F C Arnett

3.1k total citations
43 papers, 2.0k citations indexed

About

F C Arnett is a scholar working on Rheumatology, Immunology and Pathology and Forensic Medicine. According to data from OpenAlex, F C Arnett has authored 43 papers receiving a total of 2.0k indexed citations (citations by other indexed papers that have themselves been cited), including 23 papers in Rheumatology, 23 papers in Immunology and 14 papers in Pathology and Forensic Medicine. Recurrent topics in F C Arnett's work include Systemic Lupus Erythematosus Research (15 papers), Systemic Sclerosis and Related Diseases (13 papers) and T-cell and B-cell Immunology (12 papers). F C Arnett is often cited by papers focused on Systemic Lupus Erythematosus Research (15 papers), Systemic Sclerosis and Related Diseases (13 papers) and T-cell and B-cell Immunology (12 papers). F C Arnett collaborates with scholars based in United States, Canada and Greece. F C Arnett's co-authors include John D. Reveille, Morris Reichlin, Maureen D. Mayes, Pravitt Gourh, Rosalind Ramsey‐Goldman, Robert P. Kimberly, M F Gourley, Ellen M. Ginzler, J E Salmon and Susan Millard and has published in prestigious journals such as Circulation, Journal of Clinical Investigation and The Journal of Immunology.

In The Last Decade

F C Arnett

42 papers receiving 2.0k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
F C Arnett United States 24 961 947 655 501 417 43 2.0k
Junichi Kaburaki Japan 29 799 0.8× 876 0.9× 1.2k 1.9× 555 1.1× 289 0.7× 69 2.6k
Susumu Sugai Japan 25 643 0.7× 405 0.4× 349 0.5× 353 0.7× 168 0.4× 71 2.2k
Daisuke Kawabata Japan 22 1.0k 1.1× 652 0.7× 182 0.3× 637 1.3× 230 0.6× 48 1.8k
Micaela Fredi Italy 25 1.1k 1.2× 610 0.6× 293 0.4× 633 1.3× 181 0.4× 89 1.7k
Masahide Kubo Japan 25 357 0.4× 311 0.3× 1.0k 1.6× 423 0.8× 248 0.6× 68 2.1k
Masataka Kuwana Japan 18 744 0.8× 401 0.4× 703 1.1× 1.0k 2.0× 150 0.4× 29 1.9k
Yutaka Okano Japan 22 618 0.6× 433 0.5× 1.3k 2.0× 928 1.9× 110 0.3× 66 2.0k
Masanari Kodera Japan 18 521 0.5× 437 0.5× 775 1.2× 532 1.1× 111 0.3× 40 1.5k
Capucine Daridon Germany 27 649 0.7× 1.1k 1.2× 242 0.4× 88 0.2× 373 0.9× 35 2.2k
Jason W. Bauer United States 9 935 1.0× 978 1.0× 121 0.2× 366 0.7× 197 0.5× 10 1.6k

Countries citing papers authored by F C Arnett

Since Specialization
Citations

This map shows the geographic impact of F C Arnett's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by F C Arnett with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites F C Arnett more than expected).

Fields of papers citing papers by F C Arnett

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by F C Arnett. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by F C Arnett. The network helps show where F C Arnett may publish in the future.

Co-authorship network of co-authors of F C Arnett

This figure shows the co-authorship network connecting the top 25 collaborators of F C Arnett. A scholar is included among the top collaborators of F C Arnett based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with F C Arnett. F C Arnett is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
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Zhou, Xiaodong, Momiao Xiong, Feiyang Tan, Xinjian Guo, & F C Arnett. (2006). SPARC, an upstream regulator of connective tissue growth factor in response to transforming growth factor β stimulation. Arthritis & Rheumatism. 54(12). 3885–3889. 18 indexed citations
3.
Arnett, F C, et al.. (2001). Glutathione S-transferase genotypes in systemic sclerosis and their association with clinical manifestations in early disease. Genes and Immunity. 2(4). 236–238. 24 indexed citations
4.
5.
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Salmon, J E, Susan Millard, F C Arnett, et al.. (1996). Fc gamma RIIA alleles are heritable risk factors for lupus nephritis in African Americans.. Journal of Clinical Investigation. 97(5). 1348–1354. 364 indexed citations
7.
Moulds, J.M., John D. Reveille, & F C Arnett. (1996). Structural polymorphisms of complement receptor 1 (CR1) in systemic lupus erythematosus (SLE) patients and normal controls of three ethnic groups. Clinical & Experimental Immunology. 105(2). 302–305. 24 indexed citations
8.
Эрдес, Ш. Ф., et al.. (1995). HLA class II and HLA‐B27 oligotyping in two Siberian native population groups. Tissue Antigens. 46(5). 382–386. 20 indexed citations
9.
Reveille, John D., et al.. (1995). HLA‐class II alleles and C4 null genes in Greeks with systemic lupus erythematosus. Tissue Antigens. 46(5). 417–421. 13 indexed citations
10.
Arnett, F C. (1993). Ankylosing spondylitis 1992: from Connor to transgenes.. PubMed. 22(3). 207–11. 2 indexed citations
11.
Reveille, John D., et al.. (1992). Association of amino acid sequences in the HLA-DQB1 first domain with antitopoisomerase I autoantibody response in scleroderma (progressive systemic sclerosis).. Journal of Clinical Investigation. 90(3). 973–980. 115 indexed citations
12.
Tomana, Milan, Ralph E. Schrohenloher, John D. Reveille, F C Arnett, & William J. Koopman. (1992). Abnormal galactosylation of serum IgG in patients with systemic lupus erythematosus and members of families with high frequency of autoimmune diseases. Rheumatology International. 12(5). 191–194. 46 indexed citations
13.
Arnett, F C. (1992). Genetic aspects of human lupus. Clinical Immunology and Immunopathology. 63(1). 4–6. 29 indexed citations
14.
Reveille, John D., et al.. (1992). Association of polar amino acids at position 26 of the HLA-DQB1 first domain with the anticentromere autoantibody response in systemic sclerosis (scleroderma).. Journal of Clinical Investigation. 89(4). 1208–1213. 70 indexed citations
15.
Arnett, F C, Maria Olsen, Kenton L. Anderson, & John D. Reveille. (1991). Molecular analysis of major histocompatibility complex alleles associated with the lupus anticoagulant.. Journal of Clinical Investigation. 87(5). 1490–1495. 84 indexed citations
16.
Reveille, John D., Mary Joan MacLeod, Karen B. Whittington, & F C Arnett. (1991). Specific amino acid residues in the second hypervariable region of HLA-DQA1 and DQB1 chain genes promote the Ro (SS-A)/La (SS-B) autoantibody responses. The Journal of Immunology. 146(11). 3871–3876. 127 indexed citations
17.
Arnett, F C, et al.. (1990). A Novel Immunoassay for the Quantitation of Human C4 Gene Products. PubMed. 7(2). 95–101. 14 indexed citations
18.
Targoff, Ira N., F C Arnett, L Berman, Charles A. O’Brien, & Morris Reichlin. (1989). Anti-KJ: a new antibody associated with the syndrome of polymyositis and interstitial lung disease.. Journal of Clinical Investigation. 84(1). 162–172. 41 indexed citations
19.
Arnett, F C, Robert G. Hamilton, Marianne Roebber, J B Harley, & Morris Reichlin. (1988). Increased frequencies of Sm and nRNP autoantibodies in American blacks compared to whites with systemic lupus erythematosus.. PubMed. 15(12). 1773–6. 92 indexed citations
20.
Uhl, Gregory S., et al.. (1974). Primary biliary cirrhosis in systrmic sclerosis (scleroderma) and polymyositis.. PubMed. 135(3). 191–8. 28 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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