Friedrich Marks

9.8k total citations · 1 hit paper
222 papers, 8.1k citations indexed

About

Friedrich Marks is a scholar working on Molecular Biology, Pharmacology and Genetics. According to data from OpenAlex, Friedrich Marks has authored 222 papers receiving a total of 8.1k indexed citations (citations by other indexed papers that have themselves been cited), including 132 papers in Molecular Biology, 45 papers in Pharmacology and 40 papers in Genetics. Recurrent topics in Friedrich Marks's work include Protein Kinase Regulation and GTPase Signaling (40 papers), Inflammatory mediators and NSAID effects (39 papers) and Estrogen and related hormone effects (33 papers). Friedrich Marks is often cited by papers focused on Protein Kinase Regulation and GTPase Signaling (40 papers), Inflammatory mediators and NSAID effects (39 papers) and Estrogen and related hormone effects (33 papers). Friedrich Marks collaborates with scholars based in Germany, United States and France. Friedrich Marks's co-authors include Gerhard Fürstenberger, Michael Gschwendt, Walter Kittstein, Karin Müller‐Decker, Gabriele Rincke, Peter Krieg, E. Hecker, H.-J. Müller, Rukun Zang and Friedemann Horn and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

Friedrich Marks

218 papers receiving 7.7k citations

Hit Papers

Rottlerin, a Novel Protei... 1994 2026 2004 2015 1994 250 500 750

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author Last Decade Papers Cites
Friedrich Marks 4.7k 1.5k 1.2k 1.1k 1.1k 222 8.1k
Gerhard Fürstenberger 3.2k 0.7× 1.5k 1.0× 985 0.9× 1.1k 0.9× 786 0.7× 143 6.4k
R. K. Boutwell 4.9k 1.0× 503 0.3× 557 0.5× 1.4k 1.2× 593 0.6× 128 7.0k
Michael Karin 6.1k 1.3× 637 0.4× 1.1k 1.0× 2.5k 2.2× 1.0k 1.0× 35 9.5k
Hiroyasu Inoue 3.5k 0.7× 2.3k 1.5× 1.2k 1.0× 1.3k 1.2× 328 0.3× 178 8.5k
Dolores Pérez‐Sala 3.8k 0.8× 937 0.6× 294 0.3× 693 0.6× 870 0.8× 144 6.7k
Joël Raingeaud 7.9k 1.7× 542 0.4× 388 0.3× 1.7k 1.5× 1.3k 1.2× 23 11.0k
Wayne B. Anderson 6.6k 1.4× 375 0.3× 875 0.8× 449 0.4× 1.2k 1.1× 150 9.1k
Jesper Z. Haeggström 3.7k 0.8× 1.2k 0.8× 473 0.4× 612 0.5× 256 0.2× 192 9.2k
Yukihiro Akao 8.9k 1.9× 1.0k 0.7× 429 0.4× 5.1k 4.5× 764 0.7× 259 13.4k
Ichiro Kudo 6.9k 1.5× 4.2k 2.9× 2.6k 2.2× 1.5k 1.3× 1.3k 1.2× 206 13.2k

Countries citing papers authored by Friedrich Marks

Since Specialization
Citations

This map shows the geographic impact of Friedrich Marks's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Friedrich Marks with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Friedrich Marks more than expected).

Fields of papers citing papers by Friedrich Marks

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Friedrich Marks. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Friedrich Marks. The network helps show where Friedrich Marks may publish in the future.

Co-authorship network of co-authors of Friedrich Marks

This figure shows the co-authorship network connecting the top 25 collaborators of Friedrich Marks. A scholar is included among the top collaborators of Friedrich Marks based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Friedrich Marks. Friedrich Marks is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Marks, Friedrich, Ursula Klingmüller, & Karin Müller‐Decker. (2017). Cellular Signal Processing. 29 indexed citations
2.
Marks, Friedrich, Gerhard Fürstenberger, & Karin Müller‐Decker. (2007). Tumor Promotion as a Target of Cancer Prevention. PubMed. 174. 37–47. 29 indexed citations
3.
Müller‐Decker, Karin, et al.. (2005). Inhibition of Cell Proliferation by Bacterial Lipopolysaccharides in TLR4-Positive Epithelial Cells: Independence of Nitric Oxide and Cytokine Release. Journal of Investigative Dermatology. 124(3). 553–561. 16 indexed citations
4.
Marks, Friedrich, et al.. (2003). Cyclooxygenase-2 and Skin Carcinogenesis. PubMed. 37. 72–89. 9 indexed citations
5.
Müller‐Decker, Karin, et al.. (2002). The Effects of Cyclooxygenase Isozyme Inhibition onIncisional Wound Healing in Mouse Skin. Journal of Investigative Dermatology. 119(5). 1189–1195. 61 indexed citations
6.
Krieg, Peter, et al.. (2002). Epidermis-Type Lipoxygenases. Advances in experimental medicine and biology. 507. 165–170. 15 indexed citations
7.
Marks, Friedrich. (2001). Krebs- und Alzheimer-Prävention mit nicht-steroidalen Entzündungshemmern. DMW - Deutsche Medizinische Wochenschrift. 126(11). 308–313. 2 indexed citations
8.
Marks, Friedrich. (2000). Der Stoffwechsel der Arachidonsäure. Biologie in unserer Zeit. 30(6). 342–353.
9.
Richter, Karl Hartmut, et al.. (2000). Evidence against a role of general protein kinase C downregulation in skin tumor promotion. International Journal of Cancer. 85(4). 503–507. 13 indexed citations
10.
Marks, Friedrich & Gerhard Fürstenberger. (1999). Prostaglandins, leukotrienes, and other eicosanoids : from biogenesis to clinical applications. Wiley-VCH eBooks. 24 indexed citations
12.
Fürstenberger, Gerhard, et al.. (1996). Relationship between TGFα-induced DNA synthesis and prostaglandin synthesis in human HaCaT keratinocytes. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1310(1). 137–144. 31 indexed citations
13.
Gschwendt, Michael, Gerhard Fürstenberger, Walter Kittstein, et al.. (1995). Lack of an effect of novel inhibitors with high specificity for protein kinase C on the action of the phorbol ester 12-O-tetradecanoylphorbol-13-acetate on mouse skin in vivo. Carcinogenesis. 16(1). 107–111. 23 indexed citations
14.
Marks, Friedrich, et al.. (1995). Loss of Protein Kinase Cδ from Human HaCaT Keratinocytes upon Ras Transfection Is Mediated by TGFα. Experimental Cell Research. 219(1). 299–303. 35 indexed citations
15.
Müller‐Decker, Karin, G. Fürstenberger, & Friedrich Marks. (1994). Keratinocyte-Derived Proinflammatory Key Mediators and Cell Viability as in Vitro Parameters of Irritancy: A Possible Alternative to the Draize Skin Irritation Test. Toxicology and Applied Pharmacology. 127(1). 99–108. 57 indexed citations
16.
Marks, Friedrich & G. Fürstenberger. (1993). Proliferative responses of the skin to external stimuli.. Environmental Health Perspectives. 101(suppl 5). 95–101. 36 indexed citations
17.
Gschwendt, Michael, Walter Kittstein, & Friedrich Marks. (1991). Protein kinase C activation by phorbol esters: do cysteine-rich regions and pseudosubstrate motifs play a role?. Trends in Biochemical Sciences. 16(5). 167–169. 107 indexed citations
18.
Rose‐John, Stefan, Alexander Dietrich, & Friedrich Marks. (1988). Molecular cloning of mouse protein kinase C (PKC) cDNA from Swiss 3T3 fibroblasts. Gene. 74(2). 465–471. 113 indexed citations
19.
Fürstenberger, Gerhard, et al.. (1986). Isolation, characterization and in vitro cultivation of subfractions of neonatal mouse keratinocytes: effects of phorbol esters. Carcinogenesis. 7(10). 1745–1753. 51 indexed citations
20.
Marks, Friedrich. (1976). Epidermal growth control mechanisms, hyperplasia, and tumor promotion in the skin.. Munich Personal RePEc Archive (Ludwig Maximilian University of Munich). 36(7 PT 2). 2636–43. 40 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026