Gavin I. Ellis

667 total citations
10 papers, 444 citations indexed

About

Gavin I. Ellis is a scholar working on Immunology, Oncology and Genetics. According to data from OpenAlex, Gavin I. Ellis has authored 10 papers receiving a total of 444 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Immunology, 4 papers in Oncology and 4 papers in Genetics. Recurrent topics in Gavin I. Ellis's work include CAR-T cell therapy research (4 papers), T-cell and B-cell Immunology (4 papers) and Immune Cell Function and Interaction (4 papers). Gavin I. Ellis is often cited by papers focused on CAR-T cell therapy research (4 papers), T-cell and B-cell Immunology (4 papers) and Immune Cell Function and Interaction (4 papers). Gavin I. Ellis collaborates with scholars based in United States. Gavin I. Ellis's co-authors include James L. Riley, Colby R. Maldini, Neil C. Sheppard, Francesc Martí, Lianteng Zhi, Ravi Shankar Akundi, Hansruedi Büeler, A. Catalina Vélez-Ortega, Eric Rogier and Maria E. C. Bruno and has published in prestigious journals such as Nature reviews. Immunology, The Journal of Immunology and Nature Reviews Genetics.

In The Last Decade

Gavin I. Ellis

10 papers receiving 433 citations

Peers

Gavin I. Ellis

ONCOL

IMMUN

MB

GENET

BE

Xiangmin Wang China

Konrad Knöpper Germany

Radhika Kini United States

Coralie Viollet Germany

Brett S. Marro United States

Lauren E. Heusinkveld United States

Fabien Pitoiset France

Hiroyuki Takaba Japan

Martin Senekowitsch Austria

Xiangmin Wang China

Gavin I. Ellis

176 ×0.7

ONCOL

129 ×0.7

IMMUN

183 ×1.3

MB

45 ×0.5

GENET

30 ×0.5

BE

Citations per year, relative to Gavin I. Ellis Gavin I. Ellis (= 1×) peers Xiangmin Wang

Countries citing papers authored by Gavin I. Ellis

Since Specialization

Citations

This map shows the geographic impact of Gavin I. Ellis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gavin I. Ellis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gavin I. Ellis more than expected).

Fields of papers citing papers by Gavin I. Ellis

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gavin I. Ellis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gavin I. Ellis. The network helps show where Gavin I. Ellis may publish in the future.

Co-authorship network of co-authors of Gavin I. Ellis

This figure shows the co-authorship network connecting the top 25 collaborators of Gavin I. Ellis. A scholar is included among the top collaborators of Gavin I. Ellis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Gavin I. Ellis. Gavin I. Ellis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

1.

Ellis, Gavin I., et al.. (2022). Generation of non-human primate CAR Tregs using artificial antigen-presenting cells, simian tropic lentiviral vectors, and antigen-specific restimulation. STAR Protocols. 3(4). 101784–101784. 2 indexed citations

2.

Ellis, Gavin I., Vijay Bhoj, Michael C. Milone, et al.. (2022). Trafficking and persistence of alloantigen-specific chimeric antigen receptor regulatory T cells in Cynomolgus macaque. Cell Reports Medicine. 3(5). 100614–100614. 19 indexed citations

3.

Ellis, Gavin I., et al.. (2022). HIV-1–Infected CD4+ T Cells Present MHC Class II–Restricted Epitope via Endogenous Processing. The Journal of Immunology. 209(5). 864–873. 4 indexed citations

4.

Ellis, Gavin I., Neil C. Sheppard, & James L. Riley. (2021). Genetic engineering of T cells for immunotherapy. Nature Reviews Genetics. 22(7). 427–447. 107 indexed citations

5.

Maldini, Colby R., Gavin I. Ellis, & James L. Riley. (2018). CAR T cells for infection, autoimmunity and allotransplantation. Nature reviews. Immunology. 18(10). 605–616. 175 indexed citations

6.

Rowe, Rachel K., Gavin I. Ellis, Jordan L. Harrison, et al.. (2016). Diffuse traumatic brain injury induces prolonged immune dysregulation and potentiates hyperalgesia following a peripheral immune challenge. Molecular Pain. 12. 34 indexed citations

7.

Bruno, Maria E. C., Eric Rogier, Razvan Arsenescu, et al.. (2015). Correlation of Biomarker Expression in Colonic Mucosa with Disease Phenotype in Crohn’s Disease and Ulcerative Colitis. Digestive Diseases and Sciences. 60(10). 2976–2984. 33 indexed citations

8.

Ellis, Gavin I., Lianteng Zhi, Ravi Shankar Akundi, Hansruedi Büeler, & Francesc Martí. (2013). Mitochondrial and cytosolic roles of PINK1 shape induced regulatory T‐cell development and function. European Journal of Immunology. 43(12). 3355–3360. 34 indexed citations

9.

Vélez-Ortega, A. Catalina, et al.. (2012). Enhanced Generation of Suppressor T Cells in Patients with Asthma Taking Oral Contraceptives. Journal of Asthma. 50(3). 223–230. 12 indexed citations

10.

Ellis, Gavin I., et al.. (2012). Generation of Induced Regulatory T Cells from Primary Human Naïve and Memory T Cells. Journal of Visualized Experiments. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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