Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies
if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the
same subfield and year (this is the minimum needed to enter the top 1%, not the average
within it), or reaches the top citation threshold in at least one of its specific research
topics.
The innate immune response to bacterial flagellin is mediated by Toll-like receptor 5
20012.8k citationsFumitaka Hayashi, Kelly D. Smith et al.Natureprofile →
Toll-like receptors stimulate human neutrophil function
2003726 citationsFumitaka Hayashi, Terry K. Means et al.Bloodprofile →
Human lupus autoantibody–DNA complexes activate DCs through cooperation of CD32 and TLR9
2005659 citationsTerry K. Means, Eicke Latz et al.Journal of Clinical Investigationprofile →
Toll-like receptor 5 recognizes a conserved site on flagellin required for protofilament formation and bacterial motility
2003642 citationsKelly D. Smith, Erica Andersen‐Nissen et al.Nature Immunologyprofile →
Leptospiral lipopolysaccharide activates cells through a TLR2-dependent mechanism
2001558 citationsCatherine Werts, Richard I. Tapping et al.Nature Immunologyprofile →
Peers — A (Enhanced Table)
Peers by citation overlap · career bar shows stage (early→late)
cites ·
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Countries citing papers authored by Fumitaka Hayashi
Since
Specialization
Citations
This map shows the geographic impact of Fumitaka Hayashi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fumitaka Hayashi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fumitaka Hayashi more than expected).
Fields of papers citing papers by Fumitaka Hayashi
This network shows the impact of papers produced by Fumitaka Hayashi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fumitaka Hayashi. The network helps show where Fumitaka Hayashi may publish in the future.
Co-authorship network of co-authors of Fumitaka Hayashi
This figure shows the co-authorship network connecting the top 25 collaborators of Fumitaka Hayashi.
A scholar is included among the top collaborators of Fumitaka Hayashi based on the total number of
citations received by their joint publications. Widths of edges
represent the number of papers authors have co-authored together.
Node borders
signify the number of papers an author published with Fumitaka Hayashi. Fumitaka Hayashi is excluded from
the visualization to improve readability, since they are connected to all nodes in the network.
Means, Terry K., Eicke Latz, Fumitaka Hayashi, et al.. (2005). Human lupus autoantibody–DNA complexes activate DCs through cooperation of CD32 and TLR9. Journal of Clinical Investigation. 115(2). 407–417.659 indexed citations breakdown →
3.
Smith, Kelly D., Erica Andersen‐Nissen, Fumitaka Hayashi, et al.. (2003). Toll-like receptor 5 recognizes a conserved site on flagellin required for protofilament formation and bacterial motility. Nature Immunology. 4(12). 1247–1253.642 indexed citations breakdown →
Hayashi, Fumitaka, Terry K. Means, & Andrew D. Luster. (2003). Toll-like receptors stimulate human neutrophil function. Blood. 102(7). 2660–2669.726 indexed citations breakdown →
Hayashi, Fumitaka, Kelly D. Smith, Adrian Ozinsky, et al.. (2001). The innate immune response to bacterial flagellin is mediated by Toll-like receptor 5. Nature. 410(6832). 1099–1103.2793 indexed citations breakdown →
8.
Werts, Catherine, Richard I. Tapping, John C. Mathison, et al.. (2001). Leptospiral lipopolysaccharide activates cells through a TLR2-dependent mechanism. Nature Immunology. 2(4). 346–352.558 indexed citations breakdown →
Clark, William R., Craig M. Walsh, Mehrdad Matloubian, Alison A. Glass, & Fumitaka Hayashi. (1996). Mechanisms of CTL-mediated cytoxicity. Seminars in Virology. 7(2). 113–116.1 indexed citations
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive
bibliographic database. While OpenAlex provides broad and valuable coverage of the global
research landscape, it—like all bibliographic datasets—has inherent limitations. These include
incomplete records, variations in author disambiguation, differences in journal indexing, and
delays in data updates. As a result, some metrics and network relationships displayed in
Rankless may not fully capture the entirety of a scholar's output or impact.