Federico Probst

460 total citations
8 papers, 249 citations indexed

About

Federico Probst is a scholar working on Physiology, Neurology and Molecular Biology. According to data from OpenAlex, Federico Probst has authored 8 papers receiving a total of 249 indexed citations (citations by other indexed papers that have themselves been cited), including 4 papers in Physiology, 3 papers in Neurology and 2 papers in Molecular Biology. Recurrent topics in Federico Probst's work include Alzheimer's disease research and treatments (4 papers), Neuroinflammation and Neurodegeneration Mechanisms (3 papers) and Neuroscience and Neuropharmacology Research (2 papers). Federico Probst is often cited by papers focused on Alzheimer's disease research and treatments (4 papers), Neuroinflammation and Neurodegeneration Mechanisms (3 papers) and Neuroscience and Neuropharmacology Research (2 papers). Federico Probst collaborates with scholars based in Germany, Switzerland and Poland. Federico Probst's co-authors include Christian Haass, Axel Rominger, Jochen Herms, Felix Overhoff, Anna Jaworska, Matthias Brendel, Peter Bartenstein, Simon Lindner, Karlheinz Baumann and Gernot Kleinberger and has published in prestigious journals such as Theranostics, Journal of Nuclear Medicine and Frontiers in Neuroscience.

In The Last Decade

Federico Probst

8 papers receiving 247 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Federico Probst Germany 6 140 107 53 53 45 8 249
Felix Overhoff Germany 6 140 1.0× 107 1.0× 53 1.0× 53 1.0× 45 1.0× 8 249
Barbara von Ungern‐Sternberg Germany 9 104 0.7× 73 0.7× 60 1.1× 54 1.0× 18 0.4× 18 217
Carola Focke Germany 7 113 0.8× 188 1.8× 62 1.2× 34 0.6× 105 2.3× 9 306
Tanja Blume Germany 12 267 1.9× 181 1.7× 89 1.7× 76 1.4× 63 1.4× 19 427
Ludovic Perrot Switzerland 6 109 0.8× 97 0.9× 77 1.5× 35 0.7× 60 1.3× 6 266
Unai Perpiñá Spain 6 119 0.8× 116 1.1× 91 1.7× 106 2.0× 35 0.8× 7 315
Ashley B. Robbins United States 5 247 1.8× 205 1.9× 98 1.8× 82 1.5× 23 0.5× 8 405
Frank Zamudio United States 6 155 1.1× 141 1.3× 91 1.7× 43 0.8× 22 0.5× 7 292
Susana Carmona Portugal 7 172 1.2× 146 1.4× 133 2.5× 69 1.3× 58 1.3× 18 387
Christina E. Murray United Kingdom 7 173 1.2× 70 0.7× 119 2.2× 62 1.2× 23 0.5× 8 332

Countries citing papers authored by Federico Probst

Since Specialization
Citations

This map shows the geographic impact of Federico Probst's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Federico Probst with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Federico Probst more than expected).

Fields of papers citing papers by Federico Probst

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Federico Probst. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Federico Probst. The network helps show where Federico Probst may publish in the future.

Co-authorship network of co-authors of Federico Probst

This figure shows the co-authorship network connecting the top 25 collaborators of Federico Probst. A scholar is included among the top collaborators of Federico Probst based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Federico Probst. Federico Probst is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Deußing, Maximilian, Tanja Blume, Lena Kaiser, et al.. (2019). Amelioration of Tau Pathology by Anle138b Rescues Neuronal Function in a Mouse Model of Human Alzheimer's Disease Tau. Nuklearmedizin - NuclearMedicine. 1 indexed citations
2.
Brendel, Matthias, Anna Jaworska, Felix Overhoff, et al.. (2018). Efficacy of chronic BACE1 inhibition in PS2APP mice depends on the regional Aβ deposition rate and plaque burden at treatment initiation. Theranostics. 8(18). 4957–4968. 21 indexed citations
3.
Brendel, Matthias, Gernot Kleinberger, Federico Probst, et al.. (2017). Increase of TREM2 during Aging of an Alzheimer’s Disease Mouse Model Is Paralleled by Microglial Activation and Amyloidosis. Frontiers in Aging Neuroscience. 9. 8–8. 45 indexed citations
4.
Brendel, Matthias, Carola Focke, Tanja Blume, et al.. (2017). Time Courses of Cortical Glucose Metabolism and Microglial Activity Across the Life Span of Wild-Type Mice: A PET Study. Journal of Nuclear Medicine. 58(12). 1984–1990. 29 indexed citations
5.
Brendel, Matthias, Anna Jaworska, Federico Probst, et al.. (2016). Small-Animal PET Imaging of Tau Pathology with 18F-THK5117 in 2 Transgenic Mouse Models. Journal of Nuclear Medicine. 57(5). 792–798. 33 indexed citations
6.
Overhoff, Felix, Matthias Brendel, Anna Jaworska, et al.. (2016). Automated Spatial Brain Normalization and Hindbrain White Matter Reference Tissue Give Improved [18F]-Florbetaben PET Quantitation in Alzheimer's Model Mice. Frontiers in Neuroscience. 10. 45–45. 27 indexed citations
7.
Brendel, Matthias, Federico Probst, Anna Jaworska, et al.. (2016). Glial Activation and Glucose Metabolism in a Transgenic Amyloid Mouse Model: A Triple-Tracer PET Study. Journal of Nuclear Medicine. 57(6). 954–960. 92 indexed citations
8.
Brendel, Matthias, Anna Jaworska, Federico Probst, et al.. (2015). PET Imaging of Tau Pathology in Transgenic Mouse Models using [18F]THK-5117. 56. 248–248. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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