Gen Yosimichi

557 total citations
9 papers, 488 citations indexed

About

Gen Yosimichi is a scholar working on Molecular Biology, Rheumatology and Genetics. According to data from OpenAlex, Gen Yosimichi has authored 9 papers receiving a total of 488 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 2 papers in Rheumatology and 1 paper in Genetics. Recurrent topics in Gen Yosimichi's work include Connective Tissue Growth Factor Research (9 papers), Fibroblast Growth Factor Research (4 papers) and TGF-β signaling in diseases (2 papers). Gen Yosimichi is often cited by papers focused on Connective Tissue Growth Factor Research (9 papers), Fibroblast Growth Factor Research (4 papers) and TGF-β signaling in diseases (2 papers). Gen Yosimichi collaborates with scholars based in Japan, United States and Germany. Gen Yosimichi's co-authors include Masaharu Takigawa, Takashi Nishida, Tohru Nakanishi, Satoshi Kubota, Teruko Takano‐Yamamoto, Seiji Kondo, Takako Hattori, Takuo Kuboki, T. Fukunaga and Tomoichiro Yamaai and has published in prestigious journals such as Biochemical and Biophysical Research Communications, FEBS Letters and European Journal of Biochemistry.

In The Last Decade

Gen Yosimichi

9 papers receiving 481 citations

Peers

Gen Yosimichi
Cecilia A. Lopez United States
H.F. Wimer United States
Shawn P. Terkhorn United States
Samuel Varghese United States
Gen Yosimichi
Citations per year, relative to Gen Yosimichi Gen Yosimichi (= 1×) peers Yoshitaka Tajima

Countries citing papers authored by Gen Yosimichi

Since Specialization
Citations

This map shows the geographic impact of Gen Yosimichi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gen Yosimichi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gen Yosimichi more than expected).

Fields of papers citing papers by Gen Yosimichi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gen Yosimichi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gen Yosimichi. The network helps show where Gen Yosimichi may publish in the future.

Co-authorship network of co-authors of Gen Yosimichi

This figure shows the co-authorship network connecting the top 25 collaborators of Gen Yosimichi. A scholar is included among the top collaborators of Gen Yosimichi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Gen Yosimichi. Gen Yosimichi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

9 of 9 papers shown
1.
Kubota, Satoshi, Harumi Kawaki, Seiji Kondo, et al.. (2006). Multiple activation of mitogen-activated protein kinases by purified independent CCN2 modules in vascular endothelial cells and chondrocytes in culture. Biochimie. 88(12). 1973–1981. 20 indexed citations
2.
Yosimichi, Gen, Satoshi Kubota, Takashi Nishida, et al.. (2006). Roles of PKC, PI3K and JNK in multiple transduction of CCN2/CTGF signals in chondrocytes. Bone. 38(6). 853–863. 47 indexed citations
3.
Kondo, Seiji, Noriko Tanaka, Satoshi Kubota, et al.. (2006). Novel angiogenic inhibitor DN-9693 that inhibits post-transcriptional induction of connective tissue growth factor (CTGF/CCN2) by vascular endothelial growth factor in human endothelial cells. Molecular Cancer Therapeutics. 5(1). 129–137. 20 indexed citations
4.
Asano, Masahiro, Satoshi Kubota, Tohru Nakanishi, et al.. (2005). Effect of connective tissue growth factor (CCN2/CTGF) on proliferation and differentiation of mouse periodontal ligament-derived cells. Cell Communication and Signaling. 3(1). 11–11. 43 indexed citations
5.
Nishida, Takashi, Satoshi Kubota, T. Fukunaga, et al.. (2003). CTGF/Hcs24, hypertrophic chondrocyte‐specific gene product, interacts with perlecan in regulating the proliferation and differentiation of chondrocytes. Journal of Cellular Physiology. 196(2). 265–275. 86 indexed citations
6.
Inoue, Miho, Satoshi Kubota, Takashi Nishida, et al.. (2002). Tyrosine kinase‐type receptor ErbB4 in chondrocytes: interaction with connective tissue growth factor and distribution in cartilage. FEBS Letters. 528(1-3). 109–113. 18 indexed citations
7.
Nishida, Takashi, Satoshi Kubota, Tohru Nakanishi, et al.. (2002). CTGF/Hcs24, a hypertrophic chondrocyte‐specific gene product, stimulates proliferation and differentiation, but not hypertrophy of cultured articular chondrocytes. Journal of Cellular Physiology. 192(1). 55–63. 104 indexed citations
8.
Yosimichi, Gen, Satoshi Kubota, Takako Hattori, et al.. (2002). CTGF/Hcs24 interacts with the cytoskeletal protein actin in chondrocytes. Biochemical and Biophysical Research Communications. 299(5). 755–761. 11 indexed citations
9.
Yosimichi, Gen, Tohru Nakanishi, Takashi Nishida, et al.. (2001). CTGF/Hcs24 induces chondrocyte differentiation through a p38 mitogen‐activated protein kinase (p38MAPK), and proliferation through a p44/42 MAPK/extracellular‐signal regulated kinase (ERK). European Journal of Biochemistry. 268(23). 6058–6065. 139 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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