G. Koller

767 total citations
21 papers, 564 citations indexed

About

G. Koller is a scholar working on Cellular and Molecular Neuroscience, Neurology and Clinical Psychology. According to data from OpenAlex, G. Koller has authored 21 papers receiving a total of 564 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Cellular and Molecular Neuroscience, 5 papers in Neurology and 4 papers in Clinical Psychology. Recurrent topics in G. Koller's work include Neurotransmitter Receptor Influence on Behavior (6 papers), Substance Abuse Treatment and Outcomes (3 papers) and Alcoholism and Thiamine Deficiency (3 papers). G. Koller is often cited by papers focused on Neurotransmitter Receptor Influence on Behavior (6 papers), Substance Abuse Treatment and Outcomes (3 papers) and Alcoholism and Thiamine Deficiency (3 papers). G. Koller collaborates with scholars based in Germany, United States and Austria. G. Koller's co-authors include Ulrich W. Preuss, Michael Soyka, M. Bottlender, Brigitta Bondy, Kevin Wenzel, Dan Rujescu, Peter Zill, Ina Giegling, Dirk Andreas Zetzsche and Stefan Watzke and has published in prestigious journals such as Journal of Bacteriology, Journal of Affective Disorders and IEEE Transactions on Evolutionary Computation.

In The Last Decade

G. Koller

20 papers receiving 547 citations

Peers

G. Koller
Henry R. Kranzler United States
Geoffrey R. Twitchell United States
M. Fichter Germany
Travis Wearne Australia
P. Courtet France
Anita Hole United States
Marc Graf Switzerland
Henry R. Kranzler United States
G. Koller
Citations per year, relative to G. Koller G. Koller (= 1×) peers Henry R. Kranzler

Countries citing papers authored by G. Koller

Since Specialization
Citations

This map shows the geographic impact of G. Koller's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by G. Koller with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites G. Koller more than expected).

Fields of papers citing papers by G. Koller

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by G. Koller. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by G. Koller. The network helps show where G. Koller may publish in the future.

Co-authorship network of co-authors of G. Koller

This figure shows the co-authorship network connecting the top 25 collaborators of G. Koller. A scholar is included among the top collaborators of G. Koller based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with G. Koller. G. Koller is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Caneo, Constanza, et al.. (2024). Tooth loss and depression in Chilean participants of the National Health Survey 2016–2017: Oral and social functions mediation analysis. Journal of Affective Disorders. 358. 19–27. 4 indexed citations
2.
Falkai, Peter, et al.. (2020). 12- and 18-month follow-up after residential treatment of methamphetamine dependence: Influence of treatment drop-out and different treatment concepts. Journal of Psychiatric Research. 129. 103–110. 6 indexed citations
3.
Moñux, Guillermo, José J. Zamorano‐León, Pablo Marqués de Marino, et al.. (2017). FXa inhibition by rivaroxaban modifies mechanisms associated with the pathogenesis of human abdominal aortic aneurysms. British Journal of Clinical Pharmacology. 83(12). 2661–2670. 25 indexed citations
4.
Preuss, Ulrich W., et al.. (2016). Treatment of behavioral and psychological symptoms of dementia: a systematic review. Psychiatria Polska. 50(4). 679–715. 39 indexed citations
5.
Preuss, Ulrich W., F. Wurst, Monika Ridinger, et al.. (2013). Association of functional DBH genetic variants with alcohol dependence risk and related depression and suicide attempt phenotypes: Results from a large multicenter association study. Drug and Alcohol Dependence. 133(2). 459–467. 22 indexed citations
6.
Koller, G.. (2013). Langzeittherapie der Alkoholabhängigkeit. Fortschritte der Neurologie · Psychiatrie. 81(11). 655–666.
7.
Preuss, Ulrich W., Monika Ridinger, Dan Rujescu, et al.. (2010). No association of alcohol dependence with HOMER 1 and 2 genetic variants. American Journal of Medical Genetics Part B Neuropsychiatric Genetics. 153B(5). 1102–1109. 4 indexed citations
8.
Preuss, Ulrich W., Monika Johann, Christoph Fehr, et al.. (2009). Personality Disorders in Alcohol-Dependent Individuals: Relationship with Alcohol Dependence Severity. European Addiction Research. 15(4). 188–195. 39 indexed citations
9.
Preuss, U. W., Peter Zill, G. Koller, Brigitta Bondy, & Michael Soyka. (2007). D2 dopamine receptor gene haplotypes and their influence on alcohol and tobacco consumption magnitude in alcohol-dependent individuals. Alcohol and Alcoholism. 42(3). 258–266. 24 indexed citations
10.
Preuss, Ulrich W., Dan Rujescu, Ina Giegling, et al.. (2007). Psychometrische Evaluation der deutschsprachigen Version der Barratt-Impulsiveness-Skala. Der Nervenarzt. 79(3). 305–319. 129 indexed citations
11.
Raidl, Günther R., G. Koller, & Bryant A. Julstrom. (2006). Biased mutation operators for subgraph-selection problems. IEEE Transactions on Evolutionary Computation. 10(2). 145–156. 22 indexed citations
12.
Koller, G., et al.. (2005). Tryptophan hydroxylase gene 1 polymorphisms are not associated with suicide attempts in alcohol‐dependent individuals. Addiction Biology. 10(3). 269–273. 13 indexed citations
13.
Preuss, U. W., Peter Zill, G. Koller, et al.. (2005). Ionotropic glutamate receptor gene GRIK3 SER310ALA functional polymorphism is related to delirium tremens in alcoholics. The Pharmacogenomics Journal. 6(1). 34–41. 27 indexed citations
15.
Tadić, A., Norbert Dahmen, Armin Szegedi, et al.. (2004). Polymorphisms in the NMDA subunit 2B are not associated with alcohol dependence and alcohol withdrawal?induced seizures and delirium tremens. European Archives of Psychiatry and Clinical Neuroscience. 255(2). 129–135. 17 indexed citations
16.
Preuss, U. W., et al.. (2004). TRYPTOPHAN HYDROXYLASE GENE POLYMORPHISMS ARE NOT ASSOCIATED WITH SUICIDE ATTEMPTS IN ALCOHOL DEPENDENT INDIVIDUALS.. Alcoholism Clinical and Experimental Research. 28(Supplement). 31A–31A. 1 indexed citations
17.
Koller, G., et al.. (2003). NO ASSOCIATION BETWEEN A POLYMORPHISM IN THE PROMOTER REGION OF THE MAOA GENE WITH ANTISOCIAL PERSONALITY TRAITS IN ALCOHOLICS. Alcohol and Alcoholism. 38(1). 31–34. 48 indexed citations
18.
Koller, G., Ulrich W. Preuss, M. Bottlender, Kevin Wenzel, & Michael Soyka. (2002). Impulsivity and aggression as predictors of suicide attempts in alcoholics. European Archives of Psychiatry and Clinical Neuroscience. 252(4). 155–160. 112 indexed citations
19.
Preuss, Ulrich W., et al.. (2000). No association between suicidal behaviour and 5-HT2A-T102C polymorphism in alcohol dependants. European Neuropsychopharmacology. 10. 382–382. 1 indexed citations
20.
Thomm, Michael, Kathleen Sandman, Gerhard Frey, G. Koller, & Nicola Reeve. (1992). Transcription InVivoandInVitro oftheHistone-Encoding GenehmfBfromtheHyperthermophilic Archaeon Methanothermus fervidus. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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