Gábor Hild
- Cell Biology top 5%
- Cellular Mechanics and Interactions 25
- Biophysics top 2%
- Advanced Fluorescence Microscopy Techniques 10
-
- Cardiomyopathy and Myosin Studies 17
- Cardiovascular Effects of Exercise 3
- Surfaces, Coatings and Films top 10%
-
- Force Microscopy Techniques and Applications 12
-
- Muscle Physiology and Disorders 5
- Ion channel regulation and function 3
-
- thermodynamics and calorimetric analyses 5
Gábor Hild
40 papers receiving 711 citations
Peers
Comparison fields: 5 of 84
- Cell Biology 442
- Biophysics 133
- Cardiology and Cardiovascular Medicine 262
- Structural Biology 12
- Surfaces, Coatings and Films 52
Countries citing papers authored by Gábor Hild
This map shows the geographic impact of Gábor Hild's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gábor Hild with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gábor Hild more than expected).
Fields of papers citing papers by Gábor Hild
This network shows the impact of papers produced by Gábor Hild. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gábor Hild. The network helps show where Gábor Hild may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Gábor Hild, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2023 | 1 | |
| 2 | 2016 | 2 | |
| 3 | 2016 | 6 | |
| 4 | 2014 | 8 | |
| 5 | 2014 | 37 | |
| 6 | 2013 | 12 | |
| 7 | 2012 | 17 | |
| 8 | 2010 | 40 | |
| 9 | 2010 | 33 | |
| 10 | 2009 | 13 | |
| 11 | 2009 | 11 | |
| 12 | 2008 | 15 | |
| 13 | 2007 | 4 | |
| 14 | 2006 | 47 | |
| 15 | 2006 | 55 | |
| 16 | 2005 | 12 | |
| 17 | 2004 | 67 | |
| 18 | 2002 | 17 | |
| 19 | 2000 | 12 | |
| 20 | 1997 | 26 |
About Gábor Hild
Gábor Hild is a scholar working on Biophysics, Cell Biology, Cardiology and Cardiovascular Medicine, Physical and Theoretical Chemistry and Atomic and Molecular Physics, and Optics, having authored 40 papers that have together received 723 indexed citations. Recurring topics across this work include Cellular Mechanics and Interactions (25 papers), Cardiomyopathy and Myosin Studies (17 papers), Force Microscopy Techniques and Applications (12 papers), Advanced Fluorescence Microscopy Techniques (10 papers), Muscle Physiology and Disorders (5 papers), thermodynamics and calorimetric analyses (5 papers), Cardiovascular Effects of Exercise (3 papers) and Ion channel regulation and function (3 papers). The work is most often cited by research in Cell Biology (442 citations), Biophysics (133 citations), Cardiology and Cardiovascular Medicine (262 citations), Structural Biology (12 citations) and Surfaces, Coatings and Films (52 citations). Gábor Hild has collaborated with scholars based in Hungary, United States and Finland. Frequent co-authors include Miklós Nyitrai, Béla Somogyi, Dénes Lôrinczy, Beáta Bugyi, Balázs Visegrády, Zoltán Ujfalusi, B Somogyi, Gábor Papp, Elisa Nevalainen and József Belágyi. Their work appears in journals such as Biophysical Journal, Journal of Biological Chemistry, FEBS Letters, Journal of Photochemistry and Photobiology B Biology and Journal of Thermal Analysis and Calorimetry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.