Feray Köçkar
- Molecular Biology
- Organic Chemistry top 10%
- Pharmacology top 10%
- Clinical Biochemistry top 5%
- Cancer Research
- Co-authors
- Selma SinanOktay ArslanClaudiu T. SupuranMeltem AlperSemra IşıkÖzen Özensoy GülerAlessio InnocentiNahit Gençer
- Topics
- Enzyme function and inhibition (23 papers)Synthesis and Catalytic Reactions (9 papers)Cancer, Hypoxia, and Metabolism (9 papers)
- Journals
- Nucleic Acids ResearchSHILAP Revista de lepidopterologíaGene
- Partner nations
- TürkiyeItalyUnited Kingdom
In The Last Decade
Feray Köçkar
68 papers receiving 800 citations
Peers
Comparison fields: 5 of 90
- Molecular Biology 502
- Organic Chemistry 204
- Pharmacology 132
- Clinical Biochemistry 118
- Cancer Research 99
Countries citing papers authored by Feray Köçkar
This map shows the geographic impact of Feray Köçkar's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Feray Köçkar with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Feray Köçkar more than expected).
Fields of papers citing papers by Feray Köçkar
This network shows the impact of papers produced by Feray Köçkar. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Feray Köçkar. The network helps show where Feray Köçkar may publish in the future.
Co-authorship network of co-authors of Feray Köçkar
This figure shows the co-authorship network connecting the top 25 collaborators of Feray Köçkar. A scholar is included among the top collaborators of Feray Köçkar based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Feray Köçkar. Feray Köçkar is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 0 | |
| 3 | 0 | |
| 4 | 2 | |
| 5 | 3 | |
| 6 | 1 | |
| 7 | 8 | |
| 8 | 0 | |
| 9 | 12 | |
| 10 | 11 | |
| 11 | 15 | |
| 12 | 10 | |
| 13 | 3 | |
| 14 | 29 | |
| 15 | Oleuropein’in Prostat (PC-3), Meme (MCF-7) ve Hepatoma (HEP3B) Kanser Hücrelerinde Anti-tümör Etkisinin Belirlenmesi | 2 |
| 16 | 7 | |
| 17 | 5 | |
| 18 | Farklı genomik dna izolasyon yöntemlerinin satureja (labiatae) türlerinde uygulanması | 1 |
| 19 | 74 | |
| 20 | 18 |
About Feray Köçkar
Feray Köçkar is a scholar working on Clinical Biochemistry, Cancer Research and Biochemistry, having authored 77 papers that have together received 814 indexed citations. Recurring topics across this work include Enzyme function and inhibition (23 papers), Synthesis and Catalytic Reactions (9 papers) and Cancer, Hypoxia, and Metabolism (9 papers). The work is most often cited by research in Clinical Biochemistry (118 citations), Biochemistry (69 citations) and Pharmacology (132 citations). Feray Köçkar has collaborated with scholars based in Türkiye, Italy and United Kingdom. Frequent co-authors include Selma Sinan, Oktay Arslan, Claudiu T. Supuran, Meltem Alper, Oktay Arslan, Semra Işık, Özen Özensoy Güler, Alessio Innocenti, Nahit Gençer and Mehmet Ay. Their work appears in journals such as Nucleic Acids Research, SHILAP Revista de lepidopterología and Gene.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.