Gabriel Márquez

5.6k total citations · 1 hit paper
77 papers, 4.6k citations indexed

About

Gabriel Márquez is a scholar working on Immunology, Oncology and Molecular Biology. According to data from OpenAlex, Gabriel Márquez has authored 77 papers receiving a total of 4.6k indexed citations (citations by other indexed papers that have themselves been cited), including 41 papers in Immunology, 29 papers in Oncology and 23 papers in Molecular Biology. Recurrent topics in Gabriel Márquez's work include T-cell and B-cell Immunology (27 papers), Chemokine receptors and signaling (25 papers) and Immunotherapy and Immune Responses (24 papers). Gabriel Márquez is often cited by papers focused on T-cell and B-cell Immunology (27 papers), Chemokine receptors and signaling (25 papers) and Immunotherapy and Immune Responses (24 papers). Gabriel Márquez collaborates with scholars based in Spain, Cuba and Sweden. Gabriel Márquez's co-authors include William W. Agace, Marcus Svensson, Ángel Zaballos, Rosa Varona, Julio Gutiérrez, Bengt Johansson‐Lindbom, Carlos Martı́nez-A, Laura Carramolino, Carlos Ardavı́n and Jan Marsal and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

Gabriel Márquez

76 papers receiving 4.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Functional specialization of gut CD103 + dendritic cells in the regulation of tissue-selective T cell homing

2005 542 citations Bengt Johansson‐Lindbom, Marcus Svensson et al. The Journal of Experimental Medicine profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Gabriel Márquez Spain	35	2.9k	1.3k	977	351	290	77	4.6k
WanJun Chen United States	16	3.5k 1.2×	1.1k 0.9×	1.0k 1.0×	381 1.1×	266 0.9×	18	5.5k
M. Stangassinger Germany	30	1.6k 0.5×	767 0.6×	1.1k 1.2×	270 0.8×	227 0.8×	100	4.0k
Mikael Dohlsten Sweden	39	3.4k 1.2×	846 0.7×	952 1.0×	278 0.8×	199 0.7×	142	4.6k
Giovanna D’Amico Italy	28	3.2k 1.1×	1.3k 1.0×	886 0.9×	225 0.6×	290 1.0×	95	4.8k
Seema S. Ahuja United States	40	2.2k 0.8×	979 0.8×	918 0.9×	322 0.9×	200 0.7×	71	4.4k
Michio Tomura Japan	40	3.9k 1.3×	1.4k 1.1×	1.5k 1.5×	227 0.6×	161 0.6×	90	5.7k
Carlos Ardavı́n Spain	38	5.1k 1.8×	958 0.7×	1.1k 1.1×	236 0.7×	151 0.5×	79	6.3k
Terje Kalland Sweden	39	2.8k 1.0×	677 0.5×	799 0.8×	398 1.1×	226 0.8×	143	4.0k
Michael Lohoff Germany	35	4.4k 1.5×	1.1k 0.8×	844 0.9×	271 0.8×	404 1.4×	88	6.1k
Harumichi Ishigame Japan	18	3.4k 1.2×	739 0.6×	768 0.8×	344 1.0×	214 0.7×	28	4.7k

Countries citing papers authored by Gabriel Márquez

Since Specialization

Citations

This map shows the geographic impact of Gabriel Márquez's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gabriel Márquez with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gabriel Márquez more than expected).

Fields of papers citing papers by Gabriel Márquez

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gabriel Márquez. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gabriel Márquez. The network helps show where Gabriel Márquez may publish in the future.

Co-authorship network of co-authors of Gabriel Márquez

This figure shows the co-authorship network connecting the top 25 collaborators of Gabriel Márquez. A scholar is included among the top collaborators of Gabriel Márquez based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Gabriel Márquez. Gabriel Márquez is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

1.

Huerta, Vivian, Yassel Ramos, Gabriel Márquez, et al.. (2015). Dataset on protein composition of a human plasma sub-proteome able to modulate the Dengue 2 virus infection in Huh 7.5 cells. Data in Brief. 6. 352–358. 5 indexed citations

2.

Lopes, André Moreni, et al.. (2013). Adsorption of endotoxins on Ca2+ iminodiacetic acid by metal ion affinity chromatography. Chinese Journal of Chromatography. 30(11). 1194–1202. 1 indexed citations

3.

Limonta‐Fernández, Miladys, et al.. (2012). Estudios fisiológicos y nutricionales de la Escherichia coli y la combinación novedosa de métodos de separación permiten obtener ADN plasmídico con alta pureza y homogeneidad para su uso en terapia génica. Biotecnología aplicada. 29(4). 271–274. 1 indexed citations

4.

Valdés, Jorge, et al.. (2009). Improving the expression of Human Epidermal Growth Factor in Saccharomyces cerevisiae by manipulating culture conditions. SHILAP Revista de lepidopterología. 26(1). 34–38. 6 indexed citations

5.

Valdés, Jorge, et al.. (2009). Physiological study in Saccharomyces cerevisiae for overproduction of a homogeneous human epidermal growth factor molecule. SHILAP Revista de lepidopterología. 26(2). 166–167. 2 indexed citations

6.

Mira, E., Beatriz León, Domingo F. Barber, et al.. (2008). Statins Induce Regulatory T Cell Recruitment via a CCL1 Dependent Pathway. The Journal of Immunology. 181(5). 3524–3534. 75 indexed citations

7.

Varona, Rosa, et al.. (2006). CCR6 regulates the function of alloreactive and regulatory CD4⁺T cells during acute graft-versus-host disease. Leukemia & lymphoma. 47(8). 1469–1476. 20 indexed citations

8.

Johansson‐Lindbom, Bengt, Marcus Svensson, Oliver Pabst, et al.. (2005). Functional specialization of gut CD103 + dendritic cells in the regulation of tissue-selective T cell homing. The Journal of Experimental Medicine. 202(8). 1063–1073. 542 indexed citations breakdown →

9.

Zaballos, Ángel, Ricardo Villares, Juan Pablo Albar, Carlos Martı́nez-A, & Gabriel Márquez. (2004). Identification on Mouse Chromosome 8 of New β-Defensin Genes with Regionally Specific Expression in the Male Reproductive Organ. Journal of Biological Chemistry. 279(13). 12421–12426. 31 indexed citations

10.

Svensson, Marcus, Bengt Johansson‐Lindbom, Marc‐André Wurbel, et al.. (2004). Selective Generation of Gut‐Tropic T Cells in Gut‐Associated Lymphoid Tissues: Requirement for GALT Dendritic Cells and Adjuvant. Annals of the New York Academy of Sciences. 1029(1). 405–407. 10 indexed citations

11.

Goya, Íñigo, Ricardo Villares, Ángel Zaballos, et al.. (2003). Absence of CCR8 Does Not Impair the Response to Ovalbumin-Induced Allergic Airway Disease. The Journal of Immunology. 170(4). 2138–2146. 69 indexed citations

12.

Zhao, Xinyan, Ayuko Sato, Charles S. Dela Cruz, et al.. (2003). CCL9 Is Secreted by the Follicle-Associated Epithelium and Recruits Dome Region Peyer’s Patch CD11b+ Dendritic Cells. The Journal of Immunology. 171(6). 2797–2803. 146 indexed citations

13.

Svensson, Marcus, Jan Marsal, Anna Ericsson, et al.. (2002). CCL25 mediates the localization of recently activated CD8αβ+ lymphocytes to the small-intestinal mucosa. Journal of Clinical Investigation. 110(8). 1113–1121. 235 indexed citations

14.

Kremer, Leonor, Laura Carramolino, Íñigo Goya, et al.. (2001). The Transient Expression of C-C Chemokine Receptor 8 in Thymus Identifies a Thymocyte Subset Committed to Become CD4+ Single-Positive T Cells. The Journal of Immunology. 166(1). 218–225. 32 indexed citations

15.

Varona, Rosa, Ricardo Villares, Laura Carramolino, et al.. (2001). CCR6-deficient mice have impaired leukocyte homeostasis and altered contact hypersensitivity and delayed-type hypersensitivity responses. Journal of Clinical Investigation. 107(6). R37–R45. 180 indexed citations

16.

Ferrero, Isabel, Fabienne Anjuère, Pilar Martı́n, et al.. (1999). Functional and phenotypic analysis of thymic B cells: role in the induction of T cell negative selection. European Journal of Immunology. 29(5). 1598–1609. 3 indexed citations

17.

Ferrero, Isabel, Fabienne Anjuère, Pilar Martı́n, et al.. (1999). Functional and phenotypic analysis of thymic B cells: role in the induction of T cell negative selection. European Journal of Immunology. 29(5). 1598–1609. 47 indexed citations

18.

Mira, E., Santos Mañes, Rosa Ana Lacalle, Gabriel Márquez, & Carlos Martı́nez-A. (1999). Insulin-Like Growth Factor I-Triggered Cell Migration and Invasion Are Mediated by Matrix Metalloproteinase-9¹. Endocrinology. 140(4). 1657–1664. 83 indexed citations

19.

Goya, Íñigo, Julio Gutiérrez, Rosa Varona, et al.. (1998). Identification of CCR8 as the Specific Receptor for the Human β-Chemokine I-309: Cloning and Molecular Characterization of Murine CCR8 as the Receptor for TCA-3. The Journal of Immunology. 160(4). 1975–1981. 71 indexed citations

20.

Carramolino, Laura, Ángel Zaballos, Amnon Peled, et al.. (1997). SA-1, a nuclear protein encoded by one member of a novel gene family: molecular cloning and detection in hemopoietic organs. Gene. 195(2). 151–159. 38 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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