Ge Li‐Sha

689 total citations
32 papers, 514 citations indexed

About

Ge Li‐Sha is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Neurology. According to data from OpenAlex, Ge Li‐Sha has authored 32 papers receiving a total of 514 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Cardiology and Cardiovascular Medicine, 11 papers in Molecular Biology and 6 papers in Neurology. Recurrent topics in Ge Li‐Sha's work include Viral Infections and Immunology Research (9 papers), Nicotinic Acetylcholine Receptors Study (7 papers) and Vagus Nerve Stimulation Research (6 papers). Ge Li‐Sha is often cited by papers focused on Viral Infections and Immunology Research (9 papers), Nicotinic Acetylcholine Receptors Study (7 papers) and Vagus Nerve Stimulation Research (6 papers). Ge Li‐Sha collaborates with scholars based in China, United States and Canada. Ge Li‐Sha's co-authors include Yuechun Li, Zhewei Shi, Jiafeng Lin, Jinglin Zhao, Depu Zhou, Zheng Cheng, Teng Zhang, Qin Luo, Wenwu Zhang and Li Liu and has published in prestigious journals such as PLoS ONE, Scientific Reports and Biochemical and Biophysical Research Communications.

In The Last Decade

Ge Li‐Sha

30 papers receiving 510 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ge Li‐Sha China 14 223 219 85 75 68 32 514
Yumiko Takei Japan 7 190 0.9× 84 0.4× 26 0.3× 51 0.7× 100 1.5× 12 474
Luiz Ricardo C. Vasconcellos Brazil 7 180 0.8× 139 0.6× 20 0.2× 63 0.8× 69 1.0× 10 399
Olivia Osborne United States 8 195 0.9× 97 0.4× 78 0.9× 62 0.8× 222 3.3× 16 674
David Zhang United States 11 101 0.5× 105 0.5× 64 0.8× 22 0.3× 46 0.7× 28 439
Wenjun Deng China 14 147 0.7× 46 0.2× 130 1.5× 90 1.2× 91 1.3× 46 595
Stephanie Cicalese United States 11 170 0.8× 41 0.2× 57 0.7× 56 0.7× 36 0.5× 20 398
Xuehui Wang China 11 106 0.5× 78 0.4× 23 0.3× 50 0.7× 42 0.6× 34 355
Andrew Marshall United States 7 76 0.3× 65 0.3× 41 0.5× 127 1.7× 33 0.5× 17 411
Yasmine Aguib Egypt 10 351 1.6× 98 0.4× 88 1.0× 27 0.4× 152 2.2× 22 643
Sarah Rosli Australia 13 302 1.4× 131 0.6× 11 0.1× 138 1.8× 67 1.0× 19 513

Countries citing papers authored by Ge Li‐Sha

Since Specialization
Citations

This map shows the geographic impact of Ge Li‐Sha's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ge Li‐Sha with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ge Li‐Sha more than expected).

Fields of papers citing papers by Ge Li‐Sha

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ge Li‐Sha. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ge Li‐Sha. The network helps show where Ge Li‐Sha may publish in the future.

Co-authorship network of co-authors of Ge Li‐Sha

This figure shows the co-authorship network connecting the top 25 collaborators of Ge Li‐Sha. A scholar is included among the top collaborators of Ge Li‐Sha based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ge Li‐Sha. Ge Li‐Sha is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Xu, Jing, et al.. (2022). The Protective Effect of LCZ696 in Coxsackievirus B3-Induced Acute Viral Myocarditis Mice. ESC Heart Failure. 10(1). 366–376. 3 indexed citations
2.
Luo, Chunyan, Decheng Wang, Weifeng Huang, et al.. (2021). Feedback regulation of coronary artery disease susceptibility gene ADTRP and LDL receptors LDLR/CD36/LOX-1 in endothelia cell functions involved in atherosclerosis. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1867(7). 166130–166130. 9 indexed citations
3.
Li, Yuechun, Xiaohong Gu, Ge Li‐Sha, et al.. (2020). Vagus nerve plays a pivotal role in CD4+ T cell differentiation during CVB3-induced murine acute myocarditis. Virulence. 12(1). 360–376. 19 indexed citations
4.
Lin, Miaomiao, et al.. (2019). Efficacy of levofloxacin on macrolide-unresponsive and corticosteroid-resistant refractory Mycoplasma pneumoniae pneumonia in children. Annals of Palliative Medicine. 8(5). 632–639. 9 indexed citations
5.
Lin, Miaomiao, et al.. (2019). Misdiagnosis of scrub typhus complicated by hemophagocytic syndrome. BMC Pediatrics. 19(1). 102–102. 5 indexed citations
6.
Li‐Sha, Ge, Liu Li, Depu Zhou, et al.. (2018). Ivabradine Treatment Reduces Cardiomyocyte Apoptosis in a Murine Model of Chronic Viral Myocarditis. Frontiers in Pharmacology. 9. 182–182. 8 indexed citations
7.
Shi, Zhewei, Ge Li‐Sha, & Yuechun Li. (2018). The Role of Necroptosis in Cardiovascular Disease. Frontiers in Pharmacology. 9. 721–721. 118 indexed citations
8.
Li‐Sha, Ge, et al.. (2018). Role of Heart Rate Reduction in the Management of Myocarditis. Current Pharmaceutical Design. 24(3). 365–378. 10 indexed citations
9.
Gu, Xiaohong, Lulu Pan, Chunxiang Zhang, et al.. (2018). Melatonin Ameliorates Coxsackievirus B3-Induced Myocarditis by Regulating Apoptosis and Autophagy. Frontiers in Pharmacology. 9. 1384–1384. 19 indexed citations
10.
Zhang, Wenwu, et al.. (2017). Regulatory effects of PI3K/AKT2/mTOR signaling pathway on autophagy activation in cardiac tissues of mice with vial myocarditis. Zhonghua weishengwuxue he mianyixue zazhi. 37(12). 921–926. 1 indexed citations
11.
Li‐Sha, Ge, Xing-Xing Chen, Lianpin Wu, et al.. (2017). Right Cervical Vagotomy Aggravates Viral Myocarditis in Mice Via the Cholinergic Anti-inflammatory Pathway. Frontiers in Pharmacology. 8. 25–25. 23 indexed citations
12.
Li‐Sha, Ge, et al.. (2015). Dose-dependent protective effect of nicotine in a murine model of viral myocarditis induced by coxsackievirus B3. Scientific Reports. 5(1). 15895–15895. 21 indexed citations
13.
14.
Cheng, Zheng, Ge Li‐Sha, Jinglin Zhao, et al.. (2014). Protective Role of the Cholinergic Anti-Inflammatory Pathway in a Mouse Model of Viral Myocarditis. PLoS ONE. 9(11). e112719–e112719. 31 indexed citations
15.
16.
Li, Yuechun, et al.. (2013). Ivabradine inhibits the production of proinflammatory cytokines and inducible nitric oxide synthase in acute coxsackievirus B3-induced myocarditis. Biochemical and Biophysical Research Communications. 431(3). 450–455. 22 indexed citations
17.
Li, Yuechun, et al.. (2012). Comparison of Effects of Ivabradine versus Carvedilol in Murine Model with the Coxsackievirus B3-Induced Viral Myocarditis. PLoS ONE. 7(6). e39394–e39394. 36 indexed citations
18.
Li, Yuechun, et al.. (2011). Huge Negative T Waves Reaching a Depth of 36 mm Below the Isoelectric Line on ECG Associated with Myocardial Stunning. Journal of Cardiovascular Electrophysiology. 23(7). 784–785.
19.
Li, Yuechun, et al.. (2010). Establishment of a canine model of cardiac memory using endocardial pacing via internal jugular vein. BMC Cardiovascular Disorders. 10(1). 30–30. 1 indexed citations
20.
Li, Yuechun, et al.. (2008). Protective Effects of Carvedilol in Murine Model With the Coxsackievirus B3-Induced Viral Myocarditis. Journal of Cardiovascular Pharmacology. 51(1). 92–98. 21 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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