Frauke Forstreuter

615 total citations
7 papers, 511 citations indexed

About

Frauke Forstreuter is a scholar working on Molecular Biology, Cancer Research and Genetics. According to data from OpenAlex, Frauke Forstreuter has authored 7 papers receiving a total of 511 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 4 papers in Cancer Research and 2 papers in Genetics. Recurrent topics in Frauke Forstreuter's work include Cancer, Hypoxia, and Metabolism (4 papers), Angiogenesis and VEGF in Cancer (3 papers) and Metabolism, Diabetes, and Cancer (2 papers). Frauke Forstreuter is often cited by papers focused on Cancer, Hypoxia, and Metabolism (4 papers), Angiogenesis and VEGF in Cancer (3 papers) and Metabolism, Diabetes, and Cancer (2 papers). Frauke Forstreuter collaborates with scholars based in Germany, United Kingdom and Australia. Frauke Forstreuter's co-authors include Rolf Mentlein, Ralph Lucius, Janka Held‐Feindt, Olaf Eichler, Patrick H. Maxwell, Deepa Shukla, Thomas Pufe, H. Maximilian Mehdorn, Kai‐Uwe Eckardt and Seymour Rosen and has published in prestigious journals such as Journal of Clinical Investigation, Journal of the American Society of Nephrology and International Journal of Cancer.

In The Last Decade

Frauke Forstreuter

7 papers receiving 505 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Frauke Forstreuter Germany	7	241	131	88	84	79	7	511
Martin P. Powers United States	11	349 1.4×	137 1.0×	91 1.0×	38 0.5×	44 0.6×	18	805
Sepi Mahooti United States	12	258 1.1×	127 1.0×	118 1.3×	66 0.8×	29 0.4×	19	602
M. Mirakhur United Kingdom	11	175 0.7×	83 0.6×	69 0.8×	42 0.5×	44 0.6×	17	524
Zhe Huang Japan	12	332 1.4×	157 1.2×	44 0.5×	62 0.7×	72 0.9×	16	597
Vincenza Caolo Netherlands	15	312 1.3×	69 0.5×	60 0.7×	65 0.8×	46 0.6×	17	553
Lisa Honold Germany	9	251 1.0×	76 0.6×	77 0.9×	71 0.8×	74 0.9×	11	630
Hisham Alkhalidi Saudi Arabia	11	366 1.5×	71 0.5×	88 1.0×	72 0.9×	75 0.9×	50	650
Ali Jalali United States	12	302 1.3×	117 0.9×	57 0.6×	50 0.6×	94 1.2×	40	602
J Asai Japan	10	189 0.8×	39 0.3×	45 0.5×	97 1.2×	62 0.8×	16	456
Yolanda Piña United States	13	255 1.1×	134 1.0×	127 1.4×	33 0.4×	36 0.5×	62	629

Countries citing papers authored by Frauke Forstreuter

Since Specialization

Citations

This map shows the geographic impact of Frauke Forstreuter's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Frauke Forstreuter with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Frauke Forstreuter more than expected).

Fields of papers citing papers by Frauke Forstreuter

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Frauke Forstreuter. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Frauke Forstreuter. The network helps show where Frauke Forstreuter may publish in the future.

Co-authorship network of co-authors of Frauke Forstreuter

This figure shows the co-authorship network connecting the top 25 collaborators of Frauke Forstreuter. A scholar is included among the top collaborators of Frauke Forstreuter based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Frauke Forstreuter. Frauke Forstreuter is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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7 of 7 papers shown

1.

Schley, Gunnar, Bernd Klanke, Johannes Schödel, et al.. (2011). Hypoxia-Inducible Transcription Factors Stabilization in the Thick Ascending Limb Protects against Ischemic Acute Kidney Injury. Journal of the American Society of Nephrology. 22(11). 2004–2015. 83 indexed citations

2.

Cantley, James, Colin Selman, Deepa Shukla, et al.. (2008). Deletion of the von Hippel–Lindau gene in pancreatic β cells impairs glucose homeostasis in mice. Journal of Clinical Investigation. 119(1). 125–35. 102 indexed citations

3.

Mentlein, Rolf, Frauke Forstreuter, H. Maximilian Mehdorn, & Janka Held‐Feindt. (2004). Functional Significance of Vascular Endothelial Growth Factor Receptor Expression on Human Glioma Cells. Journal of Neuro-Oncology. 67(1-2). 9–18. 40 indexed citations

4.

Forstreuter, Frauke, Ralph Lucius, & Rolf Mentlein. (2002). Vascular endothelial growth factor induces chemotaxis and proliferation of microglial cells. Journal of Neuroimmunology. 132(1-2). 93–98. 141 indexed citations

5.

Mentlein, Rolf, Olaf Eichler, Frauke Forstreuter, & Janka Held‐Feindt. (2001). Somatostatin inhibits the production of vascular endothelial growth factor in human glioma cells. International Journal of Cancer. 92(4). 545–550. 90 indexed citations

6.

Held‐Feindt, Janka, Frauke Forstreuter, Thomas Pufe, & Rolf Mentlein. (2001). Influence of the somatostatin receptor sst2 on growth factor signal cascades in human glioma cells. Molecular Brain Research. 87(1). 12–21. 42 indexed citations

7.

Held‐Feindt, Janka, Brigitte Krisch, Frauke Forstreuter, & Rolf Mentlein. (2000). Somatostatin receptors in gliomas. Journal of Physiology-Paris. 94(3-4). 251–258. 13 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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