G. Tim Bowden

2.1k total citations
40 papers, 1.7k citations indexed

About

G. Tim Bowden is a scholar working on Molecular Biology, Cancer Research and Dermatology. According to data from OpenAlex, G. Tim Bowden has authored 40 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 26 papers in Molecular Biology, 17 papers in Cancer Research and 8 papers in Dermatology. Recurrent topics in G. Tim Bowden's work include Protease and Inhibitor Mechanisms (8 papers), Skin Protection and Aging (7 papers) and NF-κB Signaling Pathways (7 papers). G. Tim Bowden is often cited by papers focused on Protease and Inhibitor Mechanisms (8 papers), Skin Protection and Aging (7 papers) and NF-κB Signaling Pathways (7 papers). G. Tim Bowden collaborates with scholars based in United States, Germany and France. G. Tim Bowden's co-authors include Weixing Chen, Zigang Dong, Marianne Broome Powell, Jack Zhang, Raymond B. Nagle, Anne E. Cress, Melissa Gonzales, Frederick E. Domann, Joanne S. Finch and Isis C. Sroka and has published in prestigious journals such as Journal of Biological Chemistry, Cancer Research and Oncogene.

In The Last Decade

G. Tim Bowden

40 papers receiving 1.7k citations

Peers

G. Tim Bowden

ONCOL

DERMA

G. Tim Bowden United States

Arianna L. Kim United States

Sivaprakasam Balasubramanian United States

Wen‐Sheng Wu Taiwan

Richard M. Niles United States

Ke Yao United States

Xiuli Yi China

Sen Guo China

Baskaran Govindarajan United States

Weinan Guo China

G. Tim Bowden United States

G. Tim Bowden

1.1k ×1.2

568 ×1.5

ONCOL

273 ×0.8

DERMA

562 ×1.6

231 ×0.9

Citations per year, relative to G. Tim Bowden G. Tim Bowden (= 1×) peers G. Tim Bowden

Countries citing papers authored by G. Tim Bowden

Since Specialization

Citations

This map shows the geographic impact of G. Tim Bowden's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by G. Tim Bowden with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites G. Tim Bowden more than expected).

Fields of papers citing papers by G. Tim Bowden

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by G. Tim Bowden. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by G. Tim Bowden. The network helps show where G. Tim Bowden may publish in the future.

Co-authorship network of co-authors of G. Tim Bowden

This figure shows the co-authorship network connecting the top 25 collaborators of G. Tim Bowden. A scholar is included among the top collaborators of G. Tim Bowden based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with G. Tim Bowden. G. Tim Bowden is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Liu, Kangdong, Donghoon Yu, Yong‐Yeon Cho, et al.. (2013). Sunlight UV-Induced Skin Cancer Relies upon Activation of the p38α Signaling Pathway. Cancer Research. 73(7). 2181–2188. 53 indexed citations

Dickinson, Sally E., Erik R. Olson, Jack Zhang, et al.. (2011). p38 MAP kinase plays a functional role in UVB‐Induced mouse skin carcinogenesis. Molecular Carcinogenesis. 50(6). 469–478. 51 indexed citations

Zhang, Jack & G. Tim Bowden. (2008). UVB irradiation regulates Cox‐2 mRNA stability through AMPK and HuR in human keratinocytes. Molecular Carcinogenesis. 47(12). 974–983. 39 indexed citations

Sroka, Isis C., Kathy McDaniel, Raymond B. Nagle, & G. Tim Bowden. (2008). Differential localization of MT1‐MMP in human prostate cancer tissue: Role of IGF‐1R in MT1‐MMP expression. The Prostate. 68(5). 463–476. 18 indexed citations

Nagle, Raymond B., et al.. (2003). Fibroblast growth factor‐1 transcriptionally induces membrane type‐1 matrix metalloproteinase expression in prostate carcinoma cell line. The Prostate. 58(1). 66–75. 32 indexed citations

Gonzales, Melissa & G. Tim Bowden. (2002). The role of PI 3-kinase in the UVB-induced expression of c-fos. Oncogene. 21(17). 2721–2728. 37 indexed citations

Klein, Russell D., et al.. (1999). Promatrilysin expression is induced by fibroblast growth factors in the prostatic carcinoma cell line LNCaP but not in normal primary prostate epithelial cells. The Prostate. 41(4). 215–223. 20 indexed citations

Sundareshan, Padma, Raymond B. Nagle, & G. Tim Bowden. (1999). EGF induces the expression of matrilysin in the human prostate adenocarcinoma cell line, LNCaP. The Prostate. 40(3). 159–166. 15 indexed citations

Chen, Weixing & G. Tim Bowden. (1999). Activation of p38 MAP kinase and ERK are required for ultraviolet-B induced c-fos gene expression in human keratinocytes. Oncogene. 18(52). 7469–7476. 88 indexed citations

10.

Powell, Marianne Broome, et al.. (1999). Induction of melanoma in TPras transgenic mice. Carcinogenesis. 20(9). 1747–1753. 80 indexed citations

11.

Chen, Weixing, et al.. (1998). UVB Irradiation-induced Activator Protein-1 Activation Correlates with Increased c-fos Gene Expression in a Human Keratinocyte Cell Line. Journal of Biological Chemistry. 273(48). 32176–32181. 82 indexed citations

12.

Bowden, G. Tim & Susan M. Fischer. (1997). Chemical carcinogens and anticarcinogens. Pergamon eBooks. 13 indexed citations

13.

Bowden, G. Tim, et al.. (1997). Regulation of matrilysin expression in cells of squamous cell carcinoma by E-cadherin-mediated cell-cell contact. Journal of Cancer Research and Clinical Oncology. 123(1). 13–20. 21 indexed citations

14.

Joseloff, Elizabeth & G. Tim Bowden. (1997). Regulation of the transcription factor AP-1 in benign and malignant mouse keratinocyte cells. Molecular Carcinogenesis. 18(1). 26–36. 9 indexed citations

15.

Knox, J. David, Catherine Wolf, Kathleen McDaniel, et al.. (1996). Matrilysin expression in human prostate carcinoma. Molecular Carcinogenesis. 15(1). 57–63. 70 indexed citations

16.

Finch, Joanne S., et al.. (1996). Quantitation of early clonal expansion of two mutant 61st codon c-Ha-ras alleles in DMBA/TPA treated mouse skin by nested PCR/RFLP. Carcinogenesis. 17(12). 2551–2557. 32 indexed citations

17.

Powell, Marianne Broome, et al.. (1995). Hyperpigmentation and melanocytic hyperplasia in transgenic mice expressing the human T24 HA‐ras gene regulated by a mouse tyrosinase promoter. Molecular Carcinogenesis. 12(2). 82–90. 63 indexed citations

18.

Domann, Frederick E., John Levy, Joanne S. Finch, & G. Tim Bowden. (1994). Constitutive ap‐1 dna binding and transactivating ability of malignant but not benign mouse epidermal cells. Molecular Carcinogenesis. 9(2). 61–66. 58 indexed citations

19.

Schneider, Brandt L., et al.. (1993). 7,12-Dimethylbenz [a] anthracene-Induced Mouse Keratinocyte Malignant Transformation Independent of Harvey ras Activation. Journal of Investigative Dermatology. 101(4). 595–599. 16 indexed citations

20.

Craig, Ann Marie, G. Tim Bowden, Ann F. Chambers, et al.. (1990). Secreted phosphoprotein mrna is induced during multi‐stage carcinogenesis in mouse skin and correlates with the metastatic potential of murine fibroblasts. International Journal of Cancer. 46(1). 133–137. 108 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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