Fernanda Wisnieski
- Cancer Research top 10%
- Cancer-related molecular mechanisms research 6
- MicroRNA in disease regulation 5
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- RNA modifications and cancer 6
- Epigenetics and DNA Methylation 6
- Cancer-related gene regulation 5
- Histone Deacetylase Inhibitors Research 2
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- Peptidase Inhibition and Analysis 2
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- Helicobacter pylori-related gastroenterology studies 3
Fernanda Wisnieski
19 papers receiving 420 citations
Peers
Comparison fields: 5 of 59
- Cancer Research 164
- Molecular Biology 324
- Pathology and Forensic Medicine 42
- Oncology 61
- Surgery 75
Countries citing papers authored by Fernanda Wisnieski
This map shows the geographic impact of Fernanda Wisnieski's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fernanda Wisnieski with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fernanda Wisnieski more than expected).
Fields of papers citing papers by Fernanda Wisnieski
This network shows the impact of papers produced by Fernanda Wisnieski. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fernanda Wisnieski. The network helps show where Fernanda Wisnieski may publish in the future.
Co-authorship network
The 21 scholars most cited alongside Fernanda Wisnieski, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2022 | 8 | |
| 2 | 2021 | 3 | |
| 3 | 2020 | 15 | |
| 4 | 2020 | 18 | |
| 5 | 2019 | 14 | |
| 6 | 2019 | 27 | |
| 7 | 2017 | 14 | |
| 8 | 2017 | 27 | |
| 9 | 2016 | 13 | |
| 10 | 2016 | 12 | |
| 11 | 2016 | 36 | |
| 12 | 2016 | 14 | |
| 13 | 2015 | 7 | |
| 14 | 2015 | 8 | |
| 15 | 2014 | 34 | |
| 16 | 2014 | 14 | |
| 17 | 2013 | 24 | |
| 18 | 2013 | 40 | |
| 19 | 2012 | 92 |
About Fernanda Wisnieski
Fernanda Wisnieski is a scholar working on Cancer Research, Business and International Management, Geriatrics and Gerontology, Molecular Biology and Pathology and Forensic Medicine, having authored 19 papers that have together received 420 indexed citations. Recurring topics across this work include RNA modifications and cancer (6 papers), Epigenetics and DNA Methylation (6 papers), Cancer-related molecular mechanisms research (6 papers), Cancer-related gene regulation (5 papers), MicroRNA in disease regulation (5 papers), Helicobacter pylori-related gastroenterology studies (3 papers), Peptidase Inhibition and Analysis (2 papers) and Histone Deacetylase Inhibitors Research (2 papers). The work is most often cited by research in Cancer Research (164 citations), Molecular Biology (324 citations), Pathology and Forensic Medicine (42 citations), Oncology (61 citations) and Surgery (75 citations). Fernanda Wisnieski has collaborated with scholars based in Brazil, Austria and United States. Frequent co-authors include Marı́lia de Arruda Cardoso Smith, Rommel Mário Rodríguez Burbano, Danielle Queiroz Calcagno, Carolina Oliveira Gigek, Elizabeth Chen, Mariana Ferreira Leal, Paulo Pimentel de Assumpção, Sâmia Demachki, Lucas Trevizani Rasmussen and Laércio Gomes Lourenço. Their work appears in journals such as Tumor Biology, World Journal of Gastroenterology, Epigenomics, Future Medicinal Chemistry and Journal of Cellular Biochemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.