Frank W. King

1.5k total citations
13 papers, 1.2k citations indexed

About

Frank W. King is a scholar working on Molecular Biology, Surgery and Oncology. According to data from OpenAlex, Frank W. King has authored 13 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 2 papers in Surgery and 2 papers in Oncology. Recurrent topics in Frank W. King's work include Pluripotent Stem Cells Research (3 papers), DNA Repair Mechanisms (3 papers) and Tissue Engineering and Regenerative Medicine (2 papers). Frank W. King is often cited by papers focused on Pluripotent Stem Cells Research (3 papers), DNA Repair Mechanisms (3 papers) and Tissue Engineering and Regenerative Medicine (2 papers). Frank W. King collaborates with scholars based in United States, Poland and Germany. Frank W. King's co-authors include Harold S. Bernstein, Emma Shtivelman, Robert J. Schwartz, Edward C. Hsiao, Kathryn N. Ivey, Deepak Srivastava, Jason E. Fish, Ru-Fang Yeh, Bruce R. Conklin and Martín Steffen and has published in prestigious journals such as Cell, Journal of Biological Chemistry and The EMBO Journal.

In The Last Decade

Frank W. King

13 papers receiving 1.1k citations

Peers

Frank W. King
Young Ae Joe South Korea
Frank W. King
Citations per year, relative to Frank W. King Frank W. King (= 1×) peers Young Ae Joe

Countries citing papers authored by Frank W. King

Since Specialization
Citations

This map shows the geographic impact of Frank W. King's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Frank W. King with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Frank W. King more than expected).

Fields of papers citing papers by Frank W. King

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Frank W. King. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Frank W. King. The network helps show where Frank W. King may publish in the future.

Co-authorship network of co-authors of Frank W. King

This figure shows the co-authorship network connecting the top 25 collaborators of Frank W. King. A scholar is included among the top collaborators of Frank W. King based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Frank W. King. Frank W. King is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
Ritner, Carissa, Frank W. King, Shirley Mihardja, et al.. (2011). An Engineered Cardiac Reporter Cell Line Identifies Human Embryonic Stem Cell-Derived Myocardial Precursors. PLoS ONE. 6(1). e16004–e16004. 34 indexed citations
2.
King, Frank W., Walter Liszewski, Carissa Ritner, & Harold S. Bernstein. (2010). High-Throughput Tracking of Pluripotent Human Embryonic Stem Cells with Dual Fluorescence Resonance Energy Transfer Molecular Beacons. Stem Cells and Development. 20(3). 475–484. 27 indexed citations
3.
Fong, Sylvia, Frank W. King, & Emma Shtivelman. (2010). CC3/TIP30 affects DNA damage repair. BMC Cell Biology. 11(1). 23–23. 8 indexed citations
4.
King, Frank W., Carissa Ritner, Walter Liszewski, et al.. (2009). Subpopulations of Human Embryonic Stem Cells With Distinct Tissue-Specific Fates Can Be Selected From Pluripotent Cultures. Stem Cells and Development. 18(10). 1441–1450. 34 indexed citations
5.
King, Frank W., Sylvia Fong, Chandi Griffin, et al.. (2009). Timosaponin AIII Is Preferentially Cytotoxic to Tumor Cells through Inhibition of mTOR and Induction of ER Stress. PLoS ONE. 4(9). e7283–e7283. 74 indexed citations
6.
Ivey, Kathryn N., Frank W. King, Ru-Fang Yeh, et al.. (2008). MicroRNA Regulation of Cell Lineages in Mouse and Human Embryonic Stem Cells. Cell stem cell. 2(3). 219–229. 478 indexed citations
7.
Epting, Conrad L., et al.. (2008). Stem cell antigen‐1 localizes to lipid microdomains and associates with insulin degrading enzyme in skeletal myoblasts. Journal of Cellular Physiology. 217(1). 250–260. 25 indexed citations
8.
King, Frank W. & Emma Shtivelman. (2004). Inhibition of Nuclear Import by the Proapoptotic Protein CC3. Molecular and Cellular Biology. 24(16). 7091–7101. 45 indexed citations
9.
Walerych, Dawid, Grzegorz Kudla, Małgorzata Gutkowska, et al.. (2004). Hsp90 Chaperones Wild-type p53 Tumor Suppressor Protein. Journal of Biological Chemistry. 279(47). 48836–48845. 130 indexed citations
10.
King, Frank W., et al.. (2004). Inhibition of Chk1 by Activated PKB/Akt. Cell Cycle. 3(5). 632–635. 65 indexed citations
11.
King, Frank W.. (2001). Co-chaperones Bag-1, Hop and Hsp40 regulate Hsc70 and Hsp90 interactions with wild-type or mutant p53. The EMBO Journal. 20(22). 6297–6305. 142 indexed citations
12.
King, Frank W.. (1991). It's How You Play the Game: The Inside Story of the Calgary Olympics. Medical Entomology and Zoology. 16 indexed citations
13.
Liu, Chau‐Ching, Martín Steffen, Frank W. King, & John Ding‐E Young. (1987). Identification, isolation, and characterization of a novel cytotoxin in murine cytolytic lymphocytes. Cell. 51(3). 393–403. 94 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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