Fadi G. Akar

7.2k total citations · 2 hit papers
84 papers, 5.3k citations indexed

About

Fadi G. Akar is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Pathology and Forensic Medicine. According to data from OpenAlex, Fadi G. Akar has authored 84 papers receiving a total of 5.3k indexed citations (citations by other indexed papers that have themselves been cited), including 66 papers in Cardiology and Cardiovascular Medicine, 53 papers in Molecular Biology and 18 papers in Pathology and Forensic Medicine. Recurrent topics in Fadi G. Akar's work include Cardiac electrophysiology and arrhythmias (55 papers), Ion channel regulation and function (25 papers) and Mitochondrial Function and Pathology (19 papers). Fadi G. Akar is often cited by papers focused on Cardiac electrophysiology and arrhythmias (55 papers), Ion channel regulation and function (25 papers) and Mitochondrial Function and Pathology (19 papers). Fadi G. Akar collaborates with scholars based in United States, Türkiye and United Kingdom. Fadi G. Akar's co-authors include David Rosenbaum, Gordon F. Tomaselli, Kenneth R. Laurita, Steven Girouard, Joseph Pastore, Brian O’Rourke, Joseph G. Akar, David A. Kass, Richard S. Tunin and Miguel A. Aon and has published in prestigious journals such as Circulation, Journal of Clinical Investigation and Journal of the American College of Cardiology.

In The Last Decade

Fadi G. Akar

84 papers receiving 5.2k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Mechanism Linking T-Wave Alternans to the Genesis of Cardiac Fibrillation

1999 624 citations Fadi G. Akar, David Rosenbaum et al. Circulation profile →
Oxidative stress and inflammation as central mediators of atrial fibrillation in obesity and diabetes

2017 361 citations Basil S. Karam, Won-Joon Koh et al. Cardiovascular Diabetology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Fadi G. Akar United States	36	3.6k	3.1k	710	518	465	84	5.3k
Joshua I. Goldhaber United States	42	3.1k 0.9×	3.6k 1.2×	1.1k 1.5×	529 1.0×	466 1.0×	113	5.3k
Kathryn A. Yamada United States	41	2.5k 0.7×	3.1k 1.0×	524 0.7×	494 1.0×	363 0.8×	68	4.7k
Robert H. G. Schwinger Germany	44	4.0k 1.1×	3.5k 1.1×	741 1.0×	463 0.9×	728 1.6×	203	6.5k
John C. Shryock United States	41	3.1k 0.9×	3.0k 1.0×	859 1.2×	460 0.9×	288 0.6×	105	5.3k
Itsuo Kodama Japan	43	4.2k 1.2×	2.9k 0.9×	913 1.3×	188 0.4×	471 1.0×	209	5.9k
Steven M. Pogwizd United States	35	4.5k 1.3×	3.6k 1.2×	883 1.2×	393 0.8×	260 0.6×	96	5.6k
Masayasu Hiraoka Japan	40	4.6k 1.3×	3.7k 1.2×	1.4k 2.0×	508 1.0×	276 0.6×	296	6.1k
Barry London United States	44	4.3k 1.2×	3.3k 1.1×	992 1.4×	154 0.3×	330 0.7×	134	5.6k
Wayne Minobe United States	31	5.3k 1.4×	3.7k 1.2×	622 0.9×	534 1.0×	695 1.5×	49	7.6k
Marc A. Vos Netherlands	49	6.7k 1.8×	4.3k 1.4×	830 1.2×	193 0.4×	340 0.7×	225	7.8k

Countries citing papers authored by Fadi G. Akar

Since Specialization

Citations

This map shows the geographic impact of Fadi G. Akar's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fadi G. Akar with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fadi G. Akar more than expected).

Fields of papers citing papers by Fadi G. Akar

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Fadi G. Akar. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fadi G. Akar. The network helps show where Fadi G. Akar may publish in the future.

Co-authorship network of co-authors of Fadi G. Akar

This figure shows the co-authorship network connecting the top 25 collaborators of Fadi G. Akar. A scholar is included among the top collaborators of Fadi G. Akar based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Fadi G. Akar. Fadi G. Akar is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Wallace, Michael B., Xianyao Xu, Stuart G. Campbell, et al.. (2022). Humanized Dsp ACM Mouse Model Displays Stress-Induced Cardiac Electrical and Structural Phenotypes. Cells. 11(19). 3049–3049. 10 indexed citations

Mitchell, Lewis, Feng Xiong, Martín Aguilar, et al.. (2022). The inspection paradox: An important consideration in the evaluation of rotor lifetimes in cardiac fibrillation. Frontiers in Physiology. 13. 920788–920788. 2 indexed citations

Kuklik, Paweł, Feng Xiong, Martín Aguilar, et al.. (2019). Renewal Theory as a Universal Quantitative Framework to Characterize Phase Singularity Regeneration in Mammalian Cardiac Fibrillation. Circulation Arrhythmia and Electrophysiology. 12(12). e007569–e007569. 34 indexed citations

Strauss, Benjamin, Yassine Sassi, Carlos Bueno‐Betí, et al.. (2018). Intra-tracheal gene delivery of aerosolized SERCA2a to the lung suppresses ventricular arrhythmias in a model of pulmonary arterial hypertension. Journal of Molecular and Cellular Cardiology. 127. 20–30. 24 indexed citations

Akar, Fadi G., et al.. (2017). Abstract 17542: Novel Role of AMP-Activated Protein Kinase (AMPK) in Regulating Atrial Transcription Factors and Coordinating Channel Expression. Circulation. 1 indexed citations

Motloch, Lukas J., Kiyotake Ishikawa, Chaoqin Xie, et al.. (2017). Increased Afterload Following Myocardial Infarction Promotes Conduction-Dependent Arrhythmias That Are Unmasked by Hypokalemia. JACC Basic to Translational Science. 2(3). 258–269. 16 indexed citations

Karam, Basil S., et al.. (2017). Oxidative stress and inflammation as central mediators of atrial fibrillation in obesity and diabetes. Cardiovascular Diabetology. 16(1). 120–120. 361 indexed citations breakdown →

Chaanine, Antoine H., Mathieu Nonnenmacher, Erik Kohlbrenner, et al.. (2014). Effect of bortezomib on the efficacy of AAV9.SERCA2a treatment to preserve cardiac function in a rat pressure-overload model of heart failure. Gene Therapy. 21(4). 379–386. 20 indexed citations

Hesketh, Geoffrey G., Manish H. Shah, Victoria L. Halperin Kuhns, et al.. (2010). Ultrastructure and Regulation of Lateralized Connexin43 in the Failing Heart. Circulation Research. 106(6). 1153–1163. 123 indexed citations

10.

Jin, Hongwei, Robert D. Nass, Paul J. Joudrey, et al.. (2010). Altered Spatiotemporal Dynamics of the Mitochondrial Membrane Potential in the Hypertrophied Heart. Biophysical Journal. 98(10). 2063–2071. 14 indexed citations

11.

Xie, Chaoqin & Fadi G. Akar. (2010). Mitochondrial Dysfunction: A New Frontier in the Search for Elusive Arrhythmia Mechanisms. Frontiers in Physiology. 1. 163–163. 1 indexed citations

12.

Ulak, Güner, et al.. (2008). Neuronal NOS inhibitor 1-(2-trifluoromethylphenyl)-imidazole augment the effects of antidepressants acting via serotonergic system in the forced swimming test in rats. Pharmacology Biochemistry and Behavior. 90(4). 563–568. 48 indexed citations

13.

Roepke, Torsten K., Andrianos Kontogeorgis, Xianghua Xu, et al.. (2008). Targeted deletion of kcne2 impairs ventricular repolarization via disruption of I _K,slow1 and I _to,f. The FASEB Journal. 22(10). 3648–3660. 83 indexed citations

14.

Brown, David A., et al.. (2008). Effects of 4'-chlorodiazepam on cellular excitation-contraction coupling and ischaemia-reperfusion injury in rabbit heart. Cardiovascular Research. 79(1). 141–149. 72 indexed citations

15.

O’Rourke, Brian, Sonia Cortassa, Fadi G. Akar, & Miguel A. Aon. (2007). Mitochondrial Ion Channels in Cardiac Function and Dysfunction. Novartis Foundation symposium. 287. 140–156. 17 indexed citations

16.

Akar, Fadi G., Robert D. Nass, Eugenio Cingolani, et al.. (2007). Dynamic changes in conduction velocity and gap junction properties during development of pacing-induced heart failure. American Journal of Physiology-Heart and Circulatory Physiology. 293(2). H1223–H1230. 160 indexed citations

17.

Shah, Manish H., Fadi G. Akar, & Gordon F. Tomaselli. (2005). Molecular Basis of Arrhythmias. Circulation. 112(16). 2517–2529. 124 indexed citations

18.

Spragg, David, Fadi G. Akar, Robert Helm, et al.. (2005). Abnormal conduction and repolarization in late-activated myocardium of dyssynchronously contracting hearts. Cardiovascular Research. 67(1). 77–86. 103 indexed citations

19.

Akar, Fadi G., Richard C. Wu, Isabelle Deschênes, et al.. (2004). Phenotypic differences in transient outward K⁺ current of human and canine ventricular myocytes: insights into molecular composition of ventricular I_to. American Journal of Physiology-Heart and Circulatory Physiology. 286(2). H602–H609. 70 indexed citations

20.

Akar, Fadi G. & David Rosenbaum. (2003). Transmural Electrophysiological Heterogeneities Underlying Arrhythmogenesis in Heart Failure. Circulation Research. 93(7). 638–645. 230 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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