Friedrich Grummt

2.8k total citations
76 papers, 2.4k citations indexed

About

Friedrich Grummt is a scholar working on Molecular Biology, Genetics and Oncology. According to data from OpenAlex, Friedrich Grummt has authored 76 papers receiving a total of 2.4k indexed citations (citations by other indexed papers that have themselves been cited), including 61 papers in Molecular Biology, 13 papers in Genetics and 12 papers in Oncology. Recurrent topics in Friedrich Grummt's work include DNA Repair Mechanisms (24 papers), RNA modifications and cancer (15 papers) and RNA and protein synthesis mechanisms (14 papers). Friedrich Grummt is often cited by papers focused on DNA Repair Mechanisms (24 papers), RNA modifications and cancer (15 papers) and RNA and protein synthesis mechanisms (14 papers). Friedrich Grummt collaborates with scholars based in Germany, United States and Switzerland. Friedrich Grummt's co-authors include Ingrid Grummt, Michael Wegner, Valerie Smith, Elisabeth Sock, Vera Pütter, Josef‐Karl Gerber, Caroline Staib, Raymond Reeves, Sergei Chuvpilo and Hans‐Michael Jantzen and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

Friedrich Grummt

76 papers receiving 2.2k citations

Peers

Friedrich Grummt

GENET

ONCOL

PHYSI

IMMUN

Ian D. Hiles United Kingdom

Earl F. Baril United States

Monica Hirsch‐Kauffmann Austria

Erhard Wintersberger Austria

H. Green United States

J.E. Dixon United States

Detlef Doenecke Germany

Helen R. Flynn United Kingdom

Heinrich Gausepohlꝉ Germany

Joanne E. Hesse United States

Ian D. Hiles United Kingdom

Friedrich Grummt

2.0k ×1.1

571 ×1.3

GENET

459 ×1.2

ONCOL

176 ×0.8

PHYSI

178 ×0.9

IMMUN

Citations per year, relative to Friedrich Grummt Friedrich Grummt (= 1×) peers Ian D. Hiles

Countries citing papers authored by Friedrich Grummt

Since Specialization

Citations

This map shows the geographic impact of Friedrich Grummt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Friedrich Grummt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Friedrich Grummt more than expected).

Fields of papers citing papers by Friedrich Grummt

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Friedrich Grummt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Friedrich Grummt. The network helps show where Friedrich Grummt may publish in the future.

Co-authorship network of co-authors of Friedrich Grummt

This figure shows the co-authorship network connecting the top 25 collaborators of Friedrich Grummt. A scholar is included among the top collaborators of Friedrich Grummt based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Friedrich Grummt. Friedrich Grummt is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Gentschev, Ivaylo, Stephanie Weibel, Friedrich Grummt, et al.. (2011). Efficient Colonization and Therapy of Human Hepatocellular Carcinoma (HCC) Using the Oncolytic Vaccinia Virus Strain GLV-1h68. PLoS ONE. 6(7). e22069–e22069. 35 indexed citations

Herrmann, Thomas, et al.. (2007). Site-specific interaction of the murine pre-replicative complex with origin DNA: assembly and disassembly during cell cycle transit and differentiation. Nucleic Acids Research. 35(20). 6701–6713. 11 indexed citations

Grummt, Friedrich, et al.. (2007). Interactions and subcellular distribution of DNA replication initiation proteins in eukaryotic cells. Molecular Genetics and Genomics. 278(6). 623–632. 11 indexed citations

Pütter, Vera, et al.. (2006). Mouse pre-replicative complex proteins colocalise and interact with the centrosome. European Journal of Cell Biology. 86(1). 37–50. 45 indexed citations

Wallisch, Michael, et al.. (2002). Ku Antigen Supports Termination of Mammalian rDNA Replication by Transcription Termination Factor TTF-I. Biological Chemistry. 383(5). 765–71. 7 indexed citations

Springer, Jan, et al.. (1999). Identification and characterization of MmORC4 and MmORC5 , two subunits of the mouse origin of replication recognition complex. Chromosoma. 108(4). 243–249. 11 indexed citations

Pütter, Vera, et al.. (1999). Identification, characterization and chromosomal localization of the cognate human and murine DBF4 genes. Molecular and General Genetics MGG. 262(2). 220–229. 17 indexed citations

Gerber, Josef‐Karl, et al.. (1997). Termination of Mammalian rDNA Replication: Polar Arrest of Replication Fork Movement by Transcription Termination Factor TTF-I. Cell. 90(3). 559–567. 112 indexed citations

Budde, Allen D., et al.. (1997). P53 Activates Fanconi Anemia Group C Gene Expression. Human Molecular Genetics. 6(2). 277–283. 21 indexed citations

10.

Rottmann, O., et al.. (1996). Liposome‐mediated gene transfer via sperm cells. High transfer efficiency and persistence of transgenes by use of liposomes and sperm cells and a murine amplification element. Journal of Animal Breeding and Genetics. 113(1-6). 401–411. 13 indexed citations

11.

Staib, Caroline, et al.. (1996). p53 Inhibits JC Virus DNA Replicationin Vivoand Interacts with JC Virus Large T-Antigen. Virology. 219(1). 237–246. 61 indexed citations

12.

Sock, Elisabeth, et al.. (1995). Functional Interaction between the POU Domain Protein Tst-1/Oct-6 and the High-Mobility-Group Protein HMG-I/Y. Molecular and Cellular Biology. 15(7). 3738–3747. 88 indexed citations

13.

Grummt, Friedrich, et al.. (1994). Structural homologies and functional similarities between mammalian origins of replication and amplification promoting sequences. Chromosoma. 103(3). 209–214. 11 indexed citations

14.

Nick, Sigrid, et al.. (1993). Inhibition of HIV-1 replication by a high-copy-number vector expressing antisense RNA for reverse transcriptase. Gene. 129(2). 263–268. 16 indexed citations

15.

Sock, Elisabeth, Michael Wegner, Elizabeth A. Fortunato, & Friedrich Grummt. (1993). Large T-Antigen and Sequences within the Regulatory Region of JC Virus Both Contribute to the Features of JC Virus DNA Replication. Virology. 197(2). 537–548. 25 indexed citations

16.

Grummt, Friedrich, et al.. (1991). Mapping Eukaryotic Replication Origins In Vivo by Size Analysis of Purified Nascent DNA Strands. DNA and Cell Biology. 10(2). 149–157. 3 indexed citations

17.

Wegner, Michael, et al.. (1989). Identification of an amplification promoting DNA sequence from the hypotrichous cilateStylonychia lemnae. Nucleic Acids Research. 17(21). 8783–8802. 19 indexed citations

18.

Weidle, Ulrich H., Peter Buckel, & Friedrich Grummt. (1988). A new expression system for mammalian cells based on putative replicator sequences of the mouse and a truncated thymidine kinase gene. Gene. 73(2). 427–437. 6 indexed citations

19.

Grummt, Friedrich, et al.. (1988). Transposed human immunoglobulin Vκ gene regions carry clusters of conserved sequence elements. Gene. 69(2). 225–236. 8 indexed citations

20.

Grummt, Friedrich. (1983). Diadenosine tetraphosphate (Ap4A): a putative chemical messenger of cell proliferation control and inducer of DNA replication. Plant Molecular Biology. 2(1). 41–4. 14 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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