G J Mulder
- Molecular Biology
- Oncology
- Pharmacology top 2%
- Pathology and Forensic Medicine top 10%
- Biochemistry top 5%
- Co-authors
- Emile de HeerMarieke KruideringJ. Fred NagelkerkeBob van de WaterK. Sandy PangEgbert ScholtensR. N. StillwellHenk Koster
- Topics
- Drug Transport and Resistance Mechanisms (9 papers)Pharmacogenetics and Drug Metabolism (8 papers)Eicosanoids and Hypertension Pharmacology (6 papers)
- Partner nations
- NetherlandsCanadaUnited States
In The Last Decade
G J Mulder
28 papers receiving 696 citations
Peers
Comparison fields: 5 of 85
- Molecular Biology 248
- Oncology 227
- Pharmacology 207
- Pathology and Forensic Medicine 202
- Biochemistry 92
Countries citing papers authored by G J Mulder
This map shows the geographic impact of G J Mulder's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by G J Mulder with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites G J Mulder more than expected).
Fields of papers citing papers by G J Mulder
This network shows the impact of papers produced by G J Mulder. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by G J Mulder. The network helps show where G J Mulder may publish in the future.
Co-authorship network of co-authors of G J Mulder
This figure shows the co-authorship network connecting the top 25 collaborators of G J Mulder. A scholar is included among the top collaborators of G J Mulder based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with G J Mulder. G J Mulder is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 289 | |
| 2 | 4 | |
| 3 | 8 | |
| 4 | Metabolic activation and DNA adduct detection of PhIP in dogs, rats, and humans in relation to urinary bladder and colon carcinogenesis. | 35 |
| 5 | 19 | |
| 6 | 15 | |
| 7 | 2 | |
| 8 | 5 | |
| 9 | 8 | |
| 10 | 6 | |
| 11 | 7 | |
| 12 | 16 | |
| 13 | 11 | |
| 14 | 18 | |
| 15 | 21 | |
| 16 | 21 | |
| 17 | 9 | |
| 18 | 58 | |
| 19 | 13 | |
| 20 | Formation of chemically reactive metabolites of phenacetin and acetaminophen. | 21 |
About G J Mulder
G J Mulder is a scholar working on Biochemistry, Pharmacology and Clinical Biochemistry, having authored 29 papers that have together received 723 indexed citations. Recurring topics across this work include Drug Transport and Resistance Mechanisms (9 papers), Pharmacogenetics and Drug Metabolism (8 papers) and Eicosanoids and Hypertension Pharmacology (6 papers). The work is most often cited by research in Pharmacology (207 citations), Biochemistry (92 citations) and Pathology and Forensic Medicine (202 citations). G J Mulder has collaborated with scholars based in Netherlands, Canada and United States. Frequent co-authors include Emile de Heer, Marieke Kruidering, J. Fred Nagelkerke, Bob van de Water, K. Sandy Pang, Egbert Scholtens, R. N. Stillwell, Henk Koster, J.M. te Koppele and J A Hinson. Their work appears in journals such as Neurology, Analytical Biochemistry and Journal of Pharmacology and Experimental Therapeutics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.