Gene Lin

590 total citations
9 papers, 465 citations indexed

About

Gene Lin is a scholar working on Immunology, Physiology and Infectious Diseases. According to data from OpenAlex, Gene Lin has authored 9 papers receiving a total of 465 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Immunology, 4 papers in Physiology and 2 papers in Infectious Diseases. Recurrent topics in Gene Lin's work include Immune Cell Function and Interaction (5 papers), Adenosine and Purinergic Signaling (4 papers) and Hemoglobinopathies and Related Disorders (2 papers). Gene Lin is often cited by papers focused on Immune Cell Function and Interaction (5 papers), Adenosine and Purinergic Signaling (4 papers) and Hemoglobinopathies and Related Disorders (2 papers). Gene Lin collaborates with scholars based in United States and France. Gene Lin's co-authors include Joel Linden, Kimberley Stannard, Meriem Messaoudene, Laurence Zitvogel, Mark J. Smyth, Joshua J. Field, Nicola Waddell, Ann‐Marie Patch, Arabella Young and Jérôme D. Coudert and has published in prestigious journals such as Blood, PLoS ONE and Cancer Research.

In The Last Decade

Gene Lin

9 papers receiving 459 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Gene Lin United States	6	229	158	147	96	87	9	465
Andrea Zito Italy	7	117 0.5×	232 1.5×	127 0.9×	32 0.3×	66 0.8×	12	390
Ligen Liu China	11	113 0.5×	26 0.2×	89 0.6×	43 0.4×	142 1.6×	29	372
Manasa Madasu United States	5	320 1.4×	328 2.1×	178 1.2×	18 0.2×	7 0.1×	6	566
U Schmücker Germany	6	166 0.7×	52 0.3×	89 0.6×	191 2.0×	48 0.6×	10	401
Filippo Cortesi Italy	10	174 0.8×	43 0.3×	171 1.2×	16 0.2×	8 0.1×	13	397
Е. К. Олейник Russia	7	99 0.4×	44 0.3×	73 0.5×	11 0.1×	19 0.2×	16	357
Chia-Jui Ku United States	9	93 0.4×	15 0.1×	35 0.2×	20 0.2×	79 0.9×	11	347
Rebecca H. Horton United Kingdom	4	50 0.2×	12 0.1×	46 0.3×	43 0.4×	77 0.9×	8	295
Dimitrios Alatzoglou Switzerland	2	198 0.9×	103 0.7×	246 1.7×	8 0.1×	8 0.1×	3	409
I Montalban del Barrio Germany	5	224 1.0×	204 1.3×	175 1.2×	8 0.1×	8 0.1×	5	412

Countries citing papers authored by Gene Lin

Since Specialization

Citations

This map shows the geographic impact of Gene Lin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gene Lin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gene Lin more than expected).

Fields of papers citing papers by Gene Lin

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gene Lin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gene Lin. The network helps show where Gene Lin may publish in the future.

Co-authorship network of co-authors of Gene Lin

This figure shows the co-authorship network connecting the top 25 collaborators of Gene Lin. A scholar is included among the top collaborators of Gene Lin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Gene Lin. Gene Lin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

9 of 9 papers shown

1.

Yu, Jennifer C., Gene Lin, Joshua J. Field, & Joel Linden. (2018). Induction of antiinflammatory purinergic signaling in activated human iNKT cells. JCI Insight. 3(17). 15 indexed citations

2.

Young, Arabella, Shin Foong Ngiow, Ann‐Marie Patch, et al.. (2017). A2AR Adenosine Signaling Suppresses Natural Killer Cell Maturation in the Tumor Microenvironment. Cancer Research. 78(4). 1003–1016. 296 indexed citations

3.

Field, Joshua J., Elaine M. Majerus, Victor R. Gordeuk, et al.. (2017). Randomized phase 2 trial of regadenoson for treatment of acute vaso-occlusive crises in sickle cell disease. Blood Advances. 1(20). 1645–1649. 37 indexed citations

4.

Franco, Alessandra, Gene Lin, Chisato Shimizu, et al.. (2017). Pediatric tolerogenic DCs expressing CD4 and immunoglobulin‐like transcript receptor (ILT)‐4 secrete IL‐10 in response to Fc and adenosine. European Journal of Immunology. 48(3). 482–491. 15 indexed citations

5.

Field, Joshua J., Gene Lin, Maureen Okam, et al.. (2013). Sickle cell vaso-occlusion causes activation of iNKT cells that is decreased by the adenosine A2A receptor agonist regadenoson. Blood. 121(17). 3329–3334. 72 indexed citations

6.

Lin, Gene, Joshua J. Field, Jennifer C. Yu, et al.. (2013). NF-κB Is Activated in CD4+ iNKT Cells by Sickle Cell Disease and Mediates Rapid Induction of Adenosine A2A Receptors. PLoS ONE. 8(10). e74664–e74664. 23 indexed citations

7.

Nathan, David G., Joshua J. Field, Gene Lin, et al.. (2012). Sickle cell disease (SCD), iNKT cells, and regadenoson infusion.. PubMed. 123. 312–7; discussion 317. 5 indexed citations

8.

Hwang, Grace M., et al.. (2009). Detecting infectious and biological contaminants aboard aircraft - Is it feasible?. 51. 477–484. 1 indexed citations

9.

Lin, Gene, et al.. (2009). Bringing an Economy-Wide Perspective to NextGen Benefits Analysis. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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