Felix Rückert
- Oncology top 2%
- Surgery top 5%
- Molecular Biology
- Pulmonary and Respiratory Medicine top 10%
- Cancer Research top 5%
- Co-authors
- Robert GrützmannChristian PilarskyHans‐Detlev SaegerMarius DistlerStephan KerstingTorsten J. WilhelmEleftherios P. DiamandisEmrullah Birgin
- Topics
- Pancreatic and Hepatic Oncology Research (50 papers)Pancreatitis Pathology and Treatment (22 papers)Neuroendocrine Tumor Research Advances (11 papers)
- Cited by
- OncologyCancer ResearchSurgery
- Partner nations
- GermanyUnited StatesCanada
In The Last Decade
Felix Rückert
79 papers receiving 1.8k citations
Peers
Comparison fields: 5 of 101
- Oncology 1.1k
- Surgery 721
- Molecular Biology 572
- Pulmonary and Respiratory Medicine 357
- Cancer Research 321
Countries citing papers authored by Felix Rückert
This map shows the geographic impact of Felix Rückert's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Felix Rückert with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Felix Rückert more than expected).
Fields of papers citing papers by Felix Rückert
This network shows the impact of papers produced by Felix Rückert. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Felix Rückert. The network helps show where Felix Rückert may publish in the future.
Co-authorship network of co-authors of Felix Rückert
This figure shows the co-authorship network connecting the top 25 collaborators of Felix Rückert. A scholar is included among the top collaborators of Felix Rückert based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Felix Rückert. Felix Rückert is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 27 | |
| 2 | 7 | |
| 3 | 1 | |
| 4 | 20 | |
| 5 | 27 | |
| 6 | 1 | |
| 7 | 13 | |
| 8 | 39 | |
| 9 | 25 | |
| 10 | Hepatocyte Growth Factor Signaling Pathway as a Potential Target in Ductal Adenocarcinoma of the Pancreas | 1 |
| 11 | 1 | |
| 12 | 2 | |
| 13 | 4 | |
| 14 | 25 | |
| 15 | 3 | |
| 16 | 30 | |
| 17 | 42 | |
| 18 | 48 | |
| 19 | 76 | |
| 20 | 32 |
About Felix Rückert
Felix Rückert is a scholar working on Oncology, Surgery and Pulmonary and Respiratory Medicine, having authored 80 papers that have together received 1.8k indexed citations. Recurring topics across this work include Pancreatic and Hepatic Oncology Research (50 papers), Pancreatitis Pathology and Treatment (22 papers) and Neuroendocrine Tumor Research Advances (11 papers). The work is most often cited by research in Oncology (1.1k citations), Cancer Research (321 citations) and Surgery (721 citations). Felix Rückert has collaborated with scholars based in Germany, United States and Canada. Frequent co-authors include Robert Grützmann, Christian Pilarsky, Hans‐Detlev Saeger, Marius Distler, Stephan Kersting, Torsten J. Wilhelm, Eleftherios P. Diamandis, Emrullah Birgin, Marco Niedergethmann and Stefan Post. Their work appears in journals such as Gastroenterology, PLoS ONE and Advanced Functional Materials.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.