Frida E. Kleiman

1.9k total citations
29 papers, 1.4k citations indexed

About

Frida E. Kleiman is a scholar working on Molecular Biology, Oncology and Physiology. According to data from OpenAlex, Frida E. Kleiman has authored 29 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Molecular Biology, 3 papers in Oncology and 3 papers in Physiology. Recurrent topics in Frida E. Kleiman's work include RNA Research and Splicing (20 papers), RNA modifications and cancer (14 papers) and DNA Repair Mechanisms (6 papers). Frida E. Kleiman is often cited by papers focused on RNA Research and Splicing (20 papers), RNA modifications and cancer (14 papers) and DNA Repair Mechanisms (6 papers). Frida E. Kleiman collaborates with scholars based in United States, Sweden and Argentina. Frida E. Kleiman's co-authors include James L. Manley, Xiaokan Zhang, Dixie J. Goss, Zhen‐Qiang Pan, Toru Ouchi, Angus Chen, Anders Virtanen, Murat Cevher, Alejandra del C. Alonso and Leah Cohen and has published in prestigious journals such as Science, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

Frida E. Kleiman

29 papers receiving 1.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Frida E. Kleiman United States 19 1.2k 238 176 160 111 29 1.4k
Ko Sato Japan 17 1.1k 0.9× 182 0.8× 195 1.1× 299 1.9× 175 1.6× 25 1.5k
Jukka Kallijärvi Finland 19 753 0.6× 126 0.5× 84 0.5× 99 0.6× 134 1.2× 41 1.1k
Isabelle Pirson Belgium 25 1.1k 0.8× 239 1.0× 193 1.1× 115 0.7× 92 0.8× 57 1.6k
Benoı̂t Pierrat Switzerland 15 1.0k 0.8× 271 1.1× 234 1.3× 115 0.7× 132 1.2× 15 1.3k
Hriday K. Das United States 18 676 0.5× 147 0.6× 92 0.5× 207 1.3× 104 0.9× 46 1.2k
X-J Yang China 10 1.1k 0.9× 158 0.7× 199 1.1× 48 0.3× 143 1.3× 19 1.3k
Jennifer M. Harrell United States 12 1.0k 0.8× 122 0.5× 181 1.0× 65 0.4× 79 0.7× 14 1.3k
Jack‐Christophe Cossec France 14 699 0.6× 251 1.1× 113 0.6× 336 2.1× 44 0.4× 19 1.1k
Dennis Hellgren Sweden 18 633 0.5× 92 0.4× 202 1.1× 142 0.9× 154 1.4× 39 1.0k

Countries citing papers authored by Frida E. Kleiman

Since Specialization
Citations

This map shows the geographic impact of Frida E. Kleiman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Frida E. Kleiman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Frida E. Kleiman more than expected).

Fields of papers citing papers by Frida E. Kleiman

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Frida E. Kleiman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Frida E. Kleiman. The network helps show where Frida E. Kleiman may publish in the future.

Co-authorship network of co-authors of Frida E. Kleiman

This figure shows the co-authorship network connecting the top 25 collaborators of Frida E. Kleiman. A scholar is included among the top collaborators of Frida E. Kleiman based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Frida E. Kleiman. Frida E. Kleiman is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Yu, Amy, et al.. (2024). Estrogen receptor alpha (ERα) regulates PARN-mediated nuclear deadenylation and gene expression in breast cancer cells. RNA Biology. 21(1). 1090–1099. 1 indexed citations
2.
Ghosh, Jayashri, Claudia Wultsch, Imad Shureiqi, et al.. (2022). Epigenome-Wide Study Identifies Epigenetic Outliers in Normal Mucosa of Patients with Colorectal Cancer. Cancer Prevention Research. 15(11). 755–766. 5 indexed citations
3.
Murphy, Michael R., et al.. (2021). Poly(A) tail dynamics: Measuring polyadenylation, deadenylation and poly(A) tail length. Methods in enzymology on CD-ROM/Methods in enzymology. 655. 265–290. 3 indexed citations
4.
Ordóñez, Martha, et al.. (2019). Nuclear Tau, p53 and Pin1 Regulate PARN-Mediated Deadenylation and Gene Expression. Frontiers in Molecular Neuroscience. 12. 242–242. 18 indexed citations
5.
Alonso, Alejandra del C., et al.. (2018). Hyperphosphorylation of Tau Associates With Changes in Its Function Beyond Microtubule Stability. Frontiers in Cellular Neuroscience. 12. 338–338. 185 indexed citations
6.
Zhang, Xiaokan, et al.. (2017). Nucleolin phosphorylation regulates PARN deadenylase activity during cellular stress response. RNA Biology. 15(2). 251–260. 24 indexed citations
7.
Park, Ji Yeon, Michael R. Murphy, Xiaokan Zhang, et al.. (2016). Intronic cleavage and polyadenylation regulates gene expression during DNA damage response through U1 snRNA. Cell Discovery. 2(1). 16013–16013. 34 indexed citations
8.
Zhang, Xiaokan, et al.. (2015). PARN deadenylase is involved in miRNA-dependent degradation of TP53 mRNA in mammalian cells. Nucleic Acids Research. 43(22). 10925–10938. 42 indexed citations
9.
Goss, Dixie J. & Frida E. Kleiman. (2012). Poly(A) binding proteins: are they all created equal?. Wiley Interdisciplinary Reviews - RNA. 4(2). 167–179. 92 indexed citations
10.
Mohammed, S A, et al.. (2011). p53 inhibits mRNA 3′ processing through its interaction with the CstF/BARD1 complex. Oncogene. 30(27). 3073–3083. 33 indexed citations
11.
Cevher, Murat, Xiaokan Zhang, Sully Fernandez, et al.. (2010). Nuclear deadenylation/polyadenylation factors regulate 3′ processing in response to DNA damage. The EMBO Journal. 29(10). 1674–1687. 65 indexed citations
12.
Zhang, Xiaokan, Anders Virtanen, & Frida E. Kleiman. (2010). To polyadenylate or to deadenylate. Cell Cycle. 9(22). 4437–4449. 72 indexed citations
13.
Cevher, Murat & Frida E. Kleiman. (2010). Connections between 3′‐end processing and DNA damage response. Wiley Interdisciplinary Reviews - RNA. 1(1). 193–199. 24 indexed citations
14.
Fonseca, Dina M., et al.. (2008). The 3' processing factor CstF functions in the DNA repair response. Nucleic Acids Research. 36(6). 1792–1804. 40 indexed citations
15.
Li, Kai, et al.. (2006). DNA Damage–Induced BARD1 Phosphorylation Is Critical for the Inhibition of Messenger RNA Processing by BRCA1/BARD1 Complex. Cancer Research. 66(9). 4561–4565. 31 indexed citations
16.
Kleiman, Frida E., et al.. (2005). BRCA1/BARD1 inhibition of mRNA 3′ processing involves targeted degradation of RNA polymerase II. Genes & Development. 19(10). 1227–1237. 117 indexed citations
17.
Chen, Angus, Frida E. Kleiman, James L. Manley, Toru Ouchi, & Zhen‐Qiang Pan. (2002). Autoubiquitination of the BRCA1·BARD1 RING Ubiquitin Ligase. Journal of Biological Chemistry. 277(24). 22085–22092. 170 indexed citations
18.
Kleiman, Frida E. & James L. Manley. (2001). The BARD1-CstF-50 Interaction Links mRNA 3′ End Formation to DNA Damage and Tumor Suppression. Cell. 104(5). 743–753. 172 indexed citations
19.
Kleiman, Frida E. & James L. Manley. (1999). Functional Interaction of BRCA1-Associated BARD1 with Polyadenylation Factor CstF-50. Science. 285(5433). 1576–1579. 167 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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