Mohamed Boutjdir

6.2k total citations
155 papers, 4.5k citations indexed

About

Mohamed Boutjdir is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Mohamed Boutjdir has authored 155 papers receiving a total of 4.5k indexed citations (citations by other indexed papers that have themselves been cited), including 119 papers in Cardiology and Cardiovascular Medicine, 95 papers in Molecular Biology and 25 papers in Cellular and Molecular Neuroscience. Recurrent topics in Mohamed Boutjdir's work include Cardiac electrophysiology and arrhythmias (105 papers), Ion channel regulation and function (73 papers) and Cardiac Arrhythmias and Treatments (27 papers). Mohamed Boutjdir is often cited by papers focused on Cardiac electrophysiology and arrhythmias (105 papers), Ion channel regulation and function (73 papers) and Cardiac Arrhythmias and Treatments (27 papers). Mohamed Boutjdir collaborates with scholars based in United States, Italy and Canada. Mohamed Boutjdir's co-authors include Nabil El‐Sherif, Pietro Enea Lazzerini, Pier Leopoldo Capecchi, Yongxia Qu, Franco Laghi‐Pasini, Mohamed Chahine, Yuankun Yue, Keli Hu, Ademuyiwa S. Aromolaran and Daria Mochly‐Rosen and has published in prestigious journals such as Journal of Biological Chemistry, Circulation and SHILAP Revista de lepidopterología.

In The Last Decade

Mohamed Boutjdir

150 papers receiving 4.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mohamed Boutjdir United States 39 2.9k 2.1k 536 351 334 155 4.5k
Kyosuke Takeshita Japan 35 981 0.3× 1.7k 0.8× 178 0.3× 98 0.3× 158 0.5× 141 4.4k
Mónica Rupérez Spain 27 1.7k 0.6× 1.4k 0.7× 172 0.3× 109 0.3× 125 0.4× 29 4.0k
Ratnadeep Basu Canada 35 2.2k 0.8× 1.2k 0.6× 183 0.3× 556 1.6× 52 0.2× 52 4.1k
Stéphane Hatem France 46 4.2k 1.5× 2.6k 1.3× 674 1.3× 71 0.2× 56 0.2× 135 6.1k
Brigitte Rigat Canada 21 3.9k 1.4× 1.2k 0.6× 236 0.4× 96 0.3× 219 0.7× 32 6.5k
Jorge Jalil Chile 30 2.5k 0.9× 1.1k 0.5× 134 0.3× 320 0.9× 53 0.2× 110 4.0k
Katsuhiko Hiramori Japan 40 2.9k 1.0× 753 0.4× 215 0.4× 69 0.2× 142 0.4× 187 4.7k
Pierre Paradis Canada 40 2.3k 0.8× 1.8k 0.9× 160 0.3× 91 0.3× 83 0.2× 109 5.4k
Ming‐Sum Lee United States 20 1.0k 0.4× 1.2k 0.6× 539 1.0× 129 0.4× 65 0.2× 99 3.6k
Michael Arad Israel 37 3.2k 1.1× 2.6k 1.3× 170 0.3× 44 0.1× 306 0.9× 172 5.8k

Countries citing papers authored by Mohamed Boutjdir

Since Specialization
Citations

This map shows the geographic impact of Mohamed Boutjdir's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mohamed Boutjdir with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mohamed Boutjdir more than expected).

Fields of papers citing papers by Mohamed Boutjdir

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mohamed Boutjdir. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mohamed Boutjdir. The network helps show where Mohamed Boutjdir may publish in the future.

Co-authorship network of co-authors of Mohamed Boutjdir

This figure shows the co-authorship network connecting the top 25 collaborators of Mohamed Boutjdir. A scholar is included among the top collaborators of Mohamed Boutjdir based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mohamed Boutjdir. Mohamed Boutjdir is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Lazzerini, Pietro Enea & Mohamed Boutjdir. (2025). Autoimmune cardiac channelopathies and heart rhythm disorders: A contemporary review. Heart Rhythm. 22(6). 1541–1561. 3 indexed citations
2.
Srivastava, Ujala, et al.. (2025). Novel therapeutic cav1.3-C-terminus normalizes L-type calcium current and ejection fraction in ischemia-induced murine heart failure. Biochemical and Biophysical Research Communications. 779. 152455–152455.
3.
El‐Sherif, Nabil, et al.. (2024). Calcium handling abnormalities increase arrhythmia susceptibility in DMSXL myotonic dystrophy type 1 mice. Biomedicine & Pharmacotherapy. 180. 117562–117562. 1 indexed citations
4.
Lazzerini, Pietro Enea, Alessandra Cartocci, Stefania Bisogno, et al.. (2024). Increased interleukin‐6 levels are associated with atrioventricular conduction delay in severe COVID‐19 patients. Journal of Arrhythmia. 40(5). 1137–1148. 2 indexed citations
5.
Lazzerini, Pietro Enea, Alessandra Cartocci, Iacopo Bertolozzi, et al.. (2024). Elevated Interleukin‐6 Levels Are Associated With an Increased Risk of QTc Interval Prolongation in a Large Cohort of US Veterans. Journal of the American Heart Association. 13(4). e032071–e032071. 4 indexed citations
6.
Chahine, Mohamed, et al.. (2024). Arrhythmias and ion channelopathies causing sudden cardiac death in Hispanic/Latino and Indigenous populations. Journal of Cardiovascular Electrophysiology. 35(6). 1219–1228.
7.
8.
Lazzerini, Pietro Enea, Antonio Abbate, Mohamed Boutjdir, & Pier Leopoldo Capecchi. (2023). Fir(e)ing the Rhythm. JACC Basic to Translational Science. 8(6). 728–750. 51 indexed citations
9.
Boutjdir, Mohamed, et al.. (2023). Optical Mapping of Cardiomyocytes in Monolayer Derived from Induced Pluripotent Stem Cells. Cells. 12(17). 2168–2168. 5 indexed citations
10.
11.
Diallo, Ana F., et al.. (2022). Addressing challenges faced by underrepresented biomedical investigators and efforts to address them: An NHLBI-PRIDE perspective. Journal of the National Medical Association. 114(6). 569–577. 8 indexed citations
12.
Jauvin, Dominic, et al.. (2022). Recent Progress and Challenges in the Development of Antisense Therapies for Myotonic Dystrophy Type 1. International Journal of Molecular Sciences. 23(21). 13359–13359. 19 indexed citations
13.
Jauvin, Dominic, Frank Bennett, Frank Rigo, et al.. (2022). Enhanced Delivery of Ligand-Conjugated Antisense Oligonucleotides (C16-HA-ASO) Targeting Dystrophia Myotonica Protein Kinase Transcripts for the Treatment of Myotonic Dystrophy Type 1. Human Gene Therapy. 33(15-16). 810–820. 16 indexed citations
14.
Lazzerini, Pietro Enea, Silvia Cantara, Iacopo Bertolozzi, et al.. (2021). Transient Hypogonadism Is Associated With Heart Rate–Corrected QT Prolongation and Torsades de Pointes Risk During Active Systemic Inflammation in Men. Journal of the American Heart Association. 11(1). e023371–e023371. 10 indexed citations
15.
Lazzerini, Pietro Enea, Alessandra Cartocci, Yongxia Qu, et al.. (2021). Proton Pump Inhibitors Directly Block hERG-Potassium Channel and Independently Increase the Risk of QTc Prolongation in a Large Cohort of US Veterans. Circulation Arrhythmia and Electrophysiology. 14(7). e010042–e010042. 8 indexed citations
16.
Lazzerini, Pietro Enea, Maurizio Acampa, Franco Laghi‐Pasini, et al.. (2020). Cardiac Arrest Risk During Acute Infections. Circulation Arrhythmia and Electrophysiology. 13(8). e008627–e008627. 41 indexed citations
17.
Srivastava, Ujala, et al.. (2020). Novel re-expression of L-type calcium channel Cav1.3 in left ventricles of failing human heart. Heart Rhythm. 17(7). 1193–1197. 6 indexed citations
18.
Boutjdir, Mohamed. (2000). Molecular and Ionic Basis of Congenital Complete Heart Block. Trends in Cardiovascular Medicine. 10(3). 114–122. 36 indexed citations
19.
Bekheit, Soad, et al.. (1993). Reduction of ischemia-induced electrophysiologic abnormalities by glucose-insulin infusion. Journal of the American College of Cardiology. 22(4). 1214–1222. 7 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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