Francisco Blanco‐Vaca

7.5k total citations · 1 hit paper
178 papers, 5.5k citations indexed

About

Francisco Blanco‐Vaca is a scholar working on Surgery, Molecular Biology and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, Francisco Blanco‐Vaca has authored 178 papers receiving a total of 5.5k indexed citations (citations by other indexed papers that have themselves been cited), including 109 papers in Surgery, 52 papers in Molecular Biology and 49 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in Francisco Blanco‐Vaca's work include Cholesterol and Lipid Metabolism (59 papers), Lipoproteins and Cardiovascular Health (55 papers) and Diabetes, Cardiovascular Risks, and Lipoproteins (39 papers). Francisco Blanco‐Vaca is often cited by papers focused on Cholesterol and Lipid Metabolism (59 papers), Lipoproteins and Cardiovascular Health (55 papers) and Diabetes, Cardiovascular Risks, and Lipoproteins (39 papers). Francisco Blanco‐Vaca collaborates with scholars based in Spain, United States and Finland. Francisco Blanco‐Vaca's co-authors include Joan Carles Escolà‐Gil, Josep Julve, Laura Calpe‐Berdiel, Vicent Ribas, Jordi Ordóñez‐Llanos, Xavier Palomer, Francesc González‐Sastre, Dı́dac Mauricio, Jesús M. Martín‐Campos and Lídia Cedó and has published in prestigious journals such as Journal of Biological Chemistry, Circulation and Blood.

In The Last Decade

Francisco Blanco‐Vaca

172 papers receiving 5.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Gut Microbiota-Derived TMAO: A Causal Factor Promoting Atherosclerotic Cardiovascular Disease?

2023 88 citations Marina Canyelles, Noemí Rotllán et al. International Journal of Molecular Sciences profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Francisco Blanco‐Vaca Spain	41	2.3k	1.5k	1.3k	730	689	178	5.5k
Joan Carles Escolà‐Gil Spain	35	1.7k 0.8×	1.5k 1.0×	906 0.7×	810 1.1×	485 0.7×	151	4.1k
Jerzy–Roch Nofer Germany	36	2.0k 0.9×	2.0k 1.3×	1.3k 1.0×	628 0.9×	494 0.7×	104	5.0k
John R. Burnett Australia	36	2.5k 1.1×	1.3k 0.9×	1.3k 1.0×	694 1.0×	421 0.6×	164	4.6k
Laurent Lagrost France	47	2.9k 1.3×	2.5k 1.7×	2.2k 1.7×	894 1.2×	694 1.0×	186	6.9k
Tamio Teramoto Japan	38	2.6k 1.1×	878 0.6×	1.7k 1.3×	671 0.9×	566 0.8×	212	5.6k
Carlo Sabbà Italy	46	1.7k 0.7×	1.5k 1.0×	693 0.5×	490 0.7×	846 1.2×	211	6.7k
Hubert Scharnagl Austria	49	2.6k 1.2×	1.4k 0.9×	1.8k 1.3×	794 1.1×	1.1k 1.6×	265	7.4k
Rajasekhar Ramakrishnan United States	48	2.0k 0.9×	1.6k 1.1×	1.5k 1.1×	620 0.8×	889 1.3×	118	6.4k
Angela Pirillo Italy	36	2.2k 1.0×	1.4k 1.0×	1.4k 1.0×	767 1.1×	444 0.6×	116	6.0k
Heinrich Wieland Germany	37	1.9k 0.9×	1.0k 0.7×	1.6k 1.2×	675 0.9×	461 0.7×	144	5.1k

Countries citing papers authored by Francisco Blanco‐Vaca

Since Specialization

Citations

This map shows the geographic impact of Francisco Blanco‐Vaca's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Francisco Blanco‐Vaca with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Francisco Blanco‐Vaca more than expected).

Fields of papers citing papers by Francisco Blanco‐Vaca

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Francisco Blanco‐Vaca. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Francisco Blanco‐Vaca. The network helps show where Francisco Blanco‐Vaca may publish in the future.

Co-authorship network of co-authors of Francisco Blanco‐Vaca

This figure shows the co-authorship network connecting the top 25 collaborators of Francisco Blanco‐Vaca. A scholar is included among the top collaborators of Francisco Blanco‐Vaca based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Francisco Blanco‐Vaca. Francisco Blanco‐Vaca is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Canyelles, Marina, David Santos, Noemí Rotllán, et al.. (2025). Cerebrospinal fluid lipoprotein-mediated cholesterol delivery to neurons is impaired in Alzheimer's disease and involves APOE4. Journal of Lipid Research. 66(8). 100865–100865.

Delgado‐Lista, Javier, José María Mostaza, Francisco Blanco‐Vaca, et al.. (2024). Consensus on lipoprotein(a) of the Spanish Society of Arteriosclerosis. Literature review and recommendations for clinical practice. Clínica e Investigación en Arteriosclerosis (English Edition). 36(4). 243–266.

Canyelles, Marina, Josefa Girona, Daiana Ibarretxe, et al.. (2024). PCSK9 Antibodies Treatment Specifically Enhances the Macrophage-specific Reverse Cholesterol Transport Pathway in Heterozygous Familial Hypercholesterolemia. JACC Basic to Translational Science. 9(10). 1195–1210. 1 indexed citations

Delgado‐Lista, Javier, José María Mostaza, Francisco Blanco‐Vaca, et al.. (2024). WITHDRAWN: Consensus on lipoprotein(a) of the Spanish Society of Arteriosclerosis. Literature review and recommendations for clinical practice. Clínica e Investigación en Arteriosclerosis (English Edition).

Muñoz, Juan Pablo, et al.. (2023). FTY720-P, a Biased S1PR Ligand, Increases Mitochondrial Function through STAT3 Activation in Cardiac Cells. International Journal of Molecular Sciences. 24(8). 7374–7374. 2 indexed citations

Canyelles, Marina, et al.. (2023). Gut Microbiota-Derived TMAO: A Causal Factor Promoting Atherosclerotic Cardiovascular Disease?. International Journal of Molecular Sciences. 24(3). 1940–1940. 88 indexed citations breakdown →

Blanco‐Vaca, Francisco, Noemí Rotllán, Marina Canyelles, et al.. (2022). NAD+-Increasing Strategies to Improve Cardiometabolic Health?. Frontiers in Endocrinology. 12. 815565–815565. 7 indexed citations

Canyelles, Marina, Antonio Pérez, Alexandra Junza, et al.. (2022). Divergent Effects of Glycemic Control and Bariatric Surgery on Circulating Concentrations of TMAO in Newly Diagnosed T2D Patients and Morbidly Obese. Diagnostics. 12(11). 2783–2783. 3 indexed citations

Canyelles, Marina, Álvaro García‐Osuna, Alexandra Junza, et al.. (2021). The Capacity of APOB-Depleted Plasma in Inducing ATP-Binding Cassette A1/G1-Mediated Macrophage Cholesterol Efflux—But Not Gut Microbial-Derived Metabolites—Is Independently Associated with Mortality in Patients with ST-Segment Elevation Myocardial Infarction. Biomedicines. 9(10). 1336–1336. 4 indexed citations

10.

Climent, Elisenda, Ana M. Bea, David Benaiges, et al.. (2021). LDL Cholesterol Reduction Variability with Different Types and Doses of Statins in Monotherapy or Combined with Ezetimibe. Results from the Spanish Arteriosclerosis Society Dyslipidaemia Registry. Cardiovascular Drugs and Therapy. 36(2). 301–308. 11 indexed citations

11.

Martín‐Campos, Jesús M., Daiana Ibarretxe, Alberto Zamora, et al.. (2020). Polygenic Markers in Patients Diagnosed of Autosomal Dominant Hypercholesterolemia in Catalonia: Distribution of Weighted LDL-c-Raising SNP Scores and Refinement of Variant Selection. Biomedicines. 8(9). 353–353. 7 indexed citations

12.

Canyelles, Marina, Mireia Tondo, Jes S. Lindholt, et al.. (2020). Macrophage Cholesterol Efflux Downregulation Is Not Associated with Abdominal Aortic Aneurysm (AAA) Progression. Biomolecules. 10(4). 662–662. 2 indexed citations

13.

Blanco‐Vaca, Francisco, Jesús M. Martín‐Campos, Marina Canyelles, et al.. (2019). Molecular analysis of APOB, SAR1B, ANGPTL3, and MTTP in patients with primary hypocholesterolemia in a clinical laboratory setting: Evidence supporting polygenicity in mutation-negative patients. Atherosclerosis. 283. 52–60. 13 indexed citations

14.

Farré, Núria, David Santos, Jari Metso, et al.. (2019). Human ApoA-I Overexpression Enhances Macrophage-Specific Reverse Cholesterol Transport but Fails to Prevent Inherited Diabesity in Mice. International Journal of Molecular Sciences. 20(3). 655–655. 8 indexed citations

15.

Martínez-López, Diego, Emilio Camafeita, Lídia Cedó, et al.. (2019). APOA1 oxidation is associated to dysfunctional high-density lipoproteins in human abdominal aortic aneurysm. EBioMedicine. 43. 43–53. 33 indexed citations

16.

Santos, David, Núria Farré, Sergi Leánez, et al.. (2018). Administration of CORM-2 inhibits diabetic neuropathy but does not reduce dyslipidemia in diabetic mice. PLoS ONE. 13(10). e0204841–e0204841. 8 indexed citations

17.

Sánchez-Hernández, Rosa M., Fernando Civeira, Marianne Stef, et al.. (2016). Homozygous Familial Hypercholesterolemia in Spain. Circulation Cardiovascular Genetics. 9(6). 504–510. 43 indexed citations

18.

Sánchez-Quesada, José Luís, et al.. (2013). Impact of the LDL subfraction phenotype on Lp-PLA2 distribution, LDL modification and HDL composition in type 2 diabetes. Cardiovascular Diabetology. 12(1). 112–112. 45 indexed citations

19.

Escolà‐Gil, Joan Carles, Noemí Rotllán, Josep Julve, & Francisco Blanco‐Vaca. (2009). In vivo macrophage-specific RCT and antioxidant and antiinflammatory HDL activity measurements: New tools for predicting HDL atheroprotection. Atherosclerosis. 206(2). 321–327. 53 indexed citations

20.

Souto, Juan Carlos, Francisco Blanco‐Vaca, José Manuel Soria, et al.. (2005). A Genomewide Exploration Suggests a New Candidate Gene at Chromosome 11q23 as the Major Determinant of Plasma Homocysteine Levels: Results from the GAIT Project. The American Journal of Human Genetics. 76(6). 925–933. 89 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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