Fumiya Obata

4.2k total citations
64 papers, 2.1k citations indexed

About

Fumiya Obata is a scholar working on Immunology, Molecular Biology and Neurology. According to data from OpenAlex, Fumiya Obata has authored 64 papers receiving a total of 2.1k indexed citations (citations by other indexed papers that have themselves been cited), including 28 papers in Immunology, 22 papers in Molecular Biology and 19 papers in Neurology. Recurrent topics in Fumiya Obata's work include Parkinson's Disease Mechanisms and Treatments (18 papers), T-cell and B-cell Immunology (16 papers) and Immune Cell Function and Interaction (12 papers). Fumiya Obata is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (18 papers), T-cell and B-cell Immunology (16 papers) and Immune Cell Function and Interaction (12 papers). Fumiya Obata collaborates with scholars based in Japan, United States and Canada. Fumiya Obata's co-authors include Kazuko Hasegawa, Manabu Funayama, Hisayuki Kowa, Shoji Tsuji, Etsuro Ohta, Masaaki Saito, Sanna M. Goyert, Enza Ferrero, Wolfgang J. Rettig and Aswani Yenamandra and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and The Journal of Immunology.

In The Last Decade

Fumiya Obata

64 papers receiving 2.1k citations

Peers

Fumiya Obata
Theodoros I. Roumeliotis United Kingdom
Anthony Slavin United States
Annick You-Ten Canada
Liping Qian China
K Motoyoshi Japan
Gayathri Perera United Kingdom
Bert van het Hof Netherlands
Alice Lau United States
Carmen Espejo Spain
Hiromitsu Kimura Japan
Theodoros I. Roumeliotis United Kingdom
Fumiya Obata
Citations per year, relative to Fumiya Obata Fumiya Obata (= 1×) peers Theodoros I. Roumeliotis

Countries citing papers authored by Fumiya Obata

Since Specialization
Citations

This map shows the geographic impact of Fumiya Obata's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fumiya Obata with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fumiya Obata more than expected).

Fields of papers citing papers by Fumiya Obata

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Fumiya Obata. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fumiya Obata. The network helps show where Fumiya Obata may publish in the future.

Co-authorship network of co-authors of Fumiya Obata

This figure shows the co-authorship network connecting the top 25 collaborators of Fumiya Obata. A scholar is included among the top collaborators of Fumiya Obata based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Fumiya Obata. Fumiya Obata is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kubo, Makoto, Takefumi Satoh, Ken‐ichi Tabata, et al.. (2015). Enhanced central memory cluster of differentiation 8+ and tumor antigen-specific T cells in prostate cancer patients receiving repeated in situ adenovirus-mediated suicide gene therapy. Molecular and Clinical Oncology. 3(3). 515–521. 9 indexed citations
2.
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Ohta, Etsuro, Fumitaka Kawakami, Makoto Kubo, & Fumiya Obata. (2012). Dominant-negative effects of LRRK2 heterodimers: A possible mechanism of neurodegeneration in Parkinson’s disease caused by LRRK2 I2020T mutation. Biochemical and Biophysical Research Communications. 430(2). 560–566. 12 indexed citations
4.
Maekawa, Tatsunori, Yui Sasaki, Takashi Miyajima, et al.. (2012). The I2020T Leucine-rich repeat kinase 2 transgenic mouse exhibits impaired locomotive ability accompanied by dopaminergic neuron abnormalities. Molecular Neurodegeneration. 7(1). 15–15. 37 indexed citations
5.
Ohta, Etsuro, Fumitaka Kawakami, Makoto Kubo, & Fumiya Obata. (2011). LRRK2 directly phosphorylates Akt1 as a possible physiological substrate: Impairment of the kinase activity by Parkinson's disease-associated mutations. FEBS Letters. 585(14). 2165–2170. 56 indexed citations
6.
Kubo, Makoto, Ryuichi Nagashima, Tatsunori Maekawa, et al.. (2010). LRRK2 is expressed in B-2 but not in B-1 B cells, and downregulated by cellular activation. Journal of Neuroimmunology. 229(1-2). 123–128. 31 indexed citations
7.
Maekawa, Tatsunori, et al.. (2010). Age-dependent and cell-population-restricted LRRK2 expression in normal mouse spleen. Biochemical and Biophysical Research Communications. 392(3). 431–435. 35 indexed citations
8.
Ohta, Etsuro, Fumitaka Kawakami, Tatsunori Maekawa, et al.. (2009). I2020T leucine-rich repeat kinase 2, the causative mutant molecule of familial Parkinson’s disease, has a higher intracellular degradation rate than the wild-type molecule. Biochemical and Biophysical Research Communications. 390(3). 710–715. 23 indexed citations
9.
Funayama, Manabu, Kazuko Hasegawa, Etsuro Ohta, et al.. (2005). An LRRK2 mutation as a cause for the parkinsonism in the original PARK8 family. Annals of Neurology. 57(6). 918–921. 204 indexed citations
10.
Obata, Fumiya, Kazunari Yoshida, M Ohkubo, et al.. (2004). Contribution of CD4+ and CD8+ T cells and interferon-gamma to the progress of chronic rejection of kidney allografts: the Th1 response mediates both acute and chronic rejection. Transplant Immunology. 14(1). 21–25. 37 indexed citations
11.
Funayama, Manabu, Kazuko Hasegawa, Hisayuki Kowa, et al.. (2002). A new locus for Parkinson's disease (PARK8) maps to chromosome 12p11.2–q13.1. Annals of Neurology. 51(3). 296–301. 481 indexed citations
12.
Hasegawa, Kazuko, et al.. (2001). Analysis of <i>α-synuclein, parkin, tau,</i> and <i>UCH-L1</i> in a Japanese Family with Autosomal Dominant Parkinsonism. European Neurology. 46(1). 20–24. 6 indexed citations
13.
Obata, Fumiya, et al.. (1998). T-CELL RECEPTOR VARIABLE GENE ANALYSIS OF RENAL ALLOGRAFT-INFILTRATING CELLS IN BIOPSY SPECIMENS USING A NONRADIOISOTOPIC MICROMETHOD1. Transplantation. 66(10). 1389–1392. 7 indexed citations
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Hiki, Yoshiyuki, et al.. (1991). Association of HLA-DQw4 with IgA Nephropathy in the Japanese Population. ˜The œNephron journals/Nephron journals. 58(1). 109–111. 11 indexed citations
17.
Obata, Fumiya, Akio Abe, Ichiro Ito, et al.. (1990). Sequence analysis and oligonucleotide genotyping of HLA-DR“JX6”, a DR“blank” haplotype found in the Japanese population. Human Immunology. 27(4). 269–284. 21 indexed citations
18.
Uchiyama, Takehiko, Ken’ichi Imanishi, Shinji Saito, et al.. (1989). Activation of human t cells by toxic shock syndrome toxin‐1: the toxin‐binding structures expressed on human lymphoid cells acting as accessory cells are hla class ii molecules. European Journal of Immunology. 19(10). 1803–1809. 40 indexed citations
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20.
Obata, Fumiya, Takeshi Endo, Genshu Tate, et al.. (1986). Partial N-terminal sequence analysis of DRw53 antigens: the beta-chains of DRw53 antigens are structurally distinct from beta-chains of DR and DQ antigens.. The Journal of Immunology. 136(6). 2187–2190. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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