G. P. Hamlin

773 total citations
19 papers, 624 citations indexed

About

G. P. Hamlin is a scholar working on Molecular Biology, Endocrinology, Diabetes and Metabolism and Small Animals. According to data from OpenAlex, G. P. Hamlin has authored 19 papers receiving a total of 624 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 4 papers in Endocrinology, Diabetes and Metabolism and 3 papers in Small Animals. Recurrent topics in G. P. Hamlin's work include Growth Hormone and Insulin-like Growth Factors (4 papers), Pregnancy and preeclampsia studies (3 papers) and Neuroendocrine regulation and behavior (3 papers). G. P. Hamlin is often cited by papers focused on Growth Hormone and Insulin-like Growth Factors (4 papers), Pregnancy and preeclampsia studies (3 papers) and Neuroendocrine regulation and behavior (3 papers). G. P. Hamlin collaborates with scholars based in Australia, United States and Poland. G. P. Hamlin's co-authors include Michael J. Soares, L. A. Fitzpatrick, Charles Coleman, A. J. Parker, Katherine F. Roby, Ibolja Černak, Robert Vink, Xuancheng Lu, Julie A. Wixey and Robert Coelen and has published in prestigious journals such as Journal of Biological Chemistry, Endocrinology and Biology of Reproduction.

In The Last Decade

G. P. Hamlin

18 papers receiving 594 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
G. P. Hamlin Australia 14 183 111 102 91 86 19 624
Jeffrey D Fortman United States 17 148 0.8× 56 0.5× 18 0.2× 128 1.4× 72 0.8× 50 957
John Slevin Ireland 9 73 0.4× 103 0.9× 17 0.2× 47 0.5× 68 0.8× 18 337
Jeffrey F. Trahair Australia 17 145 0.8× 61 0.5× 132 1.3× 15 0.2× 53 0.6× 34 872
João Francisco Coelho de Oliveira Brazil 17 121 0.7× 60 0.5× 54 0.5× 335 3.7× 52 0.6× 57 831
R. A. S. Welch United States 18 111 0.6× 92 0.8× 22 0.2× 120 1.3× 92 1.1× 37 962
Peter Daels United States 23 178 1.0× 102 0.9× 83 0.8× 416 4.6× 68 0.8× 99 1.5k
Kimberly A. Greer United States 16 214 1.2× 16 0.1× 30 0.3× 77 0.8× 46 0.5× 29 1000
G.C.B. Randall Canada 15 100 0.5× 122 1.1× 28 0.3× 230 2.5× 14 0.2× 36 838
J. Martinet France 18 89 0.5× 99 0.9× 55 0.5× 75 0.8× 7 0.1× 67 934
Thomas J. Reimers United States 15 44 0.2× 68 0.6× 141 1.4× 53 0.6× 28 0.3× 22 610

Countries citing papers authored by G. P. Hamlin

Since Specialization
Citations

This map shows the geographic impact of G. P. Hamlin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by G. P. Hamlin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites G. P. Hamlin more than expected).

Fields of papers citing papers by G. P. Hamlin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by G. P. Hamlin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by G. P. Hamlin. The network helps show where G. P. Hamlin may publish in the future.

Co-authorship network of co-authors of G. P. Hamlin

This figure shows the co-authorship network connecting the top 25 collaborators of G. P. Hamlin. A scholar is included among the top collaborators of G. P. Hamlin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with G. P. Hamlin. G. P. Hamlin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Parker, A. J., G. P. Hamlin, Charles Coleman, & L. A. Fitzpatrick. (2004). Excess cortisol interferes with a principal mechanism of resistance to dehydration in Bos indicus steers1. Journal of Animal Science. 82(4). 1037–1045. 20 indexed citations
2.
Parker, A. J., G. P. Hamlin, Charles Coleman, & L. A. Fitzpatrick. (2004). Excess cortisol interferes with a principal mechanism of resistance to dehydration in Bos indicus steers1. Journal of Animal Science. 82(4). 1037–1045.
3.
Parker, A. J., G. P. Hamlin, Charles Coleman, & L. A. Fitzpatrick. (2003). Quantitative analysis of acid-base balance in Bos indicus steers subjected to transportation of long duration1. Journal of Animal Science. 81(6). 1434–1439. 62 indexed citations
4.
Parker, A. J., G. P. Hamlin, Charles Coleman, & L. A. Fitzpatrick. (2003). Dehydration in stressed ruminants may be the result of acortisol-induced diuresis1. Journal of Animal Science. 81(2). 512–519. 64 indexed citations
5.
Williams, Michael, et al.. (2003). Circular versus longitudinal myometrial contractile response to selective tachykinin receptor agonists in rat. Reproduction Fertility and Development. 15(6). 311–316. 3 indexed citations
6.
Nimmo, Alan, et al.. (2002). Estrogen-Dependent Regulation of Neurokinin 3 Receptor-Mediated Uterine Contractility in the Rat1. Biology of Reproduction. 67(5). 1480–1487. 15 indexed citations
7.
Černak, Ibolja, et al.. (2002). Temporal characterisation of pro- and anti-apoptotic mechanisms following diffuse traumatic brain injury in rats. Journal of Clinical Neuroscience. 9(5). 565–572. 38 indexed citations
8.
Hamlin, G. P., Ibolja Černak, Julie A. Wixey, & Robert Vink. (2001). Increased Expression of Neuronal Glucose Transporter 3 but Not Glial Glucose Transporter 1 Following Severe Diffuse Traumatic Brain Injury in Rats. Journal of Neurotrauma. 18(10). 1011–1018. 57 indexed citations
9.
Hamlin, G. P., et al.. (2000). Hormonal Variation of Rat Uterine Contractile Responsiveness to Selective Neurokinin Receptor Agonists1. Biology of Reproduction. 62(6). 1661–1666. 22 indexed citations
10.
Soden, Muriel, et al.. (2000). Detection of viral ribonucleic acid and histologic analysis of inflamed synovium in Ross River virus infection. Arthritis & Rheumatism. 43(2). 365–365. 83 indexed citations
11.
Peters, Thomas, Estela Bevilacqua, B. M. Chapman, et al.. (1999). Differentiation-dependent expression of gelatinase B/matrix metalloproteinase-9 in trophoblast cells. Cell and Tissue Research. 295(2). 287–296. 33 indexed citations
12.
Kamei, Takayuki, G. P. Hamlin, B. M. Chapman, et al.. (1997). Signaling Pathways Controlling Trophoblast Cell Differentiation: Src Family Protein Tyrosine Kinases in the Rat1. Biology of Reproduction. 57(6). 1302–1311. 20 indexed citations
15.
Roby, Katherine F., G. P. Hamlin, Michael J. Soares, & John A. Hunt. (1994). Differential responses of phenotypically distinct rat trophoblast cell lines to MHC class I antigen-inducing cytokines. Placenta. 15(6). 577–590. 10 indexed citations
16.
Hamlin, G. P., Xuancheng Lu, Katherine F. Roby, & Michael J. Soares. (1994). Recapitulation of the pathway for trophoblast giant cell differentiation in vitro: stage-specific expression of members of the prolactin gene family.. Endocrinology. 134(6). 2390–2396. 78 indexed citations
17.
Deb, Shoumitro, G. P. Hamlin, Katherine F. Roby, Simon C.M. Kwok, & M.J. Soares. (1993). Heterologous expression and characterization of prolactin-like protein-A. Identification of serum binding proteins.. Journal of Biological Chemistry. 268(5). 3298–3305. 26 indexed citations
18.
Waters, Michael J., Ross Barnard, PE Lobie, et al.. (1990). Growth Hormone Receptors ‐ their Structure, Location and Role. Acta Paediatrica. 79(s366). 60–72. 47 indexed citations
19.
Spencer, Steven A., et al.. (1990). Purification and partial sequence of the rabbit mammary gland prolactin receptor. International Journal of Biochemistry. 22(10). 1089–1095. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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