Géza Sas

437 total citations
11 papers, 370 citations indexed

About

Géza Sas is a scholar working on Hematology, Surgery and Molecular Biology. According to data from OpenAlex, Géza Sas has authored 11 papers receiving a total of 370 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Hematology, 3 papers in Surgery and 3 papers in Molecular Biology. Recurrent topics in Géza Sas's work include Blood Coagulation and Thrombosis Mechanisms (8 papers), Heparin-Induced Thrombocytopenia and Thrombosis (3 papers) and Blood properties and coagulation (2 papers). Géza Sas is often cited by papers focused on Blood Coagulation and Thrombosis Mechanisms (8 papers), Heparin-Induced Thrombocytopenia and Thrombosis (3 papers) and Blood properties and coagulation (2 papers). Géza Sas collaborates with scholars based in United Kingdom, Austria and Germany. Géza Sas's co-authors include Duncan S. Pepper, J. D. Cash, G Blaskó, Dénes Bánhegyi, Gyula Domján, John H. Griffin, András Gruber, S. Middleton, James K. Smith and Ingrid Pabinger and has published in prestigious journals such as British Journal of Haematology, Thrombosis and Haemostasis and Human Mutation.

In The Last Decade

Géza Sas

11 papers receiving 345 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Géza Sas United Kingdom 9 297 70 69 64 55 11 370
Akinobu Funatsu Japan 11 263 0.9× 57 0.8× 34 0.5× 63 1.0× 31 0.6× 22 354
MJ Heeb United States 6 260 0.9× 43 0.6× 53 0.8× 136 2.1× 20 0.4× 8 347
JC Meijers Netherlands 7 430 1.4× 24 0.3× 36 0.5× 318 5.0× 43 0.8× 9 538
Rowan G. Doig Australia 7 224 0.8× 24 0.3× 98 1.4× 56 0.9× 25 0.5× 11 297
Fabian Stavenuiter United States 9 165 0.6× 19 0.3× 41 0.6× 51 0.8× 25 0.5× 9 304
Verónica Palma‐Barqueros Spain 10 212 0.7× 19 0.3× 17 0.2× 53 0.8× 40 0.7× 15 288
Granada Perea Spain 8 109 0.4× 26 0.4× 21 0.3× 51 0.8× 37 0.7× 12 278
Tomoyuki Sasaki Japan 8 102 0.3× 35 0.5× 25 0.4× 22 0.3× 35 0.6× 16 268
Marco Togni Italy 8 156 0.5× 93 1.3× 16 0.2× 24 0.4× 21 0.4× 10 338
Nel H. BEESER‐VISSER Netherlands 8 394 1.3× 35 0.5× 13 0.2× 130 2.0× 57 1.0× 10 439

Countries citing papers authored by Géza Sas

Since Specialization
Citations

This map shows the geographic impact of Géza Sas's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Géza Sas with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Géza Sas more than expected).

Fields of papers citing papers by Géza Sas

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Géza Sas. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Géza Sas. The network helps show where Géza Sas may publish in the future.

Co-authorship network of co-authors of Géza Sas

This figure shows the co-authorship network connecting the top 25 collaborators of Géza Sas. A scholar is included among the top collaborators of Géza Sas based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Géza Sas. Géza Sas is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
Olds, Robin J., David A. Lane, Vijoy Chowdhury, et al.. (1994). (ATT) trinucleotide repeats in the antithrombin gene and their use in determining the origin of repeated mutations. Human Mutation. 4(1). 31–41. 20 indexed citations
2.
Sas, Géza. (1989). The biology of antithrombins. 1 indexed citations
3.
Gruber, András, G Blaskó, & Géza Sas. (1986). Functional deficiency of protein c and skin necrosis in multiple myeloma. Thrombosis Research. 42(4). 579–581. 23 indexed citations
4.
Sas, Géza, et al.. (1985). A Protein S Deficient Family with Portal Vein Thrombosis. Thrombosis and Haemostasis. 54(3). 724–724. 35 indexed citations
5.
Sas, Géza. (1984). Congenital and acquired defects of antithrombin III. La Ricerca in Clinica e in Laboratorio. 14(3). 491–497. 5 indexed citations
6.
Sas, Géza, et al.. (1980). Heterogeneity of the "Classical" Antithrombin III Deficiency. Thrombosis and Haemostasis. 43(2). 133–136. 64 indexed citations
7.
Sas, Géza & Duncan S. Pepper. (1976). Detection of thrombin-antithrombin III complex by crossed immunoelectrophoresis. Thrombosis Research. 9(1). 95–97. 17 indexed citations
8.
Sas, Géza, et al.. (1975). In Vitro Spontaneous Thrombin Generation in Human Factor‐IX Concentrates. British Journal of Haematology. 31(1). 25–35. 31 indexed citations
9.
Sas, Géza, Duncan S. Pepper, & J. D. Cash. (1975). Further Investigations on Antithrombin III in the Plasmas of Patients with the Abnormality of ‘Antithrombin III Budapest’. Thrombosis and Haemostasis. 33(3). 564–572. 40 indexed citations
10.
Sas, Géza, Duncan S. Pepper, & J. D. Cash. (1975). Plasma and serum antithrombin - III: Differentiation by crossed immunoelectrophoresis. Thrombosis Research. 6(1). 87–91. 65 indexed citations
11.
Sas, Géza, Duncan S. Pepper, & J. D. Cash. (1975). Investigations on Antithrombin III in Normal Plasma and Serum. British Journal of Haematology. 30(3). 265–272. 69 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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