Firoz Kabir

426 total citations
23 papers, 258 citations indexed

About

Firoz Kabir is a scholar working on Molecular Biology, Ophthalmology and Genetics. According to data from OpenAlex, Firoz Kabir has authored 23 papers receiving a total of 258 indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Molecular Biology, 9 papers in Ophthalmology and 7 papers in Genetics. Recurrent topics in Firoz Kabir's work include Connexins and lens biology (10 papers), Retinal Development and Disorders (5 papers) and Retinal Diseases and Treatments (4 papers). Firoz Kabir is often cited by papers focused on Connexins and lens biology (10 papers), Retinal Development and Disorders (5 papers) and Retinal Diseases and Treatments (4 papers). Firoz Kabir collaborates with scholars based in United States, Pakistan and Saudi Arabia. Firoz Kabir's co-authors include Shahid Y. Khan, Sheikh Riazuddin, Javed Akram, Muhammad Ali, J. Fielding Hejtmancik, Muhammad Asif Naeem, Shaheen N. Khan, Sheikh Riazuddin, Tayyab Husnaın and Arif O. Khan and has published in prestigious journals such as Nature Communications, PLoS ONE and Scientific Reports.

In The Last Decade

Firoz Kabir

22 papers receiving 255 citations

Peers

Firoz Kabir

OPHTH

GENET

RNMI

Muhammad Asif Naeem Pakistan

Quincy Prescott United Kingdom

Risa Sato Japan

Leo Sheck New Zealand

Aaron Black United States

Katarzyna Janik-Papis Poland

Rosario Fernández‐Godino United States

Saoud Tahsin Swafiri Spain

Mohammed E. El‐Asrag United Kingdom

Adriana Buskin United Kingdom

Muhammad Asif Naeem Pakistan

Firoz Kabir

271 ×1.4

152 ×1.6

OPHTH

76 ×1.3

GENET

35 ×0.7

RNMI

26 ×1.1

Citations per year, relative to Firoz Kabir Firoz Kabir (= 1×) peers Muhammad Asif Naeem

Countries citing papers authored by Firoz Kabir

Since Specialization

Citations

This map shows the geographic impact of Firoz Kabir's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Firoz Kabir with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Firoz Kabir more than expected).

Fields of papers citing papers by Firoz Kabir

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Firoz Kabir. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Firoz Kabir. The network helps show where Firoz Kabir may publish in the future.

Co-authorship network of co-authors of Firoz Kabir

This figure shows the co-authorship network connecting the top 25 collaborators of Firoz Kabir. A scholar is included among the top collaborators of Firoz Kabir based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Firoz Kabir. Firoz Kabir is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Masry, Ahmed, Firoz Kabir, Md Tahmid Rahman Laskar, et al.. (2025). ChartQAPro: A More Diverse and Challenging Benchmark for Chart Question Answering. 19123–19151. 1 indexed citations

Kabir, Firoz, et al.. (2022). A genomic deletion encompassing CRYBB2-CRYBB2P1 is responsible for autosomal recessive congenital cataracts. Human Genome Variation. 9(1). 31–31.

Kabir, Firoz, Jiali Li, Libe Gradstein, et al.. (2020). Mutations in CERKL and RP1 cause retinitis pigmentosa in Pakistani families. Human Genome Variation. 7(1). 14–14. 3 indexed citations

Ali, Muhammad, Firoz Kabir, Santosh Renuse, et al.. (2020). Generation and proteome profiling of PBMC-originated, iPSC-derived lentoid bodies. Stem Cell Research. 46. 101813–101813. 6 indexed citations

Khan, Shahid Y., et al.. (2020). Novel mutations in LTBP2 identified in familial cases of primary congenital glaucoma.. PubMed. 26. 14–25. 11 indexed citations

Ali, Muhammad, Firoz Kabir, Yinghong Ma, et al.. (2019). Comparative transcriptome analysis of hESC- and iPSC-derived lentoid bodies. Scientific Reports. 9(1). 18552–18552. 15 indexed citations

Khan, Shahid Y., Muhammad Ali, Firoz Kabir, et al.. (2018). Identification of novel transcripts and peptides in developing murine lens. Scientific Reports. 8(1). 11162–11162. 4 indexed citations

Khan, Shahid Y., Firoz Kabir, Xiaodong Jiao, et al.. (2018). Whole genome sequencing data for two individuals of Pakistani descent. Scientific Data. 5(1). 180174–180174. 1 indexed citations

Ali, Muhammad, Shahid Y. Khan, Firoz Kabir, John D. Gottsch, & Sheikh Riazuddin. (2018). Comparative transcriptome analysis of hESC- and iPSC-derived corneal endothelial cells. Experimental Eye Research. 176. 252–257. 10 indexed citations

10.

Khan, Shahid Y., Muhammad Ali, Firoz Kabir, et al.. (2018). Proteome Profiling of Developing Murine Lens Through Mass Spectrometry. Investigative Ophthalmology & Visual Science. 59(1). 100–100. 21 indexed citations

11.

Kabir, Firoz, Sabika Firasat, Muhammad Asif Naeem, et al.. (2016). A spectrum of CYP1B1 mutations associated with primary congenital glaucoma in families of Pakistani descent. Human Genome Variation. 3(1). 16021–16021. 18 indexed citations

12.

Khan, Shahid Y., Muhammad Ali, Haiba Kaul, et al.. (2016). Mutation in LIM2 Is Responsible for Autosomal Recessive Congenital Cataracts. PLoS ONE. 11(11). e0162620–e0162620. 20 indexed citations

13.

Khan, Shahid Y., Shivakumar Vasanth, Firoz Kabir, et al.. (2016). FOXE3 contributes to Peters anomaly through transcriptional regulation of an autophagy-associated protein termed DNAJB1. Nature Communications. 7(1). 10953–10953. 36 indexed citations

14.

Khan, Shahid Y., Muhammad Ali, Muhammad Daud, et al.. (2016). Deletion at the GCNT2 Locus Causes Autosomal Recessive Congenital Cataracts. PLoS ONE. 11(12). e0167562–e0167562. 8 indexed citations

15.

Ullah, Inayat, Firoz Kabir, Muhammad Asif Naeem, et al.. (2016). Mutations in phosphodiesterase 6 identified in familial cases of retinitis pigmentosa. Human Genome Variation. 3(1). 16036–16036. 6 indexed citations

16.

Jiao, Xiaodong, Firoz Kabir, Arif O. Khan, et al.. (2016). A Common Ancestral Mutation in CRYBB3 Identified in Multiple Consanguineous Families with Congenital Cataracts. PLoS ONE. 11(6). e0157005–e0157005. 10 indexed citations

17.

Kabir, Firoz, Inayat Ullah, Shahbaz Ali, et al.. (2016). Loss of function mutations in RP1 are responsible for retinitis pigmentosa in consanguineous familial cases.. PubMed. 22. 610–25. 11 indexed citations

18.

Khan, Shahid Y., Arif O. Khan, Qiwei Wang, et al.. (2015). Missense Mutations in CRYAB Are Liable for Recessive Congenital Cataracts. PLoS ONE. 10(9). e0137973–e0137973. 31 indexed citations

19.

Kabir, Firoz, Shagufta Naz, Sheikh Riazuddin, et al.. (2013). Novel mutations in RPE65 identified in consanguineous Pakistani families with retinal dystrophy.. PubMed. 19. 1554–64. 3 indexed citations

20.

Araman, Philip A., et al.. (2003). Unsound defect volume in hardwood pallet cants.. Forest Products Journal. 53(2). 45–49. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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