F.M. Cornelis

820 total citations
23 papers, 380 citations indexed

About

F.M. Cornelis is a scholar working on Rheumatology, Molecular Biology and Cancer Research. According to data from OpenAlex, F.M. Cornelis has authored 23 papers receiving a total of 380 indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Rheumatology, 13 papers in Molecular Biology and 5 papers in Cancer Research. Recurrent topics in F.M. Cornelis's work include Osteoarthritis Treatment and Mechanisms (18 papers), Knee injuries and reconstruction techniques (4 papers) and Inflammatory mediators and NSAID effects (4 papers). F.M. Cornelis is often cited by papers focused on Osteoarthritis Treatment and Mechanisms (18 papers), Knee injuries and reconstruction techniques (4 papers) and Inflammatory mediators and NSAID effects (4 papers). F.M. Cornelis collaborates with scholars based in Belgium, Netherlands and France. F.M. Cornelis's co-authors include Rik Lories, Silvia Monteagudo, F. Cailotto, Peter Carmeliet, Ingrid Meulenbelt, Wouter den Hollander, Frank P. Luyten, An Sermon, C. Cherifi and Nico Lakenberg and has published in prestigious journals such as Nature Communications, PLoS ONE and Development.

In The Last Decade

F.M. Cornelis

23 papers receiving 378 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
F.M. Cornelis Belgium	12	214	210	78	53	49	23	380
Heba Ismail United Kingdom	12	212 1.0×	261 1.2×	83 1.1×	99 1.9×	51 1.0×	24	519
Chin-Jung Hsu Taiwan	10	99 0.5×	186 0.9×	105 1.3×	36 0.7×	28 0.6×	11	407
Xiaoyu Nie United States	8	182 0.9×	148 0.7×	99 1.3×	55 1.0×	41 0.8×	19	320
M.H. van den Bosch Netherlands	9	240 1.1×	212 1.0×	39 0.5×	103 1.9×	44 0.9×	16	383
Kei Imagawa United Kingdom	6	291 1.4×	198 0.9×	176 2.3×	110 2.1×	23 0.5×	7	435
E. Houtman Netherlands	8	212 1.0×	124 0.6×	120 1.5×	43 0.8×	41 0.8×	16	293
Patricia Fleuranceau-Morel United States	8	230 1.1×	127 0.6×	24 0.3×	74 1.4×	89 1.8×	10	394
Angel Soto‐Hermida Spain	13	292 1.4×	302 1.4×	120 1.5×	76 1.4×	35 0.7×	17	539
Wanqing Xie China	11	85 0.4×	145 0.7×	43 0.6×	33 0.6×	21 0.4×	19	304
S.E. Usmani Canada	8	237 1.1×	114 0.5×	40 0.5×	118 2.2×	56 1.1×	9	352

Countries citing papers authored by F.M. Cornelis

Since Specialization

Citations

This map shows the geographic impact of F.M. Cornelis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by F.M. Cornelis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites F.M. Cornelis more than expected).

Fields of papers citing papers by F.M. Cornelis

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by F.M. Cornelis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by F.M. Cornelis. The network helps show where F.M. Cornelis may publish in the future.

Co-authorship network of co-authors of F.M. Cornelis

This figure shows the co-authorship network connecting the top 25 collaborators of F.M. Cornelis. A scholar is included among the top collaborators of F.M. Cornelis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with F.M. Cornelis. F.M. Cornelis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Cornelis, F.M., et al.. (2025). Lipidomics unravels lipid changes in osteoarthritis articular cartilage. Annals of the Rheumatic Diseases. 84(7). 1264–1276. 3 indexed citations

Cornelis, F.M., et al.. (2024). IGF1 drives Wnt-induced joint damage and is a potential therapeutic target for osteoarthritis. Nature Communications. 15(1). 9170–9170. 9 indexed citations

Cherifi, C., F.M. Cornelis, Sofia Pazmiño, et al.. (2023). Inhibition of KDM7A/B histone demethylases restores H3K79 methylation and protects against osteoarthritis. Annals of the Rheumatic Diseases. 82(7). 963–973. 28 indexed citations

Cornelis, F.M., Cécile Lambert, Érika Kague, et al.. (2023). Osteomodulin downregulation is associated with osteoarthritis development. Bone Research. 11(1). 49–49. 11 indexed citations

Monteagudo, Silvia, F.M. Cornelis, An Sermon, et al.. (2022). ANP32A represses Wnt signaling across tissues thereby protecting against osteoarthritis and heart disease. Osteoarthritis and Cartilage. 30(5). 724–734. 11 indexed citations

Cornelis, F.M., et al.. (2022). Hypoxia and Wnt signaling inversely regulate expression of chondroprotective molecule ANP32A in articular cartilage. Osteoarthritis and Cartilage. 31(4). 507–518. 6 indexed citations

Houtman, E., Margo Tuerlings, H. Eka D. Suchiman, et al.. (2022). Inhibiting thyroid activation in aged human explants prevents mechanical induced detrimental signalling by mitigating metabolic processes. Lara D. Veeken. 62(1). 457–466. 4 indexed citations

Zhou, Qi, F.M. Cornelis, Silvia Monteagudo, & Rik Lories. (2021). Dynamics of inflammation and resolution in the collagenase-induced mouse model of osteoarthritis. Osteoarthritis and Cartilage. 29. S361–S361. 1 indexed citations

Cornelis, F.M., C. Cherifi, An Sermon, et al.. (2021). Hypoxia induces DOT1L in articular cartilage to protect against osteoarthritis. JCI Insight. 6(24). 19 indexed citations

10.

Cornelis, F.M., Domiziana Costamagna, Robin Duelen, et al.. (2020). Frizzled related protein deficiency impairs muscle strength, gait and calpain 3 levels. Orphanet Journal of Rare Diseases. 15(1). 119–119. 7 indexed citations

11.

Cornelis, F.M., et al.. (2019). Exostosin-1 enhances canonical Wnt signaling activity during chondrogenic differentiation. Osteoarthritis and Cartilage. 27(11). 1702–1710. 24 indexed citations

12.

Clockaerts, S., et al.. (2018). The anterior tibiotalar fat pad as a source of pain and inflammation in osteoarthritis of the ankle: anatomy, histology and imaging. Osteoarthritis and Cartilage. 26. S120–S120. 1 indexed citations

13.

Cornelis, F.M., et al.. (2018). Increased susceptibility to develop spontaneous and post-traumatic osteoarthritis in Dot1l-deficient mice. Osteoarthritis and Cartilage. 27(3). 513–525. 33 indexed citations

14.

Cornelis, F.M., et al.. (2018). ANP32A regulates ATM expression and prevents oxidative stress in cartilage, brain and bone. Osteoarthritis and Cartilage. 26. S21–S21. 2 indexed citations

15.

Monteagudo, Silvia, et al.. (2017). DOT1L safeguards cartilage homeostasis and protects against osteoarthritis. Nature Communications. 8(1). 108 indexed citations

16.

Bömer, Nils, F.M. Cornelis, Y.F. Ramos, et al.. (2016). Aberrant Calreticulin Expression in Articular Cartilage of Dio2 Deficient Mice. PLoS ONE. 11(5). e0154999–e0154999. 2 indexed citations

17.

Bömer, Nils, F.M. Cornelis, Y.F. Ramos, et al.. (2014). The effect of forced exercise on knee joints in Dio2−/− mice: type II iodothyronine deiodinase-deficient mice are less prone to develop OA-like cartilage damage upon excessive mechanical stress. Annals of the Rheumatic Diseases. 75(3). 571–577. 32 indexed citations

18.

Bömer, Nils, F.M. Cornelis, Y.F. Ramos, et al.. (2014). The effect of severe exercise on knee-joints: identifying pathways involved in cartilage degradation processes following mechanical stress. Osteoarthritis and Cartilage. 22. S311–S312. 1 indexed citations

19.

Cornelis, F.M., Frank P. Luyten, & Rik Lories. (2011). Functional effects of susceptibility genes in osteoarthritis.. PubMed. 12(63). 129–39. 16 indexed citations

20.

Cox, Luk, Lieve Umans, F.M. Cornelis, Danny Huylebroeck, & An Zwijsen. (2008). A broken heart: A stretch too far. International Journal of Cardiology. 131(1). 33–44. 11 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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