Ghayath Baroudi

1.1k total citations
18 papers, 882 citations indexed

About

Ghayath Baroudi is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Cellular and Molecular Neuroscience. According to data from OpenAlex, Ghayath Baroudi has authored 18 papers receiving a total of 882 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Molecular Biology, 14 papers in Cardiology and Cardiovascular Medicine and 3 papers in Cellular and Molecular Neuroscience. Recurrent topics in Ghayath Baroudi's work include Cardiac electrophysiology and arrhythmias (13 papers), Ion channel regulation and function (12 papers) and Connexins and lens biology (5 papers). Ghayath Baroudi is often cited by papers focused on Cardiac electrophysiology and arrhythmias (13 papers), Ion channel regulation and function (12 papers) and Connexins and lens biology (5 papers). Ghayath Baroudi collaborates with scholars based in Canada, United States and Italy. Ghayath Baroudi's co-authors include Mohamed Chahine, Valérie Pouliot, Yongxia Qu, Mohamed Boutjdir, Mohamed Benderdour, Pascale Guicheney, Yuankun Yue, Guy Rousseau, Pierre Hélie and Alvin Shrier and has published in prestigious journals such as Journal of Biological Chemistry, Circulation and Circulation Research.

In The Last Decade

Ghayath Baroudi

18 papers receiving 875 citations

Peers

Countries citing papers authored by Ghayath Baroudi

Since Specialization

Citations

This map shows the geographic impact of Ghayath Baroudi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ghayath Baroudi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ghayath Baroudi more than expected).

Fields of papers citing papers by Ghayath Baroudi

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ghayath Baroudi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ghayath Baroudi. The network helps show where Ghayath Baroudi may publish in the future.

Co-authorship network of co-authors of Ghayath Baroudi

This figure shows the co-authorship network connecting the top 25 collaborators of Ghayath Baroudi. A scholar is included among the top collaborators of Ghayath Baroudi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ghayath Baroudi. Ghayath Baroudi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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18 of 18 papers shown

#	Title	Journal	Authors	Indexed citations
1	Roles of PKC Isoforms in PMA-Induced Modulation of the hERG Channel (Kv11.1)	SLAS DISCOVERY	António Guia, Ghayath Baroudi et al.	5
2	Single intravenous low-dose injections of connexin 43 mimetic peptides protect ischemic heart in vivo against myocardial infarction	Journal of Molecular and Cellular Cardiology	Pierre Hélie, Ghayath Baroudi et al.	62
3	Ramipril attenuates lipid peroxidation and cardiac fibrosis in an experimental model of rheumatoid arthritis	Arthritis Research & Therapy	Qin Shi, Jamilah Abusarah et al.	28
4	Connexin 43 mimetic peptide Gap26 confers protection to intact heart against myocardial ischemia injury	Pflügers Archiv - European Journal of Physiology	Mohamed Benderdour, Guy Rousseau et al.	88
5	Abstract 3623: Selective Inhibition of Connexin 43 Hemichannels by GAP26 Confers Cardioprotection Against Ischemia-Reperfusion Injury: In vivo Study	Circulation	Ghayath Baroudi et al.	1
6	Phosphorylation of the Consensus Sites of Protein Kinase A on α1D L-type Calcium Channel	Journal of Biological Chemistry	Yongxia Qu, Raj Wadgaonkar et al.	16
7	Differential modulation of unapposed connexin 43 hemichannel electrical conductance by protein kinase C isoforms	Pflügers Archiv - European Journal of Physiology	Ghayath Baroudi et al.	25
8	Combined effects of reduced connexin 43, depressed active generator properties and energetic stress on conduction disturbances in canine failing myocardium	Pflügers Archiv - European Journal of Physiology	Guy Rousseau, Ghayath Baroudi et al.	1
9	Protein kinase C activation inhibits Ca_v1.3 calcium channel at NH₂-terminal serine 81 phosphorylation site	American Journal of Physiology-Heart and Circulatory Physiology	Ghayath Baroudi, Yongxia Qu et al.	22
10	Novel Molecular Mechanism Involving α _1D (Cav1.3) L-Type Calcium Channel in Autoimmune-Associated Sinus Bradycardia	Circulation	Yongxia Qu, Ghayath Baroudi et al.	59
11	Localization and modulation of α_1D (Cav1.3) L-type Ca channel by protein kinase A	American Journal of Physiology-Heart and Circulatory Physiology	Yongxia Qu, Ghayath Baroudi et al.	44
12	Loss of function associated with novel mutations of the SCN5A gene in patients with Brugada syndrome.	PubMed	Ghayath Baroudi, Carlo Napolitano et al.	31
13	A Newly Characterized SCN5A Mutation Underlying Brugada Syndrome Unmasked by Hyperthermia	Journal of Cardiovascular Electrophysiology	Silvia G. Priori, Carlo Napolitano et al.	66
14	Expression and Intracellular Localization of an *SCN5A* Double Mutant R1232W/T1620M Implicated in Brugada Syndrome	Circulation Research	Ghayath Baroudi, Mohamed Chahine et al.	137
15	Novel Mechanism for Brugada Syndrome	Circulation Research	Ghayath Baroudi, Valérie Pouliot et al.	106
16	Homozygous SCN5A Mutation in Long-QT Syndrome With Functional Two-to-One Atrioventricular Block	Circulation Research	Jean‐Marc Lupoglazoff, Ghayath Baroudi et al.	74
17	SCN5A mutation (T1620M) causing Brugada syndrome exhibits different phenotypes when expressed in Xenopus oocytes and mammalian cells	FEBS Letters	Ghayath Baroudi, Valérie Pouliot et al.	59
18	Biophysical phenotypes of SCN5A mutations causing long QT and Brugada syndromes	FEBS Letters	Ghayath Baroudi, Mohamed Chahine	58

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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