G. A. Williams

446 total citations
9 papers, 190 citations indexed

About

G. A. Williams is a scholar working on Molecular Biology, Ophthalmology and Pathology and Forensic Medicine. According to data from OpenAlex, G. A. Williams has authored 9 papers receiving a total of 190 indexed citations (citations by other indexed papers that have themselves been cited), including 4 papers in Molecular Biology, 3 papers in Ophthalmology and 2 papers in Pathology and Forensic Medicine. Recurrent topics in G. A. Williams's work include Retinal Development and Disorders (2 papers), Ophthalmology and Eye Disorders (2 papers) and Retinal Diseases and Treatments (2 papers). G. A. Williams is often cited by papers focused on Retinal Development and Disorders (2 papers), Ophthalmology and Eye Disorders (2 papers) and Retinal Diseases and Treatments (2 papers). G. A. Williams collaborates with scholars based in United Kingdom, India and Australia. G. A. Williams's co-authors include Chris F. Inglehearn, Martin McKibbin, Manir Ali, Paul D. Baxter, D. Timothy Bishop, Aine Rice, Robert A. Oliver, Parveen Sen, Jenny Morrison and Sarah Lewis and has published in prestigious journals such as Investigative Ophthalmology & Visual Science, British Journal of Ophthalmology and Journal of Medical Genetics.

In The Last Decade

G. A. Williams

8 papers receiving 189 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
G. A. Williams United Kingdom	6	120	82	70	38	17	9	190
Pavlína Skalická Czechia	10	93 0.8×	149 1.8×	92 1.3×	40 1.1×	8 0.5×	34	250
Natsue Omi Japan	8	187 1.6×	71 0.9×	143 2.0×	38 1.0×	47 2.8×	12	295
Josephine Duvall-Young United Kingdom	7	178 1.5×	71 0.9×	142 2.0×	32 0.8×	13 0.8×	9	309
Helen Booler United States	9	96 0.8×	61 0.7×	102 1.5×	21 0.6×	6 0.4×	27	200
Wael Abdrabou United States	7	223 1.9×	89 1.1×	105 1.5×	23 0.6×	21 1.2×	14	338
Wenjuan Zhuang China	10	146 1.2×	110 1.3×	78 1.1×	33 0.9×	18 1.1×	23	228
Yuichi Tokuda Japan	10	168 1.4×	147 1.8×	105 1.5×	22 0.6×	36 2.1×	23	353
Lessie McCain United States	9	159 1.3×	115 1.4×	49 0.7×	40 1.1×	53 3.1×	9	274
Lama AlAbdi Saudi Arabia	12	61 0.5×	45 0.5×	203 2.9×	64 1.7×	6 0.4×	26	318
Nongnart R. Chan Hong Kong	7	303 2.5×	277 3.4×	97 1.4×	13 0.3×	16 0.9×	9	375

Countries citing papers authored by G. A. Williams

Since Specialization

Citations

This map shows the geographic impact of G. A. Williams's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by G. A. Williams with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites G. A. Williams more than expected).

Fields of papers citing papers by G. A. Williams

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by G. A. Williams. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by G. A. Williams. The network helps show where G. A. Williams may publish in the future.

Co-authorship network of co-authors of G. A. Williams

This figure shows the co-authorship network connecting the top 25 collaborators of G. A. Williams. A scholar is included among the top collaborators of G. A. Williams based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with G. A. Williams. G. A. Williams is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

McKibbin, Martin, et al.. (2011). CFH,VEGFandHTRA1promoter genotype may influence the response to intravitreal ranibizumab therapy for neovascular age-related macular degeneration. British Journal of Ophthalmology. 96(2). 208–212. 90 indexed citations

Ali, Manir, Beatriz Buentello‐Volante, Martin McKibbin, et al.. (2010). Homozygous FOXE3 mutations cause non-syndromic, bilateral, total sclerocornea, aphakia, microphthalmia and optic disc coloboma.. PubMed. 16. 1162–8. 33 indexed citations

Anand, Seema, Eamonn Sheridan, Chris F. Inglehearn, et al.. (2009). MACULAR DYSTROPHY ASSOCIATED WITH THE ARG172TRP SUBSTITUTION IN PERIPHERIN/RDS. Retina. 29(5). 682–688. 4 indexed citations

Rice, Aine, Jérémie Nsengimana, Carmel Toomes, et al.. (2009). Replication of the Recessive STBMS1 Locus but with Dominant Inheritance. Investigative Ophthalmology & Visual Science. 50(7). 3210–3210. 20 indexed citations

Ramprasad, Vedam L., Nagasamy Soumittra, Derek J. Nancarrow, et al.. (2008). Identification of a novel splice-site mutation in the Lebercilin (LCA5) gene causing Leber congenital amaurosis.. PubMed. 14. 481–6. 18 indexed citations

Williams, G. A., Eamonn Sheridan, Carmel Toomes, et al.. (2005). Investigation of Candidate Loci for Familial Nonsyndromic Human Strabismus. Investigative Ophthalmology & Visual Science. 46(13). 3825–3825.

Pal, B., Moin Mohamed, T J Keen, et al.. (2004). A new phenotype of recessively inherited foveal hypoplasia and anterior segment dysgenesis maps to a locus on chromosome 16q23.2–24.2.. Journal of Medical Genetics. 41(10). 772–777. 4 indexed citations

Mansur, Adel, G. A. Williams, D. Timothy Bishop, et al.. (2000). Evidence for a role of HLA DRB1 alleles in the control of IgE levels, strengthened by interacting TCR A/D marker alleles. Clinical & Experimental Allergy. 30(10). 1371–1378. 16 indexed citations

Spooner, R. L., Robert A. Oliver, & G. A. Williams. (1977). Polymorphic variation in the amount of sialic acid attached to bovine transferrin. Animal Blood Groups and Biochemical Genetics. 8(1). 21–24. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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