 102022–102022. 50 indexed citations
7.
Casella, Giacomo, Javad Rasouli, Alexandra Boehm, et al.. (2020). Oligodendrocyte-derived extracellular vesicles as antigen-specific therapy for autoimmune neuroinflammation in mice. Science Translational Medicine. 12(568). 80 indexed citations
8.
Colombo, Federico, Giacomo Casella, Paola Podini, et al.. (2020). Polarized cells display asymmetric release of extracellular vesicles. Traffic. 22(4). 98–110. 21 indexed citations
9.
Rasouli, Javad, Giacomo Casella, Satoshi Yoshimura, et al.. (2020). A distinct GM-CSF + T helper cell subset requires T-bet to adopt a T H 1 phenotype and promote neuroinflammation. Science Immunology. 5(52). 39 indexed citations
10.
Thomé, Rodolfo, Giacomo Casella, Noushin Lotfi, et al.. (2020). Primaquine elicits Foxp3+ regulatory T cells with a superior ability to limit CNS autoimmune inflammation. Journal of Autoimmunity. 114. 102505–102505. 4 indexed citations
11.
Casella, Giacomo, Javad Rasouli, Rodolfo Thomé, et al.. (2020). Interferon-γ/Interleukin-27 Axis Induces Programmed Death Ligand 1 Expression in Monocyte-Derived Dendritic Cells and Restores Immune Tolerance in Central Nervous System Autoimmunity. Frontiers in Immunology. 11. 576752–576752. 11 indexed citations
12.
Tian, Jing, Giacomo Casella, Yuan Zhang, Abdolmohamad Rostami, & Xing Li. (2020). Potential roles of extracellular vesicles in the pathophysiology, diagnosis, and treatment of autoimmune diseases. International Journal of Biological Sciences. 16(4). 620–632. 69 indexed citations
13.
Thomé, Rodolfo, Alexandra Boehm, Larissa Lumi Watanabe Ishikawa, et al.. (2020). Comprehensive Analysis of the Immune and Stromal Compartments of the CNS in EAE Mice Reveal Pathways by Which Chloroquine Suppresses Neuroinflammation. Brain Sciences. 10(6). 348–348.
14.
Zhang, Yuan, Giacomo Casella, Jing Tian, et al.. (2019). Generation of Oligodendrocyte Progenitor Cells From Mouse Bone Marrow Cells. Frontiers in Cellular Neuroscience. 13. 247–247. 14 indexed citations
15.
Casella, Giacomo, Federico Colombo, Annamaria Finardi, et al.. (2018). Extracellular Vesicles Containing IL-4 Modulate Neuroinflammation in a Mouse Model of Multiple Sclerosis. Molecular Therapy. 26(9). 2107–2118. 116 indexed citations
16.
Colombo, Federico, Annamaria Nigro, Paola Podini, et al.. (2018). Cytokines Stimulate the Release of Microvesicles from Myeloid Cells Independently from the P2X7 Receptor/Acid Sphingomyelinase Pathway. Frontiers in Immunology. 9. 204–204. 37 indexed citations
17.
Casella, Giacomo, Annamaria Finardi, Hélène C. Descamps, et al.. (2017). IL-27, but not IL-35, inhibits neuroinflammation through modulating GM-CSF expression. Scientific Reports. 7(1). 16547–16547. 33 indexed citations
18.
Nigro, Annamaria, Federico Colombo, Giacomo Casella, et al.. (2016). Myeloid Extracellular Vesicles: Messengers from the Demented Brain. Frontiers in Immunology. 7. 17–17. 25 indexed citations
19.
Casella, Giacomo, Livia Garzetti, Alberto T. Gatta, et al.. (2016). IL4 induces IL6-producing M2 macrophages associated to inhibition of neuroinflammation in vitro and in vivo. Journal of Neuroinflammation. 13(1). 139–139. 118 indexed citations
20.
Carandini, Tiziana, Federico Colombo, Annamaria Finardi, et al.. (2015). Microvesicles: What is the Role in Multiple Sclerosis?. Frontiers in Neurology. 6. 111–111. 39 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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